Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction
暂无分享,去创建一个
D. Kass | P. Menasché | B. Cousin | L. Casteilla | P. Bruneval | H. Rodewald | J. Vilar | J. Kirk | P. Launay | P. Bonnin | J. Silvestre | U. Blank | Luca Danelli | G. Gautier | E. Camerer | C. Heymes | Jonathan A. Kirk | C. Guerin | M. Ranek | M. Lemitre | A. Richart | E. Luche | P. Marck | Anta Ngkelo | M. Branchereau | Sylvain Le Gall | N. Renault | A. Kervadec | Mathilde Lemitre
[1] J. J. Walsh,et al. Mast cell stabilisers. , 2016, European journal of pharmacology.
[2] J. Kuiper,et al. The impact of mast cells on cardiovascular diseases. , 2016, European journal of pharmacology.
[3] O. Schmetzer,et al. Murine and human mast cell progenitors. , 2016, European journal of pharmacology.
[4] E. Marbán,et al. Macrophages mediate cardioprotective cellular postconditioning in acute myocardial infarction. , 2015, The Journal of clinical investigation.
[5] B. Keavney,et al. T lymphocytes and fractalkine contribute to myocardial ischemia/reperfusion injury in patients. , 2015, The Journal of clinical investigation.
[6] S. Herzig,et al. Hematopoietic Kit Deficiency, rather than Lack of Mast Cells, Protects Mice from Obesity and Insulin Resistance. , 2015, Cell metabolism.
[7] M. Movsesian. New pharmacologic interventions to increase cardiac contractility: challenges and opportunities , 2015, Current opinion in cardiology.
[8] R. Hajjar,et al. Altered myocardial calcium cycling and energetics in heart failure--a rational approach for disease treatment. , 2015, Cell metabolism.
[9] Sonal Singh,et al. Compartmentalization Role of A-Kinase Anchoring Proteins (AKAPs) in Mediating Protein Kinase A (PKA) Signaling and Cardiomyocyte Hypertrophy , 2014, International journal of molecular sciences.
[10] J. Davoust,et al. Mast cells aggravate sepsis by inhibiting peritoneal macrophage phagocytosis. , 2014, The Journal of clinical investigation.
[11] J. Alpert,et al. Republished: Clinical implications of the Third Universal Definition of Myocardial Infarction , 2014, Postgraduate Medical Journal.
[12] U. Rauch,et al. Role of protease-activated receptors for the innate immune response of the heart. , 2014, Trends in cardiovascular medicine.
[13] Jonathan P. Davis,et al. Combined troponin I Ser-150 and Ser-23/24 phosphorylation sustains thin filament Ca(2+) sensitivity and accelerates deactivation in an acidic environment. , 2014, Journal of molecular and cellular cardiology.
[14] G. Pejler,et al. Mast cell secretory granules: armed for battle , 2014, Nature Reviews Immunology.
[15] L. Walker,et al. Characterization and validation of new tools for measuring site-specific cardiac troponin I phosphorylation. , 2014, Journal of immunological methods.
[16] D. Kass,et al. Cardiac resynchronization sensitizes the sarcomere to calcium by reactivating GSK-3β. , 2014, The Journal of clinical investigation.
[17] S. Werner,et al. Mast Cells Are Dispensable for Normal and Activin-Promoted Wound Healing and Skin Carcinogenesis , 2013, The Journal of Immunology.
[18] M. Ruel,et al. Injectable small intestine submucosal extracellular matrix in an acute myocardial infarction model. , 2013, The Annals of thoracic surgery.
[19] Xiangru Lu,et al. Cardiomyocyte-specific overexpression of human stem cell factor protects against myocardial ischemia and reperfusion injury. , 2013, International journal of cardiology.
[20] D. Henrion,et al. Prokineticin Receptor‐1 Is a New Regulator of Endothelial Insulin Uptake and Capillary Formation to Control Insulin Sensitivity and Cardiovascular and Kidney Functions , 2013, Journal of the American Heart Association.
[21] D. Tsiantoulas,et al. B lymphocytes trigger monocyte mobilization and impair heart function after acute myocardial infarction , 2013, Nature Medicine.
[22] K. Murthy,et al. Distinctive G Protein-Dependent Signaling by Protease-Activated Receptor 2 (PAR2) in Smooth Muscle: Feedback Inhibition of RhoA by cAMP-Independent PKA , 2013, PloS one.
[23] V. Barroca,et al. In situ production of innate immune cells in murine white adipose tissue. , 2012, Blood.
[24] G. Shi,et al. Mast cell chymase and tryptase as targets for cardiovascular and metabolic diseases. , 2012, Current pharmaceutical design.
[25] M. Tsai,et al. Evidence questioning cromolyn’s effectiveness and selectivity as a ‘mast cell stabilizer’ in mice , 2012, Laboratory Investigation.
[26] K. Clément,et al. Mast cells in human adipose tissue: link with morbid obesity, inflammatory status, and diabetes. , 2012, The Journal of clinical endocrinology and metabolism.
[27] C. Benoist,et al. Cre-mediated cell ablation contests mast cell contribution in models of antibody- and T cell-mediated autoimmunity. , 2011, Immunity.
[28] K. F. Austen,et al. Mast cell deficiency, a game of kit and mouse. , 2011, Immunity.
[29] Ying Ge,et al. Augmented Phosphorylation of Cardiac Troponin I in Hypertensive Heart Failure* , 2011, The Journal of Biological Chemistry.
[30] J. Brazin,et al. Targeting cardiac mast cells: pharmacological modulation of the local renin-angiotensin system. , 2011, Current pharmaceutical design.
[31] J. S. Janicki,et al. Tryptase/Protease-Activated Receptor 2 Interactions Induce Selective Mitogen-Activated Protein Kinase Signaling and Collagen Synthesis by Cardiac Fibroblasts , 2011, Hypertension.
[32] J. S. Janicki,et al. Tryptase activates isolated adult cardiac fibroblasts via protease activated receptor-2 (PAR-2) , 2011, Journal of Cell Communication and Signaling.
[33] R. Moss,et al. Protein kinase A–induced myofilament desensitization to Ca2+ as a result of phosphorylation of cardiac myosin–binding protein C , 2010, The Journal of general physiology.
[34] C. Balke,et al. Cardiac Troponin T, a Sarcomeric AKAP, Tethers Protein Kinase A at the Myofilaments* , 2010, The Journal of Biological Chemistry.
[35] B. Cousin,et al. Adipose Tissue as a Dedicated Reservoir of Functional Mast Cell Progenitors , 2010, Stem cells.
[36] N. Mackman,et al. Protease-Activated Receptor 2 Deficiency Reduces Cardiac Ischemia/Reperfusion Injury , 2010, Arteriosclerosis, thrombosis, and vascular biology.
[37] J. Reagan,et al. G-Protein-Coupled Receptor 35 Is a Target of the Asthma Drugs Cromolyn Disodium and Nedocromil Sodium , 2010, Pharmacology.
[38] Keshava Rajagopal,et al. Teaching old receptors new tricks: biasing seven-transmembrane receptors , 2010, Nature Reviews Drug Discovery.
[39] S. Sadayappan,et al. Phosphorylation and function of cardiac myosin binding protein-C in health and disease. , 2010, Journal of molecular and cellular cardiology.
[40] S. Cortassa,et al. Calcium sensitivity, force frequency relationship and cardiac troponin I: critical role of PKA and PKC phosphorylation sites. , 2010, Journal of molecular and cellular cardiology.
[41] I. Weissman,et al. Distinguishing mast cell and granulocyte differentiation at the single-cell level. , 2010, Cell stem cell.
[42] C. Kahn,et al. Transplantation of adipose tissue and stem cells: role in metabolism and disease , 2010, Nature Reviews Endocrinology.
[43] G. Koh,et al. Adipose tissue is an extramedullary reservoir for functional hematopoietic stem and progenitor cells. , 2010, Blood.
[44] S. Matecki,et al. Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophy , 2010, Proceedings of the National Academy of Sciences.
[45] K. Clément,et al. Deficiency and pharmacological stabilization of mast cells reduce diet-induced obesity and diabetes in mice , 2009, Nature Medicine.
[46] Guoping Wang,et al. Ischaemia/reperfusion induced cardiac stem cell homing to the injured myocardium by stimulating stem cell factor expression via NF‐κB pathway , 2009, International journal of experimental pathology.
[47] P. Kovanen. Mast cells in atherogenesis: Actions and reactions , 2009, Current atherosclerosis reports.
[48] M. Hesse,et al. c-kit expression identifies cardiovascular precursors in the neonatal heart , 2009, Proceedings of the National Academy of Sciences of the United States of America.
[49] C. Ward,et al. Phosphorylation of the cAMP-dependent Protein Kinase (PKA) Regulatory Subunit Modulates PKA-AKAP Interaction, Substrate Phosphorylation, and Calcium Signaling in Cardiac Cells* , 2008, Journal of Biological Chemistry.
[50] R. Guyton,et al. PAR-2 activation at the time of reperfusion salvages myocardium via an ERK1/2 pathway in in vivo rat hearts. , 2007, American journal of physiology. Heart and circulatory physiology.
[51] R. Moss,et al. Differential Roles of Cardiac Myosin-Binding Protein C and Cardiac Troponin I in the Myofibrillar Force Responses to Protein Kinase A Phosphorylation , 2007, Circulation research.
[52] T. Arumugam,et al. Effect of cromolyn on S100P interactions with RAGE and pancreatic cancer growth and invasion in mouse models. , 2006, Journal of the National Cancer Institute.
[53] Donna H. Wang,et al. Protease-activated receptor 2-mediated protection of myocardial ischemia-reperfusion injury: role of transient receptor potential vanilloid receptors. , 2006, American journal of physiology. Regulatory, integrative and comparative physiology.
[54] J. S. Janicki,et al. Rat cardiac mast cell maturation and differentiation following acute ventricular volume overload , 2006, Inflammation Research.
[55] S. Verma,et al. Cardioprotective c-kit+ cells are from the bone marrow and regulate the myocardial balance of angiogenic cytokines. , 2006, The Journal of clinical investigation.
[56] R. Solaro,et al. Specific enhancement of sarcomeric response to Ca2+ protects murine myocardium against ischemia-reperfusion dysfunction. , 2005, American journal of physiology. Heart and circulatory physiology.
[57] C. Chen,et al. Mast cell-deficient W-sash c-kit mutant Kit W-sh/W-sh mice as a model for investigating mast cell biology in vivo. , 2005, The American journal of pathology.
[58] I. Weissman,et al. Identification of mast cell progenitors in adult mice. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[59] Ajay M Shah,et al. Regulation of cardiac contractile function by troponin I phosphorylation. , 2005, Cardiovascular research.
[60] Pamela S Douglas,et al. Distinct mouse coronary anatomy and myocardial infarction consequent to ligation , 2005, Coronary artery disease.
[61] M. Entman,et al. Mast cell tryptase may modulate endothelial cell phenotype in healing myocardial infarcts , 2005, The Journal of pathology.
[62] S. Coughlin,et al. Regulated Shedding of PAR1 N-terminal Exodomain from Endothelial Cells* , 2004, Journal of Biological Chemistry.
[63] A. Erdei,et al. Regulation of mast cell activation by complement-derived peptides. , 2004, Immunology letters.
[64] D. Ward,et al. A cross-linking study of the N-terminal extension of human cardiac troponin I. , 2003, Biochemistry.
[65] H. Tokumitsu,et al. Hsp90 is a direct target of the anti-allergic drugs disodium cromoglycate and amlexanox. , 2003, The Biochemical journal.
[66] B. Sykes. Pulling the calcium trigger , 2003, Nature Structural Biology.
[67] I. Kulikovskaya,et al. Effect of extraction of myosin binding protein C on contractility of rat heart. , 2003, American journal of physiology. Heart and circulatory physiology.
[68] K. Koike,et al. IL-9 Enhances the Growth of Human Mast Cell Progenitors Under Stimulation with Stem Cell Factor1 , 2003, The Journal of Immunology.
[69] L. Pénicaud,et al. Reconstitution of lethally irradiated mice by cells isolated from adipose tissue. , 2003, Biochemical and biophysical research communications.
[70] D. Taub,et al. Stem cell factor induces eosinophil activation and degranulation: mediator release and gene array analysis. , 2002, Blood.
[71] H. Tokumitsu,et al. Interaction of S100 proteins with the antiallergic drugs, olopatadine, amlexanox, and cromolyn: identification of putative drug binding sites on S100A1 protein. , 2002, Biochemical and biophysical research communications.
[72] A. Ahluwalia,et al. Protease-Activated Receptor-2 Activation Causes EDHF-Like Coronary Vasodilation: Selective Preservation in Ischemia/Reperfusion Injury: Involvement of Lipoxygenase Products, VR1 Receptors, and C-Fibers , 2002, Circulation research.
[73] C. Stroh,et al. Agonists of proteinase-activated receptor 2 induce cytokine release and activation of nuclear transcription factor kappaB in human dermal microvascular endothelial cells. , 2002, The Journal of investigative dermatology.
[74] K. McDonald,et al. Power Output Is Increased After Phosphorylation of Myofibrillar Proteins in Rat Skinned Cardiac Myocytes , 2001, Circulation research.
[75] R. Marthan,et al. Tryptase and agonists of PAR-2 induce the proliferation of human airway smooth muscle cells. , 2001, Journal of applied physiology.
[76] I Kulikovskaya,et al. Multiple structures of thick filaments in resting cardiac muscle and their influence on cross-bridge interactions. , 2001, Biophysical journal.
[77] C. Napoli,et al. Protease-activated receptor-2 modulates myocardial ischemia-reperfusion injury in the rat heart. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[78] M. Gautel,et al. cAPK‐phosphorylation controls the interaction of the regulatory domain of cardiac myosin binding protein C with myosin‐S2 in an on‐off fashion , 1999, FEBS letters.
[79] P. Rosevear,et al. NMR analysis of cardiac troponin C‐troponin I complexes: effects of phosphorylation , 1999, FEBS letters.
[80] M. Lindsey,et al. Stem cell factor induction is associated with mast cell accumulation after canine myocardial ischemia and reperfusion. , 1998, Circulation.
[81] D. Payan,et al. Mast cell tryptase regulates rat colonic myocytes through proteinase-activated receptor 2. , 1997, The Journal of clinical investigation.
[82] S. Coughlin,et al. The Cloned Thrombin Receptor Is Necessary and Sufficient for Activation of Mitogen-activated Protein Kinase and Mitogenesis in Mouse Lung Fibroblasts , 1996, The Journal of Biological Chemistry.
[83] K. Zsebo,et al. Effect of IL-3 and stem cell factor on the appearance of human basophils and mast cells from CD34+ pluripotent progenitor cells. , 1992, Journal of immunology.
[84] C. Agustí,et al. Identification of mast cells in bronchoalveolar lavage fluid , 1991, Allergy.
[85] Y. Kitamura,et al. Decrease of mast cells in W/Wv mice and their increase by bone marrow transplantation. , 1978, Blood.
[86] J. S. Janicki,et al. The emerging prominence of the cardiac mast cell as a potent mediator of adverse myocardial remodeling. , 2015, Methods in molecular biology.
[87] A. Ibrahim,et al. Acute myocardial infarction. , 2014, Critical care clinics.
[88] S. Phan,et al. Cardiovascular , Pulmonary and Renal Pathology Role of Stem Cell Factor and Bone Marrow-Derived Fibroblasts in Airway Remodeling , 2010 .
[89] R. Solaro,et al. Heart failure, ischemia/reperfusion injury and cardiac troponin. , 2007, Advances in experimental medicine and biology.
[90] B. Sykes,et al. Structural based insights into the role of troponin in cardiac muscle pathophysiology , 2004, Journal of Muscle Research & Cell Motility.
[91] M. Hollenberg,et al. Mast cell tryptase stimulates human lung fibroblast proliferation via protease-activated receptor-2. , 2000, American journal of physiology. Lung cellular and molecular physiology.
[92] T. Zuberbier,et al. Mast cell and myeloid marker expression during early in vitro mast cell differentiation from human peripheral blood mononuclear cells. , 2000, The Journal of investigative dermatology.