Further evidence for the contribution of the RAD51C gene in hereditary breast and ovarian cancer susceptibility

[1]  Yuntao Xie,et al.  RAD51C germline mutations in Chinese women with familial breast cancer , 2011, Breast Cancer Research and Treatment.

[2]  P. Pérez-Segura,et al.  A HRM-based screening method detects RAD51C germ-line deleterious mutations in Spanish breast and ovarian cancer families , 2011, Breast Cancer Research and Treatment.

[3]  R. Scott,et al.  BRIP1, PALB2, and RAD51C mutation analysis reveals their relative importance as genetic susceptibility factors for breast cancer , 2011, Breast Cancer Research and Treatment.

[4]  D. Palli,et al.  Mutation screening of RAD51C in male breast cancer patients , 2011, Breast Cancer Research.

[5]  Marc Tischkowitz,et al.  RAD51C germline mutations in breast and ovarian cancer patients , 2010, Breast Cancer Research.

[6]  Jing Zhang,et al.  Screening RAD51C nucleotide alterations in patients with a family history of breast and ovarian cancer , 2010, Breast Cancer Research and Treatment.

[7]  Dieter Niederacher,et al.  Germline mutations in breast and ovarian cancer pedigrees establish RAD51C as a human cancer susceptibility gene , 2010, Nature Genetics.

[8]  Franca Fraternali,et al.  Mutation of the RAD51C gene in a Fanconi anemia–like disorder , 2010, Nature Genetics.

[9]  E. Levy-Lahad,et al.  Fanconi anemia and breast cancer susceptibility meet again , 2010, Nature Genetics.

[10]  A. D’Andrea,et al.  How the fanconi anemia pathway guards the genome. , 2009, Annual review of genetics.

[11]  Joel Dudley,et al.  MEGA: A biologist-centric software for evolutionary analysis of DNA and protein sequences , 2008, Briefings Bioinform..

[12]  Katri Pylkäs,et al.  A recurrent mutation in PALB2 in Finnish cancer families , 2007, Nature.

[13]  Tom Walsh,et al.  Ten genes for inherited breast cancer. , 2007, Cancer cell.

[14]  Nazneen Rahman,et al.  Truncating mutations in the Fanconi anemia J gene BRIP1 are low-penetrance breast cancer susceptibility alleles , 2006, Nature Genetics.

[15]  A. Mannermaa,et al.  Refinement of the 22q12-q13 Breast Cancer–Associated Region: Evidence of TMPRSS6 as a Candidate Gene in an Eastern Finnish Population , 2006, Clinical Cancer Research.

[16]  S. West,et al.  RAD51C Is Required for Holliday Junction Processing in Mammalian Cells , 2004, Science.

[17]  C. A. French,et al.  Identification of Functional Domains in the RAD51L2 (RAD51C) Protein and Its Requirement for Gene Conversion* , 2003, Journal of Biological Chemistry.

[18]  Hans Joenje,et al.  Biallelic Inactivation of BRCA2 in Fanconi Anemia , 2002, Science.

[19]  N E Day,et al.  A comprehensive model for familial breast cancer incorporating BRCA1, BRCA2 and other genes , 2002, British Journal of Cancer.

[20]  S C West,et al.  Identification and purification of two distinct complexes containing the five RAD51 paralogs. , 2001, Genes & development.

[21]  S. West,et al.  Complex formation by the human RAD51C and XRCC3 recombination repair proteins , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[22]  S. Seal,et al.  BRCA1 and BRCA2 mutations in breast cancer families with multiple primary cancers. , 2000, Clinical cancer research : an official journal of the American Association for Cancer Research.

[23]  R. Winqvist,et al.  Evidence of founder mutations in Finnish BRCA1 and BRCA2 families. , 1998, American journal of human genetics.