Effect of fructose-1, 6-diphosphate versus diphenhydramine on mortality in compound 48/80-induced shock.

[1]  A. Markov,et al.  Metabolic responses to fructose-1,6-diphosphate in healthy subjects. , 2000, Metabolism: clinical and experimental.

[2]  W. Purcell,et al.  Reactive oxygen species generation and histamine release by activated mast cells: modulation by nitric oxide synthase inhibition , 1999, British journal of pharmacology.

[3]  M. Vieytes,et al.  Membrane potential changes associated with calcium signals in human lymphocytes and rat mast cells. , 1999, Life sciences.

[4]  P. D. del Nido,et al.  Administration of fructose 1,6-diphosphate during early reperfusion significantly improves recovery of contractile function in the postischemic heart. , 1998, The Journal of thoracic and cardiovascular surgery.

[5]  J. Eaton,et al.  Induction of nitric oxide synthase in macrophages: inhibition by fructose-1,6-diphosphate. , 1998, Biochemical and biophysical research communications.

[6]  S. Amir,et al.  An inhibitor of nitric oxide production, NG-nitro-L-arginine-methyl ester, improves survival in anaphylactic shock. , 1991, European journal of pharmacology.

[7]  L. Flancbaum,et al.  Effect of histamine receptor antagonists on mortality in compound 48/80-induced shock. , 1990, Circulatory shock.

[8]  A. Markov,et al.  Prevention of ischemic-hypoxic brain injury and death in rabbits with fructose-1,6-diphosphate. , 1990, Stroke.

[9]  A. Markov,et al.  Fructose 1-6 diphosphate prevents intestinal ischemic reperfusion injury and death in rats. , 1990, Gastroenterology.

[10]  A. Markov,et al.  Attenuation of ischemic renal injury with fructose 1,6-diphosphate. , 1989, The Journal of surgical research.

[11]  T. Forrester,et al.  Protective effect of exogenous fructose-1,6-diphosphate in cardiogenic shock. , 1988, Cardiovascular research.

[12]  A. Markov,et al.  Increasing survival of dogs subjected to hemorrhagic shock by administration of fructose 1-6 diphosphate. , 1987, Surgery.

[13]  A. Markov,et al.  Hemodynamics and metabolic effects of fructose 1-6 diphosphate in ischemia and shock--experimental and clinical observations. , 1986, Annals of emergency medicine.

[14]  A. Ichikawa,et al.  Histamine release induced from mast cells by active components of compound 48/80. , 1985, European journal of pharmacology.

[15]  L. Ferrucci,et al.  Hemodynamic and electrocardiographic effects of fructose-1,6-diphosphate in acute myocardial infarction. , 1985, The American journal of cardiology.

[16]  G. Lazzarino,et al.  Mast cell histamine release induced by doxorubicin and the inhibitory effect of fructose 1,6-diphosphate. , 1983, Arzneimittel-Forschung.

[17]  G. Lazzarino,et al.  Inhibiting effect of fructose 1,6 diphosphate on rat mast cell histamine release. , 1983, International journal of tissue reactions.

[18]  M. D. Turner,et al.  Fructose-1,6-diphosphate: an agent for treatment of experimental endotoxin shock. , 1981, Surgery.

[19]  A. Markov,et al.  Hemodynamic, electrocardiographic, and metabolic effects of fructose diphosphate on acute myocardial ischemia. , 1980, American heart journal.

[20]  M. Bianchi,et al.  Fructose-1, 6-diphosphate dependence on the toxicity and uptake of potassium ions. , 1980, Agressologie: revue internationale de physio-biologie et de pharmacologie appliquees aux effets de l'agression.

[21]  C. Niemegeers,et al.  Protection of rats from compound 48/80-induced lethality. A simple test for inhibitors of mast cell-mediated shock. , 1978, Archives internationales de pharmacodynamie et de therapie.

[22]  B. Altura,et al.  Beneficial and detrimental actions of histamine H1- and H2-receptor antagonists in circulatory shock. , 1978, Proceedings of the National Academy of Sciences of the United States of America.

[23]  M. Rovetto,et al.  Mechanisms of Glycolytic Inhibition in Ischemic Rat Hearts , 1975, Circulation research.

[24]  W. Kübler,et al.  Regulation of glycolysis in the ischemic and the anoxic myocardium. , 1970, Journal of molecular and cellular cardiology.