Effects of C‐type natriuretic peptide on rat cardiac contractility
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[1] Y. Furukawa,et al. CNP causes receptor-mediated positive dromotropic effects in anesthetized dog hearts. , 1998, American journal of physiology. Heart and circulatory physiology.
[2] J. Mehta,et al. Evidence for the presence of L-arginine-nitric oxide pathway in human red blood cells: relevance in the effects of red blood cells on platelet function. , 1998, Journal of cardiovascular pharmacology.
[3] G. Feuerstein,et al. Neurohormonal activation, oxygen free radicals, and apoptosis in the pathogenesis of congestive heart failure. , 1998, Journal of cardiovascular pharmacology.
[4] H. H. Chen,et al. C-type natriuretic peptide: the endothelial component of the natriuretic peptide system. , 1998, Journal of cardiovascular pharmacology.
[5] J. Diamond,et al. Cyclic GMP‐dependent protein kinase activation in the absence of negative inotropic effects in the rat ventricle , 1997, British journal of pharmacology.
[6] A. Takeshita,et al. Local expression of C-type natriuretic peptide markedly suppresses neointimal formation in rat injured arteries through an autocrine/paracrine loop. , 1997, Circulation.
[7] R. Cardinal,et al. Direct chronotropic effects of atrial and C‐type natriuretic peptides in anaesthetized dogs , 1996, British journal of pharmacology.
[8] W. Paulus,et al. Myocardial contractile response to nitric oxide and cGMP. , 1996, Circulation.
[9] O. Carretero,et al. Mechanisms of action of atrial natriuretic factor and C-type natriuretic peptide. , 1996, Hypertension.
[10] KotaroSumii,et al. cGMP-Dependent Protein Kinase Regulation of the L-Type Ca2+ Current in Rat Ventricular Myocytes , 1995 .
[11] N. Sperelakis,et al. cGMP-dependent protein kinase regulation of the L-type Ca2+ current in rat ventricular myocytes. , 1995, Circulation research.
[12] J. Potter,et al. Cardiac troponin I phosphorylation increases the rate of cardiac muscle relaxation. , 1995, Circulation research.
[13] E. A. Espiner,et al. Natriuretic hormones. , 1995, Endocrinology and metabolism clinics of North America.
[14] T. Doetschman,et al. Targeted ablation of the phospholamban gene is associated with markedly enhanced myocardial contractility and loss of beta-agonist stimulation. , 1994, Circulation research.
[15] E. Lakatta,et al. 8-bromo-cGMP reduces the myofilament response to Ca2+ in intact cardiac myocytes. , 1994, Circulation research.
[16] H. Itoh,et al. Cytokine-induced C-type natriuretic peptide (CNP) secretion from vascular endothelial cells--evidence for CNP as a novel autocrine/paracrine regulator from endothelial cells. , 1993, Endocrinology.
[17] M. Anand-Srivastava,et al. Atrial natriuretic factor receptors and signal transduction mechanisms. , 1993, Pharmacological reviews.
[18] W. Edwards,et al. Natriuretic peptide system in human heart failure. , 1993, Circulation.
[19] W. Barry,et al. Intracellular Calcium Homeostasis in Cardiac Myocytes , 1993, Circulation.
[20] M. Brown,et al. Natriuretic peptide receptor mRNAs in the rat and human heart. , 1992, The Journal of clinical investigation.
[21] K. Nakao,et al. Endothelial production of C-type natriuretic peptide and its marked augmentation by transforming growth factor-beta. Possible existence of "vascular natriuretic peptide system". , 1992, The Journal of clinical investigation.
[22] Simon C Watkins,et al. Negative inotropic effects of cytokines on the heart mediated by nitric oxide. , 1992, Science.
[23] D. Goeddel,et al. Selective activation of the B natriuretic peptide receptor by C-type natriuretic peptide (CNP). , 1991, Science.
[24] R Fischmeister,et al. Ca2+ current is regulated by cyclic GMP-dependent protein kinase in mammalian cardiac myocytes. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[25] A. Katz. Interplay between inotropic and lusitropic effects of cyclic adenosine monophosphate on the myocardial cell. , 1990, Circulation.
[26] D. Mccall,et al. Effect of atriopeptin II on Ca influx, contractile behavior and cyclic nucleotide content of cultured neonatal rat myocardial cells. , 1990, Journal of molecular and cellular cardiology.
[27] L. Neyses,et al. Action of atrial natriuretic peptide and angiotensin II on the myocardium: studies in isolated rat ventricular cardiomyocytes. , 1989, Biochemical and biophysical research communications.
[28] D. Brutsaert,et al. Effects of Damaging the Endocardial Surface on the Mechanical Performance of Isolated Cardiac Muscle , 1988, Circulation research.
[29] H. C. Hartzell,et al. Regulation of cardiac ion channels by catecholamines, acetylcholine and second messenger systems. , 1988, Progress in biophysics and molecular biology.
[30] H. Cingolani,et al. Critical evaluation of isometric indexes of relaxation in rat and cat papillary muscles and toad ventricular strips. , 1986, Journal of molecular and cellular cardiology.
[31] M. Inui,et al. Regulation of calcium transport by the ATPase-phospholamban system. , 1983, Journal of molecular and cellular cardiology.
[32] E. Kranias,et al. Phosphorylation of troponin I and phospholamban during catecholamine stimulation of rabbit heart , 1982, Nature.
[33] P. Spieckermann,et al. Culturing of calcium stable adult cardiac myocytes. , 1982, Journal of molecular and cellular cardiology.
[34] H. Cingolani,et al. Effect of isoproterenol on relation between maximal rate of contraction and maximal rate of relaxation. , 1977, The American journal of physiology.
[35] S. Epstein,et al. Positive Inotropic Effects of Dibutyryl Cyclic Adenosine 3′,5′‐Monophosphate , 1970, Circulation research.