Visualization of doxorubicin-induced oxidative stress in isolated cardiac myocytes.
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[1] R. Ulrich,et al. A digitized-fluorescence-imaging study of mitochondrial Ca2+ increase by doxorubicin in cardiac myocytes. , 1992, The Biochemical journal.
[2] D. Acosta,et al. Mitochondrial regulation of superoxide by Ca2+: an alternate mechanism for the cardiotoxicity of doxorubicin. , 1991, Toxicology and applied pharmacology.
[3] I. Summerhayes,et al. Unusual retention of rhodamine 123 by mitochondria in muscle and carcinoma cells. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[4] I. Reynolds,et al. Glutamate induces the production of reactive oxygen species in cultured forebrain neurons following NMDA receptor activation , 1995, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[5] J. Noordhoek,et al. The involvement of an oxidative mechanism in the adriamycin induced toxicity in neonatal rat heart cell cultures. , 1985, Research communications in chemical pathology and pharmacology.
[6] R. Jennings,et al. The cell biology of acute myocardial ischemia. , 1991, Annual review of medicine.
[7] N. Sarvazyan,et al. Effects of doxorubicin on cardiomyocytes with reduced level of superoxide dismutase. , 1995, Life sciences.
[8] C. Higgins,et al. Acute effects of doxorubicin (adriamycin) on left ventricular function in dogs. , 1984, International journal of cardiology.
[9] J. Lown,,et al. Anthracycline and anthraquinone anticancer agents: current status and recent developments. , 1993, Pharmacology & therapeutics.
[10] G. Schuurhuis,et al. Doxorubicin (adriamycin): a critical review of free radical-dependent mechanisms of cytotoxicity. , 1990, Pharmacology & therapeutics.
[11] I. Pessah,et al. Anthraquinone-sensitized Ca2+ release channel from rat cardiac sarcoplasmic reticulum: possible receptor-mediated mechanism of doxorubicin cardiomyopathy. , 1990, Molecular pharmacology.
[12] P. van der Meer,et al. Xanthine oxidoreductase activity in perfused hearts of various species, including humans. , 1990, Circulation research.
[13] L. Kedes,et al. Doxorubicin selectively inhibits muscle gene expression in cardiac muscle cells in vivo and in vitro. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[14] R. Olson,et al. Doxorubicin cardiotoxicity: analysis of prevailing hypotheses , 1990, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[15] R. Friedman,et al. Nuclear catalyzed antibiotic free radical formation. , 1982, Cancer research.
[16] R. Danesi,et al. Superoxide anion production by doxorubicin analogs in heart sarcosomes and by mitochondrial NADH dehydrogenase. , 1990, Research communications in chemical pathology and pharmacology.
[17] S. Bondy. The Relation of Oxidative Stress and Hyperexcitation to Neurological Disease , 1995, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.
[18] J. Doroshow. Effect of anthracycline antibiotics on oxygen radical formation in rat heart. , 1983, Cancer research.
[19] H. Ischiropoulos,et al. Evaluation of 2',7'-dichlorofluorescin and dihydrorhodamine 123 as fluorescent probes for intracellular H2O2 in cultured endothelial cells. , 1993, Archives of biochemistry and biophysics.
[20] F. Civoli,et al. Flow cytometric detection of hydrogen peroxide production induced by doxorubicin in cancer cells. , 1994, Free radical biology & medicine.
[21] E. Thiel,et al. Biochemical modulation of cytotoxic drugs by cytokines: molecular mechanisms in experimental oncology. , 1995, Recent results in cancer research. Fortschritte der Krebsforschung. Progres dans les recherches sur le cancer.