The respective and interaction effects of spinal GRs and MRs on radicular pain induced by chronic compression of the dorsal root ganglion in the rat

[1]  S. Nada,et al.  Effect of spironolactone on pain responses in mice , 2010, EXCLI journal.

[2]  B. Myers,et al.  Divergent effects of amygdala glucocorticoid and mineralocorticoid receptors in the regulation of visceral and somatic pain. , 2010, American journal of physiology. Gastrointestinal and liver physiology.

[3]  Kathryn M Refshauge,et al.  Prognosis for patients with chronic low back pain: inception cohort study , 2009, BMJ : British Medical Journal.

[4]  Jessica K. Alexander,et al.  Stress exacerbates neuropathic pain via glucocorticoid and NMDA receptor activation , 2009, Brain, Behavior, and Immunity.

[5]  Shuxing Wang,et al.  Regulation of the trigeminal NR1 subunit expression induced by inflammation of the temporomandibular joint region in rats , 2009, PAIN®.

[6]  M. Vinciguerra,et al.  Aldosterone activates NF-kappaB in the collecting duct. , 2009, Journal of the American Society of Nephrology : JASN.

[7]  G. Geisslinger,et al.  The IKK‐NF‐κB pathway: a source for novel molecular drug targets in pain therapy? , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[8]  R. Ji,et al.  Cytokine Mechanisms of Central Sensitization: Distinct and Overlapping Role of Interleukin-1β, Interleukin-6, and Tumor Necrosis Factor-α in Regulating Synaptic and Neuronal Activity in the Superficial Spinal Cord , 2008, The Journal of Neuroscience.

[9]  N. Sousa,et al.  Corticosteroid receptors and neuroplasticity , 2008, Brain Research Reviews.

[10]  Shuxing Wang,et al.  A Rat Model of Radicular Pain Induced by Chronic Compression of Lumbar Dorsal Root Ganglion with SURGIFLO ™ , 2008, Anesthesiology.

[11]  Shuxing Wang,et al.  Time-dependent effect of epidural steroid on pain behavior induced by chronic compression of dorsal root ganglion in rats , 2007, Brain Research.

[12]  X. Gu,et al.  Effects of intrathecal injection of prednisolone acetate on expression of NR2B subunit and nNOS in spinal cord of rats after chronic compression of dorsal root ganglia. , 2007, Annals of clinical and laboratory science.

[13]  Shuxing Wang,et al.  Central glucocorticoid receptors regulate the upregulation of spinal cannabinoid-1 receptors after peripheral nerve injury in rats , 2007, Pain.

[14]  Jun-Ming Zhang,et al.  Systemic Antiinflammatory Corticosteroid Reduces Mechanical Pain Behavior, Sympathetic Sprouting, and Elevation of Proinflammatory Cytokines in a Rat Model of Neuropathic Pain , 2007, Anesthesiology.

[15]  W. Guo,et al.  Glial–Cytokine–Neuronal Interactions Underlying the Mechanisms of Persistent Pain , 2007, The Journal of Neuroscience.

[16]  J. Mallet,et al.  Lentiviral-mediated Targeted NF-κB Blockade in Dorsal Spinal Cord Glia Attenuates Sciatic Nerve Injury-induced Neuropathic Pain in the Rat. , 2007, Molecular therapy : the journal of the American Society of Gene Therapy.

[17]  Y. Meng,et al.  Angiotensin II and Aldosterone stimulating NF-κB and AP-1 activation in hepatic fibrosis of rat , 2007, Regulatory Peptides.

[18]  J. Mallet,et al.  Lentiviral-mediated targeted NF-kappaB blockade in dorsal spinal cord glia attenuates sciatic nerve injury-induced neuropathic pain in the rat. , 2007, Molecular therapy : the journal of the American Society of Gene Therapy.

[19]  D. Cha,et al.  Spironolactone prevents diabetic nephropathy through an anti-inflammatory mechanism in type 2 diabetic rats. , 2006, Journal of the American Society of Nephrology : JASN.

[20]  Zhi-qiang Zhou,et al.  Effect of betamethasone on neuropathic pain and cerebral expression of NF-κB and cytokines , 2006, Neuroscience Letters.

[21]  Shuxing Wang,et al.  Downregulation of spinal glutamate transporter EAAC1 followingnerve injury is regulated by central glucocorticoid receptors in rats , 2006, Pain.

[22]  E. Schiffrin,et al.  Role of aldosterone in angiotensin II-induced cardiac and aortic inflammation, fibrosis, and hypertrophy. , 2005, Canadian journal of physiology and pharmacology.

[23]  S. Maier,et al.  Involvement of spinal cord nuclear factor κB activation in rat models of proinflammatory cytokine‐mediated pain facilitation , 2005, The European journal of neuroscience.

[24]  S. Maier,et al.  Central Proinflammatory Cytokines and Pain Enhancement , 2005, Neurosignals.

[25]  Shuxing Wang,et al.  Central Glucocorticoid Receptors Modulate the Expression and Function of Spinal NMDA Receptors after Peripheral Nerve Injury , 2005, The Journal of Neuroscience.

[26]  Shuxing Wang,et al.  Expression of Central Glucocorticoid Receptors after Peripheral Nerve Injury Contributes to Neuropathic Pain Behaviors in Rats , 2004, The Journal of Neuroscience.

[27]  M. Kawata,et al.  Visualization of Glucocorticoid Receptor and Mineralocorticoid Receptor Interactions in Living Cells with GFP-Based Fluorescence Resonance Energy Transfer , 2004, The Journal of Neuroscience.

[28]  K. Takeda,et al.  Effect of Methylprednisolone on Neuropathic Pain and Spinal Glial Activation in Rats , 2004, Anesthesiology.

[29]  A. Nácher,et al.  Pharmacokinetic models for the saturable absorption of cefuroxime axetil and saturable elimination of cefuroxime. , 2004, European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences.

[30]  G. Haegeman,et al.  The interplay between the glucocorticoid receptor and nuclear factor-kappaB or activator protein-1: molecular mechanisms for gene repression. , 2003, Endocrine reviews.

[31]  S. Maier,et al.  Spinal Glia and Proinflammatory Cytokines Mediate Mirror-Image Neuropathic Pain in Rats , 2003, The Journal of Neuroscience.

[32]  H. Akil,et al.  Mineralocorticoid receptor function in major depression. , 2003, Archives of general psychiatry.

[33]  B. Winkelstein,et al.  Nerve injury proximal or distal to the DRG induces similar spinal glial activation and selective cytokine expression but differential behavioral responses to pharmacologic treatment , 2001, The Journal of comparative neurology.

[34]  T. Harkany,et al.  Action of Glucocorticoids on Survival of Nerve Cells: Promoting Neurodegeneration or Neuroprotection? 1 , 2001, Journal of neuroendocrinology.

[35]  S. Doggrell,et al.  The spironolactone renaissance , 2001, Expert opinion on investigational drugs.

[36]  N. Kushwaha,et al.  Heterodimerization of Mineralocorticoid and Glucocorticoid Receptors at a Novel Negative Response Element of the 5-HT1A Receptor Gene* , 2001, The Journal of Biological Chemistry.

[37]  S. Makarov NF-κB as a therapeutic target in chronic inflammation: recent advances , 2000 .

[38]  F. Holsboer,et al.  The brain mineralocorticoid receptor: greedy for ligand, mysterious in function. , 2000, European journal of pharmacology.

[39]  A. Reeve,et al.  Intrathecally administered endotoxin or cytokines produce allodynia, hyperalgesia and changes in spinal cord neuronal responses to nociceptive stimuli in the rat , 2000, European journal of pain.

[40]  S. Makarov NF-kappaB as a therapeutic target in chronic inflammation: recent advances. , 2000, Molecular medicine today.

[41]  J. Cidlowski,et al.  Molecular control of immune/inflammatory responses: interactions between nuclear factor-kappa B and steroid receptor-signaling pathways. , 1999, Endocrine reviews.

[42]  S. Rivest,et al.  Effects of Systemic Immunogenic Insults and Circulating Proinflammatory Cytokines on the Transcription of the Inhibitory Factor κBα Within Specific Cellular Populations of the Rat Brain , 1999, Journal of neurochemistry.

[43]  M. Barrot,et al.  Dopamine-dependent responses to morphine depend on glucocorticoid receptors. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[44]  M. Joëls,et al.  Brain corticosteroid receptor balance in health and disease. , 1998, Endocrine reviews.

[45]  M. Herkenham,et al.  Induction of inhibitory factor kappaBalpha mRNA in the central nervous system after peripheral lipopolysaccharide administration: an in situ hybridization histochemistry study in the rat. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[46]  M. Sakanaka,et al.  Glucocorticoid‐ and mineralocorticoid receptors in microglial cells: The two receptors mediate differential effects of corticosteroids , 1997, Glia.

[47]  F. Holsboer,et al.  Heterodimerization between mineralocorticoid and glucocorticoid receptors increases the functional diversity of corticosteroid action. , 1996, Trends in pharmacological sciences.

[48]  K. Fuxe,et al.  Mapping and computer assisted morphometry and microdensitometry of glucocorticoid receptor immunoreactive neurons and glial cells in the rat central nervous system , 1994, Neuroscience.

[49]  E. Baulieu,et al.  Differential intracellular localization of human mineralocorticosteroid receptor on binding of agonists and antagonists. , 1994, The Biochemical journal.

[50]  G. Bennett,et al.  Extra-territorial pain in rats with a peripheral mononeuropathy: mechano-hyperalgesia and mechano-allodynia in the territory of an uninjured nerve , 1994, Pain.

[51]  Z. Krozowski,et al.  Glial cells express both mineralocorticoid and glucocorticoid receptors , 1991, The Journal of Steroid Biochemistry and Molecular Biology.

[52]  R. Ahima,et al.  Type I corticosteroid receptor‐like immunoreactivity in the rat CNS: Distribution and regulation by corticosteroids , 1991, The Journal of comparative neurology.

[53]  R. Ahima,et al.  Charting of Type II glucocorticoid receptor-like immunoreactivity in the rat central nervous system , 1990, Neuroscience.

[54]  R. Dubner,et al.  A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia , 1987, Pain.

[55]  E. R. Kloet,et al.  Two receptor systems for corticosterone in rat brain: microdistribution and differential occupation. , 1985, Endocrinology.

[56]  T. Yaksh,et al.  Chronic catheterization of the spinal subarachnoid space , 1976, Physiology & Behavior.

[57]  J. Funder,et al.  Glucocorticoid and mineralocorticoid receptors in gut mucosa. , 1975, Endocrinology.