Reversible and phorbol ester-specific defect of protein kinase C translocation in hepatocytes isolated from phenobarbital-treated rats.
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[1] S. Fischer,et al. Correlation of phorbol ester promotion in the resistant C57BL/6J mouse with sustained hyperplasia but not ornithine decarboxylase or protein kinase C. , 1989, Cancer research.
[2] G. Nelsestuen,et al. Differences in the effects of phorbol esters and diacylglycerols on protein kinase C. , 1989, Biochemistry.
[3] R. Jirtle,et al. Independent mechanisms for tumor promoters phenobarbital and 12-O-tetradecanoylphorbol-13-acetate in reduction of epidermal growth factor binding by rat hepatocytes. , 1989, Cancer research.
[4] R. Isseroff,et al. Subcellular distribution of protein kinase C/phorbol ester receptors in differentiating mouse keratinocytes , 1989, Journal of cellular physiology.
[5] S. Kaufmann,et al. Type 3 protein kinase C localization to the nuclear envelope of phorbol ester-treated NIH 3T3 cells , 1989, The Journal of cell biology.
[6] K. Yanagimoto,et al. Transmembrane signaling: tumor promoter distribution. , 1989, Biochimica et biophysica acta.
[7] M. Houweling,et al. Isozymic forms of protein kinase C in regenerating rat liver , 1989, FEBS letters.
[8] Y. Nishizuka,et al. The molecular heterogeneity of protein kinase C and its implications for cellular regulation , 1988, Nature.
[9] R. Davis. Independent mechanisms account for the regulation by protein kinase C of the epidermal growth factor receptor affinity and tyrosine-protein kinase activity. , 1988, The Journal of biological chemistry.
[10] M. Ingelman-Sundberg,et al. Evidence for separate control by phorbol esters of CD18-dependent adhesion and translocation of protein kinase C in U-937 cells. , 1988, Cancer research.
[11] M. Hagiwara,et al. Cell type-specific expression of protein kinase C isozymes in the rabbit cerebellum. , 1988, The Journal of biological chemistry.
[12] Y. Okamoto,et al. Protein kinase C in the regenerating rat liver. , 1988, Biochemical and biophysical research communications.
[13] R. Jirtle,et al. Phenobarbital reduces EGF receptors and the ability of physiological concentrations of calcium to suppress hepatocyte proliferation. , 1988, Carcinogenesis.
[14] Y. Nozawa,et al. Biphasic effects of 12-O-tetradecanoylphorbol-13-acetate on the cell morphology of low calcium-grown human epidermal carcinoma cells: involvement of translocation and down regulation of protein kinase C. , 1988, Cancer research.
[15] C. Putnam,et al. Hepatic protein kinase C: translocation stimulated by prolactin and partial hepatectomy. , 1987, Life sciences.
[16] H. Koeffler,et al. Effects of phorbol ester on translocation and down-regulation of protein kinase C and phosphorylation of endogenous proteins in human acute myeloid leukemia cell line KG-1 and its phorbol ester-resistant subline KG-1a. , 1987, Cancer research.
[17] H. Pitot,et al. Effects of tumor-promoting agents 12-O-tetradecanoylphorbol-13-acetate and phenobarbital on DNA synthesis of rat hepatocytes in primary culture. , 1987, Cancer research.
[18] T. Starzl,et al. Different response to epidermal growth factor of hepatocytes in cultures isolated from male or female rat liver. Inhibitor effect of estrogen on binding and mitogenic effect of epidermal growth factor. , 1987, Gastroenterology.
[19] P. Blackshear,et al. Immunocytochemical evidence for phorbol ester-induced directional translocations of protein kinase C in HL60, K562, CHO, and E7SKS cells: possible role in differentiation. , 1987, Cancer research.
[20] A. Tashjian,et al. Time-dependent changes in protein kinase C distribution and disappearance in phorbol ester-treated human osteosarcoma cells. , 1987, Cancer research.
[21] C. McArdle,et al. Phorbol esters reduce gonadotrope responsiveness to protein kinase C activators but not to Ca2+-mobilizing secretagogues. Does protein kinase C mediate gonadotropin-releasing hormone action? , 1987, The Journal of biological chemistry.
[22] M. Vallotton,et al. Identification of a Major Defect in Insulin-Resistant Tissues of Genetically Obese (fa/fa) Rats: Impaired Protein Kinase C , 1987, Diabetes.
[23] E. Huberman,et al. Translocation of protein kinase C in human leukemia cells susceptible or resistant to differentiation induced by phorbol 12-myristate 13-acetate. , 1986, Proceedings of the National Academy of Sciences of the United States of America.
[24] M. Shoji,et al. Immunocytochemical evidence for phorbol ester-induced protein kinase C translocation in HL60 cells. , 1986, Biochemical and biophysical research communications.
[25] R. Evans,et al. Protein kinase C phosphorylation at Thr 654 of the unoccupied EGF receptor and EGF binding regulate functional receptor loss by independent mechanisms , 1986, Cell.
[26] M. Ingelman-Sundberg,et al. Phorbol ester-induced alteration of differentiation and proliferation in human hematopoietic tumor cell lines: relationship to the presence and subcellular distribution of protein kinase C. , 1985, Cancer research.
[27] P. Blackmore,et al. Stimulation of 1,2-diacylglycerol accumulation in hepatocytes by vasopressin, epinephrine, and angiotensin II. , 1985, The Journal of biological chemistry.
[28] G A McPherson,et al. Analysis of radioligand binding experiments. A collection of computer programs for the IBM PC. , 1985, Journal of pharmacological methods.
[29] R. W. Wrenn,et al. Redistribution of phospholipid/calcium-dependent protein kinase and altered phosphorylation of its soluble and particulate substrate proteins in phorbol ester-treated rat pancreatic acini. , 1985, Cancer research.
[30] Y. Nishizuka. The role of protein kinase C in cell surface signal transduction and tumour promotion , 1984, Nature.
[31] M. James,et al. Characterization of a specific phorbol ester aporeceptor in mouse brain cytosol. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[32] A. Kraft,et al. Phorbol esters increase the amount of Ca2+, phospholipid-dependent protein kinase associated with plasma membrane , 1983, Nature.
[33] Y. Nishizuka,et al. Calcium-activated, phospholipid-dependent protein kinase from rat brain. Subcellular distribution, purification, and properties. , 1982, The Journal of biological chemistry.
[34] G. Michalopoulos,et al. Liver regeneration studies with rat hepatocytes in primary culture. , 1982, Cancer research.
[35] M. Houslay,et al. Phenobarbital selectively modulates the glucagon-stimulated activity of adenylate cyclase by depressing the lipid phase separation occurring in the outer half of the bilayer of liver plasma membranes. , 1981, The Biochemical journal.
[36] M. Shoji,et al. Calcium-dependent protein kinase: widespread occurrence in various tissues and phyla of the animal kingdom and comparison of effects of phospholipid, calmodulin, and trifluoperazine. , 1980, Proceedings of the National Academy of Sciences of the United States of America.
[37] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.
[38] A. Ullrich,et al. Protein kinase C , 1987, Journal of cellular physiology. Supplement.
[39] E. Farber. Liver cell cancer: insights into the pathogenesis of hepatocellular carcinoma in humans from experimental hepatocarcinogenesis in the rat. , 1987, Monographs in pathology.
[40] W. Anderson,et al. Hormone- and tumor promoter-induced activation or membrane association of protein kinase C in intact cells. , 1987, Methods in enzymology.
[41] 吉川 潮. Calcium-activated, phospholipid-dependent protein kinase from rat brain , 1984 .
[42] J. Niedel,et al. Phorbol diester receptor copurifies with protein kinase C. , 1983, Proceedings of the National Academy of Sciences of the United States of America.