The suppression of long-term potentiation in rat hippocampus by inhibitors of nitric oxide synthase is temperature and age dependent
暂无分享,去创建一个
K. Murphy | M. Er̀rington | J. Williams | Y.-G. Li | A. Nayak | T. Bliss
[1] G. Lynch,et al. Effects of high-frequency synaptic stimulation on glumate receptor binding studied with a modified in vitro hippocampal slice preparation , 1982, Brain Research.
[2] H. Wigström,et al. Facilitated induction of hippocampal long-lasting potentiation during blockade of inhibition , 1983, Nature.
[3] P W Gage,et al. Inhibitory post‐synaptic currents in rat hippocampal CA1 neurones. , 1984, The Journal of physiology.
[4] D. Prince,et al. Temperature dependence of intrinsic membrane properties and synaptic potentials in hippocampal CA1 neurons in vitro , 1985, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[5] G. Lynch,et al. Patterned stimulation at the theta frequency is optimal for the induction of hippocampal long-term potentiation , 1986, Brain Research.
[6] T. Bliss,et al. Correlation between long‐term potentiation and release of endogenous amino acids from dentate gyrus of anaesthetized rats. , 1986, The Journal of physiology.
[7] G. Collingridge,et al. Low-frequency activation of the NMDA receptor system can prevent the induction of LTP , 1989, Neuroscience Letters.
[8] R. Nicoll,et al. Mechanisms generating the time course of dual component excitatory synaptic currents recorded in hippocampal slices , 1990, Neuron.
[9] G. Böhme,et al. Possible involvement of nitric oxide in long-term potentiation. , 1991, European journal of pharmacology.
[10] E. Kandel,et al. Tests of the roles of two diffusible substances in long-term potentiation: evidence for nitric oxide as a possible early retrograde messenger. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[11] D. Madison,et al. A requirement for the intercellular messenger nitric oxide in long-term potentiation. , 1991, Science.
[12] J. Garthwaite,et al. NMDA receptor activation in rat hippocampus induces cyclic GMP formation through the l-arginine-nitric oxide pathway , 1991, Neuroscience Letters.
[13] S. Lipton,et al. Effect of nitric oxide production on the redox modulatory site of the NMDA receptor-channel complex , 1992, Neuron.
[14] G. Böhme,et al. A Role for Nitric Oxide in Long‐term Potentiation , 1992, The European journal of neuroscience.
[15] D. Clifford,et al. Inhibition of long-term potentiation by NMDA-mediated nitric oxide release. , 1992, Science.
[16] O. Manzoni,et al. Nitric oxide-induced blockade of NMDA receptors , 1992, Neuron.
[17] R. Malenka,et al. The influence of prior synaptic activity on the induction of long-term potentiation. , 1992, Science.
[18] V. Gribkoff,et al. Evidence for nitric oxide synthase inhibitor-sensitive and insensitive hippocampal synaptic potentiation. , 1992, Journal of neurophysiology.
[19] George L. Wilcox,et al. The role of nitric oxide in hippocampal long-term potentiation , 1992, Neuron.
[20] T. Bliss,et al. A synaptic model of memory: long-term potentiation in the hippocampus , 1993, Nature.
[21] E. Kandel,et al. Nitric oxide and carbon monoxide produce activity-dependent long-term synaptic enhancement in hippocampus. , 1993, Science.
[22] P. Chapman,et al. Nitric oxide synthase inhibitors block long-term potentiation induced by weak but not strong tetanic stimulation at physiological brain temperatures in rat hippocampal slices , 1993, Neuroscience Letters.
[23] C. Zorumski,et al. Nitric oxide inhibitors facilitate the induction of hippocampal long-term potentiation by modulating NMDA responses. , 1993, Journal of neurophysiology.
[24] J. Sweatt,et al. Nitric oxide synthase-independent long-term potentiation in area CA1 of hippocampus. , 1993, Neuroreport.
[25] D. Madison,et al. Nitric oxide as an intercellular signal in long-term potentiation , 1993 .
[26] J. Davies,et al. Age-related effects of NMDA-stimulated concomitant release of nitric oxide and glutamate in cortical slices prepared from DBA/2 mice , 1993, Brain Research.