Targeting the molecular mechanisms of ischemic damage: Protective effects of alpha-crystallin-B.
暂无分享,去创建一个
G. Vilahur | L. Badimón | O. Juan-Babot | L. Casaní | T. Padró | G. Mendieta | E. Gómez-Jabalera | J. Cubedo
[1] N. Mewton,et al. Cyclosporine before PCI in Patients with Acute Myocardial Infarction. , 2015, The New England journal of medicine.
[2] G. Heusch. Molecular basis of cardioprotection: signal transduction in ischemic pre-, post-, and remote conditioning. , 2015, Circulation research.
[3] G. Tarone,et al. Keep your heart in shape: molecular chaperone networks for treating heart disease. , 2014, Cardiovascular research.
[4] M. Hermann,et al. A p38MAPK/MK2 signaling pathway leading to redox stress, cell death and ischemia/reperfusion injury , 2014, Cell Communication and Signaling.
[5] Susan Lindquist,et al. Mechanisms of protein-folding diseases at a glance , 2014, Disease Models & Mechanisms.
[6] J. Badimón,et al. Reperfusion-triggered stress protein response in the myocardium is blocked by post-conditioning. Systems biology pathway analysis highlights the key role of the canonical aryl-hydrocarbon receptor pathway. , 2013, European heart journal.
[7] A. Sanbe,et al. Changes in small heat shock proteins HSPB1, HSPB5 and HSPB8 in mitochondria of the failing heart following myocardial infarction in rats. , 2013, Biological & pharmaceutical bulletin.
[8] H. Kampinga,et al. HSPBs: small proteins with big implications in human disease. , 2012, The international journal of biochemistry & cell biology.
[9] Marta P Pereira,et al. Systemic augmentation of αB-crystallin provides therapeutic benefit twelve hours post-stroke onset via immune modulation , 2011, Proceedings of the National Academy of Sciences.
[10] Andreas Bracher,et al. Molecular chaperones in protein folding and proteostasis , 2011, Nature.
[11] L. Badimón,et al. Proteomic signature of Apolipoprotein J in the early phase of new-onset myocardial infarction. , 2011, Journal of proteome research.
[12] J. I. Izpisúa Belmonte,et al. Human progenitor cells derived from cardiac adipose tissue ameliorate myocardial infarction in rodents. , 2010, Journal of molecular and cellular cardiology.
[13] E. Barrón,et al. αB Crystallin Is Apically Secreted within Exosomes by Polarized Human Retinal Pigment Epithelium and Provides Neuroprotection to Adjacent Cells , 2010, PloS one.
[14] Alain Lilienbaum,et al. Serine 59 Phosphorylation of αB-Crystallin Down-regulates Its Anti-apoptotic Function by Binding and Sequestering Bcl-2 in Breast Cancer Cells* , 2010, The Journal of Biological Chemistry.
[15] Xuejun Wang,et al. Protein quality control in protection against systolic overload cardiomyopathy: the long term role of small heat shock proteins. , 2010, American journal of translational research.
[16] I. Cuthill,et al. Reporting : The ARRIVE Guidelines for Reporting Animal Research , 2010 .
[17] G. Vilahur,et al. Short‐term myocardial ischemia induces cardiac modified C‐reactive protein expression and proinflammatory gene (cyclo‐oxygenase‐2, monocyte chemoattractant protein‐1, and tissue factor) upregulation in peripheral blood mononuclear cells , 2009, Journal of thrombosis and haemostasis : JTH.
[18] G. Heusch,et al. Ischemic Postconditioning in Pigs: No Causal Role for Risk Activation , 2008, Circulation research.
[19] G. Finet,et al. Long-Term Benefit of Postconditioning , 2008, Circulation.
[20] I. Benjamin,et al. Unmasking different mechanical and energetic roles for the small heat shock proteins CryAB and HSPB2 using genetically modified mouse hearts , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[21] I. Benjamin,et al. CRYAB and HSPB2 deficiency alters cardiac metabolism and paradoxically confers protection against myocardial ischemia in aging mice. , 2007, American journal of physiology. Heart and circulatory physiology.
[22] S. Simon,et al. Hsp27 (HspB1) and αB‐crystallin (HspB5) as therapeutic targets , 2007 .
[23] Gemma Vilahur,et al. Early Metoprolol Administration Before Coronary Reperfusion Results in Increased Myocardial Salvage: Analysis of Ischemic Myocardium at Risk Using Cardiac Magnetic Resonance , 2007, Circulation.
[24] Randall J. Lee,et al. Myocardial infarct size measurement in the mouse chronic infarction model: comparison of area- and length-based approaches. , 2007, Journal of applied physiology.
[25] Y. Tao,et al. Small heat shock protein alphaB-crystallin binds to p53 to sequester its translocation to mitochondria during hydrogen peroxide-induced apoptosis. , 2007, Biochemical and biophysical research communications.
[26] John I. Clark,et al. Structure-based analysis of the β8 interactive sequence of human αB crystallin , 2006 .
[27] I. Benjamin,et al. CRYAB and HSPB2 deficiency increases myocyte mitochondrial permeability transition and mitochondrial calcium uptake. , 2006, Journal of molecular and cellular cardiology.
[28] G. Finet,et al. Postconditioning the Human Heart , 2005, Circulation.
[29] S. Slørdahl,et al. Myocardial ischaemia and the inflammatory response: release of heat shock protein 70 after myocardial infarction , 2005, Heart.
[30] Pamela S Douglas,et al. Distinct mouse coronary anatomy and myocardial infarction consequent to ligation , 2005, Coronary artery disease.
[31] R. Bolli,et al. Overexpression of Mitogen-activated Protein Kinase Kinase 6 in the Heart Improves Functional Recovery from Ischemia in Vitro and Protects against Myocardial Infarction in Vivo* , 2005, Journal of Biological Chemistry.
[32] H. Xiang,et al. Human αA- and αB-crystallins bind to Bax and Bcl-XS to sequester their translocation during staurosporine-induced apoptosis , 2004, Cell Death and Differentiation.
[33] E. Wawrousek,et al. Roles for alphaB-crystallin and HSPB2 in protecting the myocardium from ischemia-reperfusion-induced damage in a KO mouse model. , 2004, American journal of physiology. Heart and circulatory physiology.
[34] D. Das,et al. Transgene overexpression of αB crystallin confers simultaneous protection against cardiomyocyte apoptosis and necrosis during myocardial ischemia and reperfusion , 2001 .
[35] S. Saga,et al. Phosphorylation of αB-crystallin in Mitotic Cells and Identification of Enzymatic Activities Responsible for Phosphorylation* , 1998, The Journal of Biological Chemistry.
[36] D. McMillan,et al. Stress (heat shock) proteins: molecular chaperones in cardiovascular biology and disease. , 1998, Circulation research.
[37] W. Schaper,et al. Ischemia-induced phosphorylation and translocation of stress protein αB-crystallin to Z lines of myocardium. , 1998, American journal of physiology. Heart and circulatory physiology.
[38] N J Izzo,et al. HL-1 cells: a cardiac muscle cell line that contracts and retains phenotypic characteristics of the adult cardiomyocyte. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[39] F. Hartl. Molecular chaperones in cellular protein folding , 1996, Nature.
[40] G Grupp,et al. Comparison of normal, hypodynamic, and hyperdynamic mouse hearts using isolated work-performing heart preparations. , 1993, The American journal of physiology.
[41] M. Gaestel,et al. Small heat shock proteins are molecular chaperones. , 1993, The Journal of biological chemistry.
[42] M. Chiesi,et al. Alpha B-crystallin in cardiac tissue. Association with actin and desmin filaments. , 1992, Circulation research.
[43] R. Solaro,et al. Alterations in myofibrillar function and protein profiles after complete global ischemia in rat hearts. , 1992, Circulation research.
[44] L. Hightower,et al. Heat shock, stress proteins, chaperones, and proteotoxicity , 1991, Cell.
[45] M. Hermann,et al. A p 38 MAPK / MK 2 signaling pathway leading to redox stress , cell death and ischemia / reperfusion injury , 2014 .
[46] John I. Clark,et al. Structure-based analysis of the beta8 interactive sequence of human alphaB crystallin. , 2006, Biochemistry.
[47] E. Wawrousek,et al. CARDIOVASCULAR SYSTEM , 2005 .
[48] H. Xiang,et al. Human alphaA- and alphaB-crystallins bind to Bax and Bcl-X(S) to sequester their translocation during staurosporine-induced apoptosis. , 2004, Cell death and differentiation.
[49] S. Lindquist,et al. Heat Shock , 1991, Springer Berlin Heidelberg.