Swelling-activated ClC-3 activity regulates prostaglandin E2 release in human OUMS-27 chondrocytes.

[1]  W. Giles,et al.  K+ and Ca2+ Channels Regulate Ca2+ Signaling in Chondrocytes: An Illustrated Review , 2020, Cells.

[2]  A. Mobasheri,et al.  The Antihypertensive Drug Nifedipine Modulates the Metabolism of Chondrocytes and Human Bone Marrow-Derived Mesenchymal Stem Cells , 2019, Front. Endocrinol..

[3]  Takahiro Shimizu,et al.  Cell Volume-Activated and Volume-Correlated Anion Channels in Mammalian Cells: Their Biophysical, Molecular, and Pharmacological Properties , 2019, Pharmacological Reviews.

[4]  Michael Pusch,et al.  CLC Chloride Channels and Transporters: Structure, Function, Physiology, and Disease. , 2018, Physiological reviews.

[5]  W. Giles,et al.  A New Splice Variant of Large Conductance Ca2+-activated K+ (BK) Channel α Subunit Alters Human Chondrocyte Function* , 2016, The Journal of Biological Chemistry.

[6]  Yoshinobu Nakamura,et al.  OATP2A1/SLCO2A1-mediated prostaglandin E2 loading into intracellular acidic compartments of macrophages contributes to exocytotic secretion. , 2015, Biochemical pharmacology.

[7]  C. Fahlke,et al.  Neuronal ClC-3 Splice Variants Differ in Subcellular Localizations, but Mediate Identical Transport Functions* , 2015, The Journal of Biological Chemistry.

[8]  W. Giles,et al.  The ClC-7 Chloride Channel Is Downregulated by Hypoosmotic Stress in Human Chondrocytes , 2015, Molecular Pharmacology.

[9]  W. Giles,et al.  Orai1-Orai2 complex is involved in store-operated calcium entry in chondrocyte cell lines. , 2015, Cell calcium.

[10]  K. Konstantopoulos,et al.  Fluid shear stress‐induced osteoarthritis: roles of cyclooxygenase‐2 and its metabolic products in inducing the expression of proinflammatory cytokines and matrix metalloproteinases , 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[11]  Haifeng Zhang,et al.  Tamoxifen inhibits migration of estrogen receptor‐negative hepatocellular carcinoma cells by blocking the swelling‐activated chloride current , 2013, Journal of cellular physiology.

[12]  A. Ponce,et al.  The Role of Swelling-Activated Chloride Currents (ICL,swell) in the Regulatory Volume Decrease Response of Freshly Dissociated Rat Articular Chondrocytes , 2012, Cellular Physiology and Biochemistry.

[13]  R. Krawetz,et al.  Osmolarity regulates chondrogenic differentiation potential of synovial fluid derived mesenchymal progenitor cells. , 2012, Biochemical and biophysical research communications.

[14]  R. Barrett-Jolley,et al.  Cell Volume Regulation in Chondrocytes , 2011, Cellular Physiology and Biochemistry.

[15]  C. Lau,et al.  Effects of ion channels on proliferation in cultured human cardiac fibroblasts. , 2011, Journal of molecular and cellular cardiology.

[16]  A. Mobasheri,et al.  The Emerging Chondrocyte Channelome , 2010, Front. Physio..

[17]  W. Giles,et al.  Accelerated Ca2+ entry by membrane hyperpolarization due to Ca2+-activated K+ channel activation in response to histamine in chondrocytes. , 2010, American journal of physiology. Cell physiology.

[18]  F. Guilak,et al.  Functional characterization of TRPV4 as an osmotically sensitive ion channel in porcine articular chondrocytes. , 2009, Arthritis and rheumatism.

[19]  F. Toyoda,et al.  Regulatory role of tyrosine phosphorylation in the swelling‐activated chloride current in isolated rabbit articular chondrocytes , 2009, The Journal of physiology.

[20]  C. Lau,et al.  Regulation of cell proliferation by intermediate-conductance Ca2+-activated potassium and volume-sensitive chloride channels in mouse mesenchymal stem cells. , 2008, American journal of physiology. Cell physiology.

[21]  K. Toh,et al.  Intracellular ClC-3 chloride channels promote bone resorption in vitro through organelle acidification in mouse osteoclasts. , 2008, American journal of physiology. Cell physiology.

[22]  D. Duan,et al.  Altered properties of volume‐sensitive osmolyte and anion channels (VSOACs) and membrane protein expression in cardiac and smooth muscle myocytes from Clcn3‐/‐ mice , 2004, The Journal of physiology.

[23]  J. Riordan,et al.  The PDZ-binding Chloride Channel ClC-3B Localizes to the Golgi and Associates with Cystic Fibrosis Transmembrane Conductance Regulator-interacting PDZ Proteins* , 2003, The Journal of Biological Chemistry.

[24]  M. Musch,et al.  Regulation of Human CLC-3 Channels by Multifunctional Ca2+/Calmodulin-dependent Protein Kinase* , 2001, The Journal of Biological Chemistry.

[25]  Takahiro Shimizu,et al.  Receptor‐mediated control of regulatory volume decrease (RVD) and apoptotic volume decrease (AVD) , 2001, The Journal of physiology.

[26]  A. Draguhn,et al.  Disruption of ClC-3, a Chloride Channel Expressed on Synaptic Vesicles, Leads to a Loss of the Hippocampus , 2001, Neuron.

[27]  K. Shimada,et al.  Expression and canalicular localization of two isoforms of the ClC-3 chloride channel from rat hepatocytes. , 2000, American journal of physiology. Gastrointestinal and liver physiology.