PRKC-isoform mRNA expression in human kidney transplant protocol biopsies: is there a high-glucose-induced regulation in the diabetic state?
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T. Krech | H. Haller | J. Becker | W. Gwinner | M. Meier | T. Kirsch | Michaela Beese | S. Krech
[1] F. Cobelli,et al. 31P-magnetic resonance spectroscopy (31P-MRS) detects early changes in kidney high-energy phosphate metabolism during a 6-month Valsartan treatment in diabetic and non-diabetic kidney-transplanted patients , 2012, Acta Diabetologica.
[2] H. Haller,et al. Targeting the protein kinase C family in the diabetic kidney: lessons from analysis of mutant mice , 2009, Diabetologia.
[3] Yuan Zhang,et al. Increased renal gene transcription of protein kinase C-β in human diabetic nephropathy: relationship to long-term glycaemic control , 2008, Diabetologia.
[4] Anke Schwarz,et al. Safety and Adequacy of Renal Transplant Protocol Biopsies , 2005, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.
[5] D. Suzuki,et al. High expression of PKC-MAPK pathway mRNAs correlates with glomerular lesions in human diabetic nephropathy. , 2004, Kidney international.
[6] A. Salahudeen. Cold ischemic injury of transplanted kidneys: new insights from experimental studies. , 2004, American journal of physiology. Renal physiology.
[7] J. Kapor-Drezgic,et al. Effect of high glucose on mesangial cell protein kinase C-delta and -epsilon is polyol pathway-dependent. , 1999, Journal of the American Society of Nephrology : JASN.
[8] G. Rosenberg,et al. Magnetic Resonance Spectroscopy , 2021, Encyclopedia of Autism Spectrum Disorders.