Soluble E-selectin, ICAM-1 and VCAM-1 levels in systemic and coronary circulation in patients with variant angina.
暂无分享,去创建一个
[1] G. Specchia,et al. Clinical and angiographic correlates of leukocyte activation in unstable angina. , 1995, Journal of the American College of Cardiology.
[2] K. Okumura,et al. Soluble P-selectin is released into the coronary circulation after coronary spasm. , 1995, Circulation.
[3] T. Ueno,et al. Increased soluble form of P-selectin in patients with unstable angina. , 1995, Circulation.
[4] S. Maxwell,et al. Increased levels of the soluble adhesion molecule E‐selectin in essential hypertension , 1994, Journal of hypertension.
[5] M. Entman,et al. Inflammation in Acute Coronary Syndromes , 1993, Circulation.
[6] Y. Yazaki,et al. Expression of intercellular adhesion molecule-1 in rat heart with ischemia/reperfusion and limitation of infarct size by treatment with antibodies against cell adhesion molecules. , 1993, The American journal of pathology.
[7] G. Specchia,et al. Increased Expression of Neutrophil and Monocyte Adhesion Molecules in Unstable Coronary Artery Disease , 1993, Circulation.
[8] P. Selby,et al. Circulating intercellular adhesion molecule-1 (ICAM-1), E-selectin and vascular cell adhesion molecule-1 (VCAM-1) in human malignancies. , 1993, British Journal of Cancer.
[9] T. V. Gopal,et al. Soluble E-selectin is found in supernatants of activated endothelial cells and is elevated in the serum of patients with septic shock. , 1993, Journal of immunology.
[10] J. M. Munro,et al. Endothelial-leukocyte adhesive interactions in inflammatory diseases. , 1993, European heart journal.
[11] S. J. Cashman,et al. Soluble Forms of Vascular Adhesion Molecules, E‐Selectin, ICAM‐1, and VCAM‐1: Pathological Significance , 1992, Annals of the New York Academy of Sciences.
[12] A. Gearing,et al. Soluble forms of E-selectin, ICAM-1 and VCAM-1 are present in the supernatants of cytokine activated cultured endothelial cells. , 1992, Biochemical and biophysical research communications.
[13] M. Fujita,et al. The sensitivity of intracoronary injection of acetylcholine in inducing coronary spasm differs in patients with stable and unstable angina. , 1992, International journal of cardiology.
[14] M. Isobe,et al. Specific acceptance of cardiac allograft after treatment with antibodies to ICAM-1 and LFA-1. , 1992, Science.
[15] V. Fuster,et al. The pathogenesis of coronary artery disease and the acute coronary syndromes (2). , 1992, The New England journal of medicine.
[16] M. Fujita,et al. Biphasic changes (initial increase and late decrease) in coronary sinus venous oxygen saturation during anginal attacks induced by intracoronary acetylcholine in patients with variant angina. , 1992, Cardiology.
[17] G. Specchia,et al. Role of granulocytes in endothelial injury in coronary heart disease in humans. , 1991, Atherosclerosis.
[18] M. Cybulsky,et al. Endothelial expression of a mononuclear leukocyte adhesion molecule during atherogenesis. , 1991, Science.
[19] P. Newman,et al. Structure of the human gene encoding granule membrane protein-140, a member of the selectin family of adhesion receptors for leukocytes. , 1990, The Journal of biological chemistry.
[20] J. Gamble,et al. Prevention of activated neutrophil adhesion to endothelium by soluble adhesion protein GMP140. , 1990, Science.
[21] J. V. van Mourik,et al. Characterization of the enhanced adhesion of neutrophil leukocytes to thrombin-stimulated endothelial cells. , 1990, Journal of immunology.
[22] J. Mehta,et al. Increased neutrophil elastase release in unstable angina pectoris and acute myocardial infarction. , 1990, Journal of the American College of Cardiology.
[23] Rodger P. McEver,et al. Rapid neutrophil adhesion to activated endothelium mediated by GMP-140 , 1990, Nature.
[24] J. Mehta,et al. Neutrophil function in ischemic heart disease. , 1989, Circulation.
[25] S. Weiss. Tissue destruction by neutrophils. , 1989, The New England journal of medicine.
[26] C W Smith,et al. Recognition of an endothelial determinant for CD 18-dependent human neutrophil adherence and transendothelial migration. , 1988, The Journal of clinical investigation.
[27] P. J. Simpson,et al. Reduction of experimental canine myocardial reperfusion injury by a monoclonal antibody (anti-Mo1, anti-CD11b) that inhibits leukocyte adhesion. , 1988, The Journal of clinical investigation.