Superantigen-induced corticosteroid resistance of human T cells occurs through activation of the mitogen-activated protein kinase kinase/extracellular signal-regulated kinase (MEK-ERK) pathway.
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L. Ou | D. Leung | Ling-bo Li | E. Goleva | C. Hall
[1] P. Rothman,et al. Enhancement of MEK/ERK signaling promotes glucocorticoid resistance in CD4+ T cells , 2004 .
[2] G. Haegeman,et al. The interplay between the glucocorticoid receptor and nuclear factor-kappaB or activator protein-1: molecular mechanisms for gene repression. , 2003, Endocrine reviews.
[3] H. Schneider,et al. Unifying concepts in CD28, ICOS and CTLA4 co-receptor signalling , 2003, Nature Reviews Immunology.
[4] Claus Bachert,et al. IgE to Staphylococcus aureus enterotoxins in serum is related to severity of asthma. , 2003, The Journal of allergy and clinical immunology.
[5] J. Bloom,et al. Update on glucocorticoid action and resistance. , 2003, The Journal of allergy and clinical immunology.
[6] K. Kisich,et al. A Role for STAT5 in the Pathogenesis of IL-2-Induced Glucocorticoid Resistance1 , 2002, The Journal of Immunology.
[7] K. Imai,et al. Nuclear export of glucocorticoid receptor is enhanced by c-Jun N-terminal kinase-mediated phosphorylation. , 2002, Molecular endocrinology.
[8] D. Zaller,et al. Staging and resetting T cell activation in SMACs , 2002, Nature Immunology.
[9] M. Garabedian,et al. Deciphering the Phosphorylation “Code” of the Glucocorticoid Receptor in Vivo * 210 , 2002, The Journal of Biological Chemistry.
[10] B. Myones,et al. Prevalence of superantigen-secreting bacteria in patients with Kawasaki disease. , 2002, The Journal of pediatrics.
[11] I. Adcock,et al. p38 Mitogen-activated protein kinase-induced glucocorticoid receptor phosphorylation reduces its activity: role in steroid-insensitive asthma. , 2002, The Journal of allergy and clinical immunology.
[12] D. Olive,et al. Differential effect of CD28 versus B7 blockade on direct pathway of allorecognition and self-restricted responses. , 2002, Blood.
[13] A. Chow,et al. Temporal sequence and functional implications of V beta-specific T cell receptor down-regulation and costimulatory molecule expression following in vitro stimulation with the staphylococcal superantigen Toxic shock syndrome toxin-1. , 2002, The Journal of infectious diseases.
[14] Søren Buus,et al. Role of the T Cell Receptor Ligand Affinity in T Cell Activation by Bacterial Superantigens* , 2001, The Journal of Biological Chemistry.
[15] J. Gutiérrez-Ramos,et al. Cutting Edge: The Related Molecules CD28 and Inducible Costimulator Deliver Both Unique and Complementary Signals Required for Optimal T Cell Activation , 2001, The Journal of Immunology.
[16] K. Yamamoto,et al. Crosstalk pathway for inhibition of glucocorticoid-induced apoptosis by T cell receptor signaling. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[17] G. Chrousos,et al. Induction of corticosteroid insensitivity in human PBMCs by microbial superantigens. , 2000, The Journal of allergy and clinical immunology.
[18] S. Wenzel,et al. Increased T-cell receptor vbeta8+ T cells in bronchoalveolar lavage fluid of subjects with poorly controlled asthma: a potential role for microbial superantigens. , 1999, The Journal of allergy and clinical immunology.
[19] L. Picker,et al. Evidence for superantigen involvement in skin homing of T cells in atopic dermatitis. , 1999, The Journal of investigative dermatology.
[20] Michael Loran Dustin,et al. Costimulation: Building an Immunological Synapse , 1999, Science.
[21] Colin R. F. Monks,et al. Three-dimensional segregation of supramolecular activation clusters in T cells , 1998, Nature.
[22] J. Cidlowski,et al. Glucocorticoid receptor phosphorylation: Overview, function and cell cycle-dependence 1 1 Proceedings of the 13th International Symposium of the Journal of Steroid Biochemistry & Molecular Biology “Recent Advances in Steroid Biochemistry & Molecular Biology” Monaco 25–28 May 1997. , 1998, The Journal of Steroid Biochemistry and Molecular Biology.
[23] S. Logan,et al. Antagonism of glucocorticoid receptor transcriptional activation by the c-Jun N-terminal kinase. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[24] Mitogen-activated and cyclin-dependent protein kinases selectively and differentially modulate transcriptional enhancement by the glucocorticoid receptor , 1997, Molecular and cellular biology.
[25] J. Cidlowski,et al. Mouse Glucocorticoid Receptor Phosphorylation Status Influences Multiple Functions of the Receptor Protein* , 1997, The Journal of Biological Chemistry.
[26] M. Berridge. Lymphocyte activation in health and disease. , 1997, Critical reviews in immunology.
[27] H. Jabara,et al. The superantigen toxic shock syndrome toxin-1 induces CD40 ligand expression and modulates IgE isotype switching. , 1996, International immunology.
[28] K. Durongpisitkul,et al. The prevention of coronary artery aneurysm in Kawasaki disease: a meta-analysis on the efficacy of aspirin and immunoglobulin treatment. , 1995, Pediatrics.
[29] J. Travers,et al. Evidence for a streptococcal superantigen-driven process in acute guttate psoriasis. , 1995, The Journal of clinical investigation.
[30] J. Allison,et al. CD28 and CTLA-4 have opposing effects on the response of T cells to stimulation , 1995, The Journal of experimental medicine.
[31] M. Moser,et al. Activation of murine T cells by bacterial superantigens requires B7-mediated costimulation. , 1995, Cellular immunology.
[32] P. R. Housley,et al. Site-directed mutagenesis of the phosphorylation sites in the mouse glucocorticoid receptor. , 1993, The Journal of biological chemistry.
[33] S. Walker,et al. Intravenous immunoglobulin contains specific antibodies inhibitory to activation of T cells by staphylococcal toxin superantigens [see comment]. , 1993, The Journal of clinical investigation.
[34] P. Marrack,et al. Superantigens and their potential role in human disease. , 1993, Advances in immunology.
[35] J. Allison,et al. CD28-mediated signalling co-stimulates murine T cells and prevents induction of anergy in T-cell clones , 1992, Nature.
[36] P. Marrack,et al. Evidence for the effects of a superantigen in rheumatoid arthritis. , 1991, Science.
[37] M. Merćep,et al. Programmed T lymphocyte death. Cell activation- and steroid-induced pathways are mutually antagonistic. , 1990, Journal of immunology.
[38] P. Schlievert,et al. Clindamycin-induced suppression of toxic-shock syndrome--associated exotoxin production. , 1984, The Journal of infectious diseases.
[39] J. Leyden,et al. The case for steroid—antibiotic combinations , 1977, The British journal of dermatology.
[40] H. Maibach,et al. Co‐operative double‐blind trial of an antibiotic/ corticoid combination in impetiginized atopic dermatitis , 1976, The British journal of dermatology.