Caudate Nucleus Is Enlarged in High-Functioning Medication-Naive Subjects with Autism
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Marieke Langen | Sarah Durston | Wouter G. Staal | H. Engeland | W. Staal | M. Langen | S. Durston | S. Palmen | Herman van Engeland | Saskia J. M. C. Palmen
[1] H. Ring,et al. Neuropsychiatry of the basal ganglia , 2002, Journal of neurology, neurosurgery, and psychiatry.
[2] R. Hales,et al. J Neuropsychiatry Clin Neurosci , 1992 .
[3] J. Pierri,et al. Frontostriatal measurement in treatment-naive children with obsessive-compulsive disorder. , 1997, Archives of general psychiatry.
[4] M. Keshavan,et al. Changes in caudate volume with neuroleptic treatment , 1994, The Lancet.
[5] W. Honer,et al. Reduced basal ganglia volumes after switching to olanzapine in chronically treated patients with schizophrenia. , 2004, The American journal of psychiatry.
[6] A. Couteur,et al. Autism Diagnostic Interview-Revised: A revised version of a diagnostic interview for caregivers of individuals with possible pervasive developmental disorders , 1994, Journal of autism and developmental disorders.
[7] P. Santosh,et al. Repetitive and stereotyped behaviours in pervasive developmental disorders. , 2006, Journal of child psychology and psychiatry, and allied disciplines.
[8] Heping Zhang,et al. Basal Ganglia volumes in patients with Gilles de la Tourette syndrome. , 2003, Archives of general psychiatry.
[9] E. Smeraldi,et al. Increased right caudate nucleus size in obsessive-compulsive disorder: Detection with magnetic resonance imaging , 1992, Psychiatry Research: Neuroimaging.
[10] J. Arnt. Antistereotypic effects of dopamine D-1 and D-2 antagonists after intrastriatal injection in rats. pharmacological and regional specificity , 1985, Naunyn-Schmiedeberg's Archives of Pharmacology.
[11] J. Sweeney,et al. Response-inhibition deficits in obsessive-compulsive disorder: an indicator of dysfunction in frontostriatal circuits. , 1997, Journal of psychiatry & neuroscience : JPN.
[12] D. Segal,et al. Multiple daily amphetamine administration: behavioral and neurochemical alterations. , 1980, Science.
[13] G C Curtis,et al. Neurophysiologic dysfunction in basal ganglia/limbic striatal and thalamocortical circuits as a pathogenetic mechanism of obsessive-compulsive disorder. , 1989, The Journal of neuropsychiatry and clinical neurosciences.
[14] J N Giedd,et al. Case study: acute basal ganglia enlargement and obsessive-compulsive symptoms in an adolescent boy. , 1996, Journal of the American Academy of Child and Adolescent Psychiatry.
[15] Ann M Graybiel,et al. Repetitive Behaviors in Monkeys Are Linked to Specific Striatal Activation Patterns , 2004, The Journal of Neuroscience.
[16] J. Mink,et al. Recent advances in Tourette syndrome research , 2006, Trends in Neurosciences.
[17] Joseph Piven,et al. An MRI study of the basal ganglia in autism , 1999, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[18] Erin A Hazlett,et al. Volumetric analysis and three-dimensional glucose metabolic mapping of the striatum and thalamus in patients with autism spectrum disorders. , 2006, The American journal of psychiatry.
[19] M. Mehmet Haznedar,et al. Striatal Volume on Magnetic Resonance Imaging and Repetitive Behaviors in Autism , 2005, Biological Psychiatry.
[20] Eileen Daly,et al. Brain and behaviour in children with 22q11.2 deletion syndrome: a volumetric and voxel-based morphometry MRI study. , 2006, Brain : a journal of neurology.
[21] Alan C. Evans,et al. Developmental trajectories of brain volume abnormalities in children and adolescents with attention-deficit/hyperactivity disorder. , 2002, JAMA.
[22] R. McCarley,et al. MRI anatomy of schizophrenia , 1999, Biological Psychiatry.
[23] J. Lieberman,et al. Increase in caudate nuclei volumes of first-episode schizophrenic patients taking antipsychotic drugs. , 1994, The American journal of psychiatry.
[24] H. Fibiger,et al. Attenuation of amphetamine‐induced motor stimulation and stereotypy by 6‐hydroxydopamine in the rat , 1973, British journal of pharmacology.
[25] Paul Maruff,et al. Cognitive Deficits in Obsessive–Compulsive Disorder on Tests of Frontal–Striatal Function , 1998, Biological Psychiatry.
[26] David A. Ziegler,et al. Dissociations of cerebral cortex, subcortical and cerebral white matter volumes in autistic boys. , 2003, Brain : a journal of neurology.
[27] M. Cuccaro,et al. Repetitive behaviors in autism: relationships with associated clinical features. , 2005, Research in developmental disabilities.
[28] R. Kahn,et al. The Effect of Clozapine on Caudate Nucleus Volume in Schizophrenic Patients Previously Treated with Typical Antipsychotics , 2001, Neuropsychopharmacology.
[29] Joost Janssen,et al. Larger Brains in Medication Naive High-Functioning Subjects with Pervasive Developmental Disorder , 2004, Journal of autism and developmental disorders.
[30] Philip D. Harvey,et al. Dorsal striatal size, shape, and metabolic rate in never-medicated and previously medicated schizophrenics performing a verbal learning task. , 1998, Archives of general psychiatry.
[31] M. Prior,et al. Repetitive Behaviour in Children with High Functioning Autism and Obsessive Compulsive Disorder , 2007, Journal of autism and developmental disorders.
[32] T. Sigmundsson,et al. Brain anatomy and sensorimotor gating in Asperger's syndrome. , 2002, Brain : a journal of neurology.
[33] John L. Bradshaw,et al. The Neurodevelopmental Frontostriatal Disorders: Evolutionary Adaptiveness and Anomalous Lateralization , 2000, Brain and Language.
[34] Sarah Durston,et al. Increased gray-matter volume in medication-naive high-functioning children with autism spectrum disorder , 2004, Psychological Medicine.
[35] R. Kahn,et al. Effect of clozapine on caudate nucleus volume in relation to symptoms of schizophrenia. , 2001, The American journal of psychiatry.
[36] R. Ridley,et al. The psychology of perseverative and stereotyped behaviour , 1994, Progress in Neurobiology.
[37] B. Peterson,et al. Reduced basal ganglia volumes in Tourette's syndrome using three‐dimensional reconstruction techniques from magnetic resonance images , 1993, Neurology.
[38] H. van Engeland,et al. No evidence for preferential involvement of medial temporal lobe structures in high-functioning autism , 2006, Psychological Medicine.
[39] S. Bryson,et al. Investigating the structure of the restricted, repetitive behaviours and interests domain of autism. , 2006, Journal of child psychology and psychiatry, and allied disciplines.