Functional role of PPAR-γ on the proliferation and migration of fibroblast-like synoviocytes in rheumatoid arthritis
暂无分享,去创建一个
Cheng Huang | Xiao-ming Meng | Jun Li | Lei Zhang | Xin Chen | Xiao-Feng Li | Yang Yang | Yu-huan Li | Jing Bao | Bao‐ming Wu | Ying-yin Sun
[1] Cheng Huang,et al. MicroRNA-20a negatively regulates expression of NLRP3-inflammasome by targeting TXNIP in adjuvant-induced arthritis fibroblast-like synoviocytes. , 2016, Joint, bone, spine : revue du rhumatisme.
[2] B. Ghanim,et al. The prognostic significance of β-catenin, cyclin D1 and PIN1 in minor salivary gland carcinoma: β-catenin predicts overall survival , 2016, European Archives of Oto-Rhino-Laryngology.
[3] Shu Wu,et al. Overexpression of Suprabasin is Associated with Proliferation and Tumorigenicity of Esophageal Squamous Cell Carcinoma , 2016, Scientific Reports.
[4] J. Koh,et al. HIF-2α-induced chemokines stimulate motility of fibroblast-like synoviocytes and chondrocytes into the cartilage-pannus interface in experimental rheumatoid arthritis mouse models , 2015, Arthritis Research & Therapy.
[5] M. Volin,et al. The pathogenic role of angiogenesis in rheumatoid arthritis , 2015, Angiogenesis.
[6] W. Maśliński,et al. The effect of multimeric adiponectin isoforms and leptin on the function of rheumatoid fibroblast-like synoviocytes , 2015, Scandinavian journal of rheumatology.
[7] T. Gajewski,et al. Melanoma-intrinsic β-catenin signalling prevents anti-tumour immunity , 2015, Nature.
[8] J. Zwerina,et al. Brief Report: Tumor Necrosis Factor and Interleukin‐6 Differentially Regulate Dkk‐1 in the Inflamed Arthritic Joint , 2015, Arthritis & rheumatology.
[9] A. Lavecchia,et al. On the Metabolically Active Form of Metaglidasen: Improved Synthesis and Investigation of Its Peculiar Activity on Peroxisome Proliferator‐Activated Receptors and Skeletal Muscles , 2015, ChemMedChem.
[10] Ying Shi,et al. MiR-148b suppresses cell proliferation and invasion in hepatocellular carcinoma by targeting WNT1/β-catenin pathway , 2015, Scientific Reports.
[11] Kyunglim Lee,et al. Translationally Controlled Tumor Protein induces epithelial to mesenchymal transition and promotes cell migration, invasion and metastasis , 2015, Scientific Reports.
[12] L. Saha,et al. Anti-kindling Effect of Bezafibrate, a Peroxisome Proliferator-activated Receptors Alpha Agonist, in Pentylenetetrazole Induced Kindling Seizure Model , 2014, Journal of epilepsy research.
[13] Wei Hu,et al. Pioglitazone Improves Potassium Channel Remodeling Induced by Angiotensin II in Atrial Myocytes , 2014, Medical science monitor basic research.
[14] A. Shintani,et al. Reversing Vascular Dysfunction in Rheumatoid Arthritis: Improved Augmentation Index but Not Endothelial Function With Peroxisome Proliferator–Activated Receptor γ Agonist Therapy , 2014, Arthritis & Rheumatology.
[15] Zhiyuan Niu,et al. Chrysin attenuates inflammation by regulating M1/M2 status via activating PPARγ. , 2014, Biochemical pharmacology.
[16] Julio Collazos,et al. Role of plasma matrix-metalloproteases (MMPs) and their polymorphisms (SNPs) in sepsis development and outcome in ICU patients , 2014, Scientific Reports.
[17] S. Bae,et al. Meta-analysis of associations between the peroxisome proliferator-activated receptor-γ Pro12Ala polymorphism and susceptibility to nonalcoholic fatty liver disease, rheumatoid arthritis, and psoriatic arthritis. , 2014, Genetic testing and molecular biomarkers.
[18] Cheng Huang,et al. Inhibition of transient receptor potential melastatin 7 (TRPM7) channel induces RA FLSs apoptosis through endoplasmic reticulum (ER) stress , 2014, Clinical Rheumatology.
[19] G. Watts,et al. New peroxisome proliferator-activated receptor agonists: potential treatments for atherogenic dyslipidemia and non-alcoholic fatty liver disease , 2014, Expert opinion on pharmacotherapy.
[20] D. Chappard,et al. The peroxisome proliferator-activated receptor γ agonist pioglitazone preserves bone microarchitecture in experimental arthritis by reducing the interleukin-17-dependent osteoclastogenic pathway. , 2013, Arthritis and rheumatism.
[21] R. Brook,et al. The Peroxisome Proliferator Activated Receptor‐γ Pioglitazone Improves Vascular Function and Decreases Disease Activity in Patients With Rheumatoid Arthritis , 2013, Journal of the American Heart Association.
[22] Ki-Jo Kim,et al. Interaction of Mesenchymal Stem Cells with Fibroblast-like Synoviocytes via Cadherin-11 Promotes Angiogenesis by Enhanced Secretion of Placental Growth Factor , 2014, The Journal of Immunology.
[23] J. Rullmann,et al. p38 inhibition and not MK2 inhibition enhances the secretion of chemokines from TNF-α activated rheumatoid arthritis fibroblast-like synoviocytes. , 2013, Clinical and experimental rheumatology.
[24] Sung-Hwan Park,et al. MRP8 promotes Th17 differentiation via upregulation of IL-6 production by fibroblast-like synoviocytes in rheumatoid arthritis , 2013, Experimental & Molecular Medicine.
[25] M. Feldmann,et al. Differential effects of Th1 versus Th2 cytokines in combination with hypoxia on HIFs and angiogenesis in RA , 2012, Arthritis Research & Therapy.
[26] D. Mukherjee,et al. PPAR- γ agonist in treatment of diabetes: cardiovascular safety considerations. , 2012, Cardiovascular & hematological agents in medicinal chemistry.
[27] S. Yamasaki,et al. Post-transcriptional regulation of IL-6 production by Zc3h12a in fibroblast-like synovial cells. , 2011, Clinical and experimental rheumatology.
[28] Licia Maria Henrique da Mota,et al. The Wnt signaling pathway and rheumatoid arthritis. , 2010, Autoimmunity reviews.
[29] I. Palomo,et al. Peroxisome proliferator-activated receptors: Targets for the treatment of metabolic illnesses (Review). , 2008, Molecular medicine reports.
[30] Yuanyuan Wu,et al. The roles of endogenous reactive oxygen species and nitric oxide in triptolide-induced apoptotic cell death in macrophages , 2006, Journal of Molecular Medicine.
[31] Y. Okada,et al. Differential expression of WNTs and FRPs in the synovium of rheumatoid arthritis and osteoarthritis. , 2006, Biochemical and biophysical research communications.
[32] R. Nusse,et al. The Wnt signaling pathway in development and disease. , 2004, Annual review of cell and developmental biology.
[33] F. Breedveld,et al. Analysis of the cell infiltrate and expression of proinflammatory cytokines and matrix metalloproteinases in arthroscopic synovial biopsies: comparison with synovial samples from patients with end stage, destructive rheumatoid arthritis , 2003, Annals of the rheumatic diseases.
[34] G. Firestein,et al. Modulation of fibroblast-mediated cartilage degradation by articular chondrocytes in rheumatoid arthritis. , 2000, Arthritis and rheumatism.
[35] S. Roberts-Thomson. Peroxisome proliferator‐activated receptors in tumorigenesis: Targets of tumour promotion and treatment , 2000, Immunology and cell biology.
[36] R. Moon,et al. WNTs modulate cell fate and behavior during vertebrate development. , 1997, Trends in genetics : TIG.
[37] N. Olsen,et al. Vascular permeability factor/endothelial growth factor (VPF/VEGF): accumulation and expression in human synovial fluids and rheumatoid synovial tissue , 1994, The Journal of experimental medicine.
[38] C. Werning. [Rheumatoid arthritis]. , 1983, Medizinische Monatsschrift fur Pharmazeuten.