Comparison of functional variants in IFNL4 and IFNL3 for association with HCV clearance.
暂无分享,去创建一个
L. Prokunina-Olsson | T. O'Brien | H. Bonkovsky | R. Pfeiffer | C. Howell | A. Paquin | B. Edlin | H. Strickler | G. Kirk | T. Morgan | David L. Thomas | M. Kuniholm | Sabrina Chen | K. L. Lang Kuhs | D. Thomas | Krystle A Lang Kuhs
[1] Aasld Idsa Hcv Guidance Panel. Hepatitis C guidance: AASLD‐IDSA recommendations for testing, managing, and treating adults infected with hepatitis C virus , 2015, Hepatology.
[2] T. O'Brien,et al. Reply: Subgroup Differences in Response to 8 Weeks of Ledipasvir/Sofosbuvir for Chronic Hepatitis C , 2015, Open forum infectious diseases.
[3] Z. Kutalik,et al. Correction: Corrigendum: Reduced IFNλ4 activity is associated with improved HCV clearance and reduced expression of interferon-stimulated genes , 2014, Nature Communications.
[4] T. O'Brien,et al. Subgroup Differences in Response to 8 Weeks of Ledipasvir/Sofosbuvir for Chronic Hepatitis C , 2014, Open forum infectious diseases.
[5] L. Prokunina-Olsson,et al. IFN-λ4: the paradoxical new member of the interferon lambda family. , 2014, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[6] Z. Kutalik,et al. 75: A variant of IFNλ4 displaying decreased activity is associated with better hepatitis C virus clearance and reduced ISG expression , 2014 .
[7] M. Dennis,et al. Selection on a Variant Associated with Improved Viral Clearance Drives Local, Adaptive Pseudogenization of Interferon Lambda 4 (IFNL4) , 2014, PLoS genetics.
[8] L. Prokunina-Olsson,et al. Endogenous intrahepatic IFNs and association with IFN-free HCV treatment outcome. , 2014, The Journal of clinical investigation.
[9] L. Prokunina-Olsson,et al. IFNL4-ΔG genotype is associated with slower viral clearance in hepatitis C, genotype-1 patients treated with sofosbuvir and ribavirin. , 2014, The Journal of infectious diseases.
[10] Robert Herring,et al. Ledipasvir and sofosbuvir for 8 or 12 weeks for chronic HCV without cirrhosis. , 2014, The New England journal of medicine.
[11] J. Hoofnagle,et al. Therapy for hepatitis C--the costs of success. , 2014, The New England journal of medicine.
[12] E. Galmozzi,et al. Nonsynonymous variant Pro70Ser (rs117648444) in IFNL4 gene identifies carriers of the rs368234815 ΔG allele with higher HCV RNA decline during the first 4 weeks of pegylated interferon and ribavirin therapy in HCV-1 patients. , 2014, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.
[13] P. Ferenci,et al. Review article: genetic factors that modify the outcome of viral hepatitis , 2014, Alimentary pharmacology & therapeutics.
[14] L. Prokunina-Olsson,et al. Association of the IFNL4-ΔG Allele With Impaired Spontaneous Clearance of Hepatitis C Virus. , 2014, The Journal of infectious diseases.
[15] B. Clotet,et al. IFNL4 ss469415590 variant is a better predictor than rs12979860 of pegylated interferon-alpha/ribavirin therapy failure in hepatitis C virus/HIV-1 coinfected patients , 2014, AIDS.
[16] M. Carrington,et al. The favorable IFNL3 genotype escapes mRNA decay mediated by AU-rich elements and hepatitis C virus–induced microRNAs , 2013, Nature Immunology.
[17] T. Pietschmann,et al. Interferon lambda 4 signals via the IFNλ receptor to regulate antiviral activity against HCV and coronaviruses , 2013, The EMBO journal.
[18] M. Buti,et al. Faldaprevir and deleobuvir for HCV genotype 1 infection. , 2013, The New England journal of medicine.
[19] F. Negro,et al. IL28B expression depends on a novel TT/-G polymorphism which improves HCV clearance prediction , 2013, The Journal of experimental medicine.
[20] Barbara Rehermann,et al. A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus , 2013, Nature Genetics.
[21] Pål Sætrom,et al. Single Nucleotide Polymorphisms Can Create Alternative Polyadenylation Signals and Affect Gene Expression through Loss of MicroRNA-Regulation , 2012, PLoS Comput. Biol..
[22] M. Abdel-hamid,et al. Analysis of IL28B Variants in an Egyptian Population Defines the 20 Kilobases Minimal Region Involved in Spontaneous Clearance of Hepatitis C Virus , 2012, PloS one.
[23] B. Clotet,et al. Deciphering the Interleukin 28B Variants That Better Predict Response to Pegylated Interferon-α and Ribavirin Therapy in HCV/HIV-1 Coinfected Patients , 2012, PloS one.
[24] J. Casanova,et al. Evolutionary genetic dissection of human interferons , 2011, The Journal of experimental medicine.
[25] F. Pereyra,et al. Differential microRNA regulation of HLA-C expression and its association with HIV control , 2011, Nature.
[26] Sven Bergmann,et al. Genetic variation in IL28B is associated with chronic hepatitis C and treatment failure: a genome-wide association study. , 2010, Gastroenterology.
[27] David B. Goldstein,et al. Genetic variation in IL28B and spontaneous clearance of hepatitis C virus , 2009, Nature.
[28] A. Koike,et al. Genome-wide association of IL28B with response to pegylated interferon-α and ribavirin therapy for chronic hepatitis C , 2009, Nature Genetics.
[29] Thomas Berg,et al. IL28B is associated with response to chronic hepatitis C interferon-α and ribavirin therapy , 2009, Nature Genetics.
[30] Jacques Fellay,et al. Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance , 2009, Nature.
[31] J. Hoofnagle,et al. Early changes in hepatitis C virus (HCV) levels in response to peginterferon and ribavirin treatment in patients with chronic HCV genotype 1 infection. , 2009, The Journal of infectious diseases.
[32] B. A. Fox,et al. The Role of Genomic Data in the Discovery, Annotation and Evolutionary Interpretation of the Interferon-Lambda Family , 2009, PloS one.
[33] T. O'Brien,et al. Seroprevalence of hepatitis C virus and hepatitis B virus among San Francisco injection drug users, 1998 to 2000 , 2007, Hepatology.
[34] H. Conjeevaram,et al. Peginterferon and ribavirin treatment in African American and Caucasian American patients with hepatitis C genotype 1. , 2006, Gastroenterology.
[35] M. Young,et al. The Women's Interagency HIV Study: an Observational Cohort Brings Clinical Sciences to the Bench , 2005, Clinical Diagnostic Laboratory Immunology.
[36] Mark Daly,et al. Haploview: analysis and visualization of LD and haplotype maps , 2005, Bioinform..
[37] William M. Lee,et al. Evolution of the HALT-C Trial: pegylated interferon as maintenance therapy for chronic hepatitis C in previous interferon nonresponders. , 2004, Controlled clinical trials.
[38] M. Pepe. The Statistical Evaluation of Medical Tests for Classification and Prediction , 2003 .
[39] J. Watters,et al. HIV seroprevalence in injection drug users. , 1995, JAMA.
[40] A Munoz,et al. The Alive Study: A Longitudinal Study of HIV-1 Infection in Intravenous Drug Users: Description of Methods , 1991, NIDA research monograph.
[41] D. Harnois. Prolonged Therapy of Advanced Chronic Hepatitis C with Low-Dose Peginterferon , 2009 .
[42] A. Lewis-Antes,et al. IFN-lambdas mediate antiviral protection through a distinct class II cytokine receptor complex. , 2003, Nature immunology.
[43] Scott R. Presnell,et al. IL-28, IL-29 and their class II cytokine receptor IL-28R , 2002, Nature Immunology.