Hydrogen sulfide prevents the vascular dysfunction induced by severe traumatic brain injury in rats by reducing reactive oxygen species and modulating eNOS and H2S-synthesizing enzyme expression.
暂无分享,去创建一个
A. Sánchez-López | Jorge Tapia-Martínez | Cindy L Santiago-Castañeda | Diana L. Silva-Velasco | D. Centurión | Saúl Huerta de la Cruz | F. I. López-Preza | Luisa Rocha | Jesús H. Beltrán-Ornelas | Cindy L. Santiago-Castañeda | Jorge A Tapia-Martínez
[1] K. Freeman,et al. Targeting hydrogen sulfide and nitric oxide to repair cardiovascular injury after trauma. , 2022, Nitric oxide : biology and chemistry.
[2] A. Sánchez-López,et al. Exogenous hydrogen sulfide restores CSE and CBS but no 3-MST protein expression in the hypothalamus and brainstem after severe traumatic brain injury , 2022, Metabolic Brain Disease.
[3] M. Mader,et al. The role of L-arginine metabolism in neurocritical care patients , 2021, Neural regeneration research.
[4] Wangxiao Bao,et al. The Peripheral Immune System and Traumatic Brain Injury: Insight into the role of T-helper cells , 2021, International journal of medical sciences.
[5] D. Loane,et al. Brain-gut axis dysfunction in the pathogenesis of traumatic brain injury. , 2021, The Journal of clinical investigation.
[6] K. Yaffe,et al. Epidemiology of Chronic Effects of Traumatic Brain Injury , 2021, Journal of neurotrauma.
[7] Gengshen Zhang,et al. Neuroprotective effect of hydrogen sulfide against glutamate-induced oxidative stress is mediated via the p53/glutaminase 2 pathway after traumatic brain injury , 2021, Aging.
[8] L. Rocha,et al. NaHS subchronic treatment improves hypertension induced by traumatic brain injury in rats through vasopressor sympathetic outflow inhibition. , 2021, Journal of neurotrauma.
[9] L. Concha,et al. Propylparaben Reduces the Long-Term Consequences in Hippocampus Induced by Traumatic Brain Injury in Rats: Its Implications as Therapeutic Strategy to Prevent Neurodegenerative Diseases. , 2020, Journal of Alzheimer's disease : JAD.
[10] Xindao Yin,et al. Altered hypothalamic functional connectivity in post-traumatic headache after mild traumatic brain injury , 2020, The Journal of Headache and Pain.
[11] T. Eleftheriadis,et al. Mistimed H2S upregulation, Nrf2 activation and antioxidant proteins levels in renal tubular epithelial cells subjected to anoxia and reoxygenation. , 2020, Biomedical reports.
[12] L. Concha,et al. Sodium cromoglycate decreases sensorimotor impairment and hippocampal alterations induced by severe traumatic brain injury in rats. , 2020, Journal of neurotrauma.
[13] N. Nagahara,et al. The protective role of the 3-mercaptopyruvate sulfurtransferase (3-MST)-hydrogen sulfide (H2S) pathway against experimental osteoarthritis , 2020, Arthritis Research & Therapy.
[14] Andrew J. Hoisington,et al. Inflammation in Traumatic Brain Injury. , 2020, Journal of Alzheimer's disease : JAD.
[15] N. Nagahara,et al. Cardiovascular phenotype of mice lacking 3-mercaptopyruvate sulfurtransferase. , 2020, Biochemical pharmacology.
[16] T. Huynh,et al. Wogonin attenuates the deleterious effects of traumatic brain injury in anesthetized Wistar rats , 2019, European journal of pharmacology.
[17] S. Tyagi,et al. Hyperhomocysteinemia induced endothelial progenitor cells dysfunction through hyper-methylation of CBS promoter. , 2019, Biochemical and biophysical research communications.
[18] F. White,et al. The TRPC6 inhibitor, larixyl acetate, is effective in protecting against traumatic brain injury-induced systemic endothelial dysfunction , 2019, Journal of Neuroinflammation.
[19] T. Novack,et al. Prevalence of Medical and Psychiatric Comorbidities Following Traumatic Brain Injury , 2019, The Journal of head trauma rehabilitation.
[20] S. Hicks,et al. A Review of MicroRNA Biomarkers in Traumatic Brain Injury , 2019, Journal of experimental neuroscience.
[21] G. O. Oludare,et al. L-arginine attenuates blood pressure and reverses the suppression of angiogenic risk factors in a rat model of preeclampsia. , 2018, Pathophysiology : the official journal of the International Society for Pathophysiology.
[22] S. Driver,et al. Achieving Weight Loss Among Individuals With Traumatic Brain Injury Through Lifestyle Interventions , 2018, Archives of Physical Medicine and Rehabilitation.
[23] Wen-Ning Wu,et al. GYY4137 Promotes Mice Feeding Behavior via Arcuate Nucleus Sulfur-Sulfhydrylation and AMPK Activation , 2018, Front. Pharmacol..
[24] A. Diedrich,et al. Cystathionine &ggr;-Lyase–Produced Hydrogen Sulfide Controls Endothelial NO Bioavailability and Blood Pressure , 2018, Hypertension.
[25] Andreea C. Bostan,et al. The basal ganglia and the cerebellum: nodes in an integrated network , 2018, Nature Reviews Neuroscience.
[26] A. Sánchez-López,et al. NaHS prejunctionally inhibits the cardioaccelerator sympathetic outflow in pithed rats , 2018, European journal of pharmacology.
[27] John D Lambris,et al. Innate immune responses to trauma , 2018, Nature Immunology.
[28] Junyang Jung,et al. Antioxidant and Cell-Signaling Functions of Hydrogen Sulfide in the Central Nervous System , 2018, Oxidative medicine and cellular longevity.
[29] S. Bhowmick,et al. Neurodegeneration and Sensorimotor Deficits in the Mouse Model of Traumatic Brain Injury , 2018, Brain sciences.
[30] Yuan Yang,et al. Unveiling neural coupling within the sensorimotor system: directionality and nonlinearity , 2017, The European journal of neuroscience.
[31] Saurabh Sharma,et al. Recent Advances in Pathophysiology of Traumatic Brain Injury , 2017, Current neuropharmacology.
[32] John H. Zhang,et al. A composite neurobehavioral test to evaluate acute functional deficits after cerebellar haemorrhage in rats , 2017, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[33] M. Janahmadi,et al. The protective effect of hydrogen sulfide (H2S) on traumatic brain injury (TBI) induced memory deficits in rats , 2017, Brain Research Bulletin.
[34] D. Menon,et al. DNA Methylation: Basic Biology and Application to Traumatic Brain Injury. , 2017, Journal of neurotrauma.
[35] B. Greenwald,et al. Autonomic Dysfunction after Mild Traumatic Brain Injury , 2017, Brain sciences.
[36] N. Villalba,et al. Traumatic brain injury impairs sensorimotor function in mice. , 2017, The Journal of surgical research.
[37] Ming-Jie Wang,et al. H2S Donor NaHS Changes the Production of Endogenous H2S and NO in D-Galactose-Induced Accelerated Ageing , 2017, Oxidative medicine and cellular longevity.
[38] Hongzhu Li,et al. The interaction of estrogen and CSE/H2S pathway in the development of atherosclerosis. , 2017, American journal of physiology. Heart and circulatory physiology.
[39] K. Inaba,et al. Catecholamines as outcome markers in isolated traumatic brain injury: the COMA-TBI study , 2017, Critical Care.
[40] D. Prough,et al. Persistent Behavioral Deficits in Rats after Parasagittal Fluid Percussion Injury. , 2016, Journal of neurotrauma.
[41] M. Nelson,et al. Traumatic Brain Injury Causes Endothelial Dysfunction in the Systemic Microcirculation through Arginase-1-Dependent Uncoupling of Endothelial Nitric Oxide Synthase. , 2017, Journal of neurotrauma.
[42] M. Vavilala,et al. Association of Early Hemodynamic Profile and the Development of Systolic Dysfunction Following Traumatic Brain Injury , 2017, Neurocritical Care.
[43] Xi-ping Chen,et al. Upregulation of 3-MST Relates to Neuronal Autophagy After Traumatic Brain Injury in Mice , 2017, Cellular and Molecular Neurobiology.
[44] T. Sloten. Vascular dysfunction: At the heart of cardiovascular disease, cognitive impairment and depressive symptoms , 2016 .
[45] N. Greig,et al. Neuroinflammation in animal models of traumatic brain injury , 2016, Journal of Neuroscience Methods.
[46] P. Adelson,et al. Diffuse traumatic brain injury affects chronic corticosterone function in the rat , 2016, Endocrine connections.
[47] Cheng Wang,et al. Elevated serum miR‐93, miR‐191, and miR‐499 are noninvasive biomarkers for the presence and progression of traumatic brain injury , 2016, Journal of neurochemistry.
[48] Fen Wang,et al. DNA methylation in cystathionine-γ-lyase (CSE) gene promoter induced by ox-LDL in macrophages and in apoE knockout mice. , 2016, Biochemical and biophysical research communications.
[49] A. Sánchez-López,et al. Pharmacological evidence that NaHS inhibits the vasopressor responses induced by stimulation of the preganglionic sympathetic outflow in pithed rats , 2016, European journal of pharmacology.
[50] P. Chakraborty,et al. Cystathionine β‐synthase regulates endothelial function via protein S‐sulfhydration , 2016, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[51] N. Carney,et al. Global neurotrauma research challenges and opportunities , 2015, Nature.
[52] J. Sweeney,et al. Sensorimotor dysfunctions as primary features of autism spectrum disorders , 2015, Science China Life Sciences.
[53] Kevin W. Swan,et al. Analysis of cardiovascular responses to the H2S donors Na2S and NaHS in the rat. , 2015, American journal of physiology. Heart and circulatory physiology.
[54] Datis Kharrazian. Traumatic Brain Injury and the Effect on the Brain-Gut Axis. , 2015, Alternative therapies in health and medicine.
[55] R. d'Emmanuele di Villa Bianca,et al. Cystathionine γ-lyase, a H2S-generating enzyme, is a GPBAR1-regulated gene and contributes to vasodilation caused by secondary bile acids. , 2015, American journal of physiology. Heart and circulatory physiology.
[56] C. Kevil,et al. Working with nitric oxide and hydrogen sulfide in biological systems. , 2015, American journal of physiology. Lung cellular and molecular physiology.
[57] S. Cuzzocrea,et al. Hydrogen sulfide-releasing cyclooxygenase inhibitor ATB-346 enhances motor function and reduces cortical lesion volume following traumatic brain injury in mice , 2014, Journal of Neuroinflammation.
[58] D. Tsikas,et al. Nitric Oxide-Related Oxidative Stress and Redox Status in Health and Disease , 2014, Oxidative medicine and cellular longevity.
[59] A. Waisman,et al. eNOS uncoupling in cardiovascular diseases--the role of oxidative stress and inflammation. , 2014, Current pharmaceutical design.
[60] A. Pitkänen,et al. Effect of lacosamide on structural damage and functional recovery after traumatic brain injury in rats , 2014, Epilepsy Research.
[61] P. Azouvi,et al. Changes in weight after traumatic brain injury in adult patients: a longitudinal study. , 2014, Clinical nutrition.
[62] Xi-ping Chen,et al. Hydrogen Sulfide Offers Neuroprotection on Traumatic Brain Injury in Parallel with Reduced Apoptosis and Autophagy in Mice , 2014, PloS one.
[63] A. Colantonio,et al. Defining traumatic brain injury in children and youth using International Classification of Diseases version 10 codes: a systematic review protocol , 2013, Systematic Reviews.
[64] H. Perlman,et al. Traumatic brain injury–induced alterations in peripheral immunity , 2013, The journal of trauma and acute care surgery.
[65] Z. Fei,et al. Protective effects of hydrogen sulfide in a rat model of traumatic brain injury via activation of mitochondrial adenosine triphosphate-sensitive potassium channels and reduction of oxidative stress. , 2013, The Journal of surgical research.
[66] Xi-ping Chen,et al. Dynamic Change of Hydrogen Sulfide After Traumatic Brain Injury and its Effect in Mice , 2013, Neurochemical Research.
[67] S. Mohapatra,et al. New perspectives on central and peripheral immune responses to acute traumatic brain injury , 2012, Journal of Neuroinflammation.
[68] L. Birnbaumer,et al. TLR4 activation of TRPC6-dependent calcium signaling mediates endotoxin-induced lung vascular permeability and inflammation , 2012, The Journal of experimental medicine.
[69] A. Peitzman,et al. Effect of the modified Glasgow Coma Scale score criteria for mild traumatic brain injury on mortality prediction: comparing classic and modified Glasgow Coma Scale score model scores of 13. , 2011, The Journal of trauma.
[70] D. Kline,et al. Hydrogen sulfide augments synaptic neurotransmission in the nucleus of the solitary tract. , 2011, Journal of neurophysiology.
[71] Guangdong Yang,et al. Specificity Protein-1 as a Critical Regulator of Human Cystathionine γ-Lyase in Smooth Muscle Cells* , 2011, The Journal of Biological Chemistry.
[72] Lingyun Wu,et al. Rat pancreatic level of cystathionine γ-lyase is regulated by glucose level via specificity protein 1 (SP1) phosphorylation , 2011, Diabetologia.
[73] Yan Sun,et al. The vasorelaxing effect of hydrogen sulfide on isolated rat aortic rings versus pulmonary artery rings , 2011, Acta Pharmacologica Sinica.
[74] D. Julian,et al. Hydrogen Sulfide Increases Nitric Oxide Production from Endothelial Cells by an Akt-Dependent Mechanism , 2011, Front. Physio..
[75] P. Dash,et al. Altered adrenergic receptor signaling following traumatic brain injury contributes to working memory dysfunction , 2011, Neuroscience.
[76] C. Roussos,et al. Hydrogen Sulfide Is an Endogenous Inhibitor of Phosphodiesterase Activity , 2010, Arteriosclerosis, thrombosis, and vascular biology.
[77] Lauren C. Frey,et al. A novel apparatus for lateral fluid percussion injury in the rat , 2009, Journal of Neuroscience Methods.
[78] Asla Pitkänen,et al. Quantitative MRI predicts long-term structural and functional outcome after experimental traumatic brain injury , 2009, NeuroImage.
[79] Norihiro Shibuya,et al. 3-Mercaptopyruvate sulfurtransferase produces hydrogen sulfide and bound sulfane sulfur in the brain. , 2009, Antioxidants & redox signaling.
[80] R. Banerjee,et al. Human Polycomb 2 Protein Is a SUMO E3 Ligase and Alleviates Substrate-Induced Inhibition of Cystathionine β-Synthase Sumoylation , 2008, PloS one.
[81] R. Teasell,et al. Hypermetabolism following moderate to severe traumatic acute brain injury: a systematic review. , 2008, Journal of neurotrauma.
[82] S. Snyder,et al. H2S as a Physiologic Vasorelaxant: Hypertension in Mice with Deletion of Cystathionine γ-Lyase , 2008, Science.
[83] A. Redington,et al. Effect of inhaled hydrogen sulfide on metabolic responses in anesthetized, paralyzed, and mechanically ventilated piglets* , 2008, Pediatric critical care medicine : a journal of the Society of Critical Care Medicine and the World Federation of Pediatric Intensive and Critical Care Societies.
[84] W. Durante,et al. Hyperhomocystinemia Impairs Endothelial Function and eNOS Activity via PKC Activation , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[85] R. Granacher. The Epidemiology and Pathophysiology of Traumatic Brain Injury , 2003 .
[86] Rui Wang,et al. The vasorelaxant effect of H2S as a novel endogenous gaseous KATP channel opener , 2001 .
[87] J. Trojanowski,et al. Enduring cognitive, neurobehavioral and histopathological changes persist for up to one year following severe experimental brain injury in rats , 1998, Neuroscience.
[88] R. Bartus,et al. Calpain inhibitor AK295 attenuates motor and cognitive deficits following experimental brain injury in the rat. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[89] L. Noble,et al. Traumatic brain injury in the rat: Characterization of a lateral fluid-percussion model , 1989, Neuroscience.