Genome-wide association study of Alzheimer's disease
暂无分享,去创建一个
M A Pericak-Vance | O L Lopez | E Feingold | J. Haines | M. Pericak-Vance | V. Pankratz | A. Saykin | O. Lopez | S. DeKosky | L. Farrer | M. Barmada | M. Carrasquillo | S. Younkin | R. Sweet | M. Ganguli | Jacqueline Buros | G. Jun | C. Baldwin | M. Kamboh | E. Feingold | Xingbin Wang | F. Demirci | M. Logue | J. Haines | R. Minster | X. Wang | M Ganguli | S. Younkin | R A Sweet | V S Pankratz | A. Saykin | G Jun | M I Kamboh | S T DeKosky | M. Carrasquillo | C. Baldwin | J. Buros | L. Farrer | F Y Demirci | X Wang | R L Minster | M M Carrasquillo | S G Younkin | A J Saykin | C Baldwin | M W Logue | J Buros | L Farrer | J L Haines | M M Barmada | Kamboh Mi | J. Haines
[1] E. Peles,et al. Protein tyrosine kinase PYK2 involved in Ca2+-induced regulation of ion channel and MAP kinase functions , 1995, Nature.
[2] C. Ferri,et al. World Alzheimer Report 2009 , 2009 .
[3] A. J. Slater,et al. Candidate single-nucleotide polymorphisms from a genomewide association study of Alzheimer disease. , 2008, Archives of neurology.
[4] Ronald C. Petersen,et al. Genetic variation in PCDH11X is associated with susceptibility to late-onset Alzheimer's disease , 2009, Alzheimer's & Dementia.
[5] Christoph Lange,et al. Genome-wide association analysis reveals putative Alzheimer's disease susceptibility loci in addition to APOE. , 2008, American journal of human genetics.
[6] H. Brodaty,et al. ALZHEIMER'S DISEASE INTERNATIONAL , 1997, International journal of geriatric psychiatry.
[7] Eden R Martin,et al. Genome-wide association study implicates a chromosome 12 risk locus for late-onset Alzheimer disease. , 2009, American journal of human genetics.
[8] P. Gregersen,et al. Recommendations for publication of genetic association studies in Arthritis & Rheumatism. , 2011, Arthritis and rheumatism.
[9] Jason H. Moore,et al. Alzheimer's Disease Neuroimaging Initiative biomarkers as quantitative phenotypes: Genetics core aims, progress, and plans , 2010, Alzheimer's & Dementia.
[10] Nick C Fox,et al. Common variants in ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer’s disease , 2011, Nature Genetics.
[11] Sudha Seshadri,et al. Genome-wide analysis of genetic loci associated with Alzheimer disease. , 2010, JAMA.
[12] Nick C Fox,et al. Letter abstract - Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's Disease , 2009 .
[13] M. Albert,et al. Prevalence of Alzheimer's disease in a community population of older persons. Higher than previously reported. , 1989, JAMA.
[14] Sarah E. Brockwell,et al. A comparison of statistical methods for meta‐analysis , 2001, Statistics in medicine.
[15] D. Altman,et al. Measuring inconsistency in meta-analyses , 2003, BMJ : British Medical Journal.
[16] Sven Cichon,et al. A genome-wide association study for late-onset Alzheimer's disease using DNA pooling , 2008, BMC Medical Genomics.
[17] L. Kiemeney,et al. Corrigendum: Genetic variation in the prostate stem cell antigen gene PSCA confers susceptibility to urinary bladder cancer , 2009, Nature Genetics.
[18] Winnie S. Liang,et al. GAB2 Alleles Modify Alzheimer's Risk in APOE ɛ4 Carriers , 2007, Neuron.
[19] Nick C Fox,et al. Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease, and shows evidence for additional susceptibility genes , 2009, Nature Genetics.
[20] C. Jack,et al. Alzheimer's Disease Neuroimaging Initiative (ADNI) , 2010, Neurology.
[21] D. G. Clark,et al. Common variants in MS4A4/MS4A6E, CD2uAP, CD33, and EPHA1 are associated with late-onset Alzheimer’s disease , 2011, Nature Genetics.
[22] R. Hamman,et al. The relationship of APOE polymorphism and cholesterol levels in normoglycemic and diabetic subjects in a biethnic population from the San Luis Valley, Colorado. , 1995, Atherosclerosis.
[23] Inês Barroso,et al. Genetic Variants Influencing Circulating Lipid Levels and Risk of Coronary Artery Disease , 2010, Arteriosclerosis, thrombosis, and vascular biology.
[24] L. Fratiglioni,et al. Role of genes and environments for explaining Alzheimer disease. , 2006, Archives of general psychiatry.
[25] J. Morris,et al. Clinical core of the Alzheimer's disease neuroimaging initiative: Progress and plans , 2010, Alzheimer's & Dementia.
[26] Etienne Sibille,et al. Brain molecular aging, promotion of neurological disease and modulation by Sirtuin5 longevity gene polymorphism , 2011, Neurobiology of Disease.
[27] Rebecca F. Halperin,et al. A high-density whole-genome association study reveals that APOE is the major susceptibility gene for sporadic late-onset Alzheimer's disease. , 2007, The Journal of clinical psychiatry.
[28] Manuel A. R. Ferreira,et al. PLINK: a tool set for whole-genome association and population-based linkage analyses. , 2007, American journal of human genetics.
[29] M. Gill,et al. Evidence for novel susceptibility genes for late-onset Alzheimer's disease from a genome-wide association study of putative functional variants. , 2007, Human molecular genetics.
[30] Nick C Fox,et al. Genetic Variability in CLU and Its Association with Alzheimer's Disease , 2010, PloS one.
[31] Donald A. Wilson,et al. ApoE-Directed Therapeutics Rapidly Clear β-Amyloid and Reverse Deficits in AD Mouse Models , 2012, Science.
[32] Winnie S. Liang,et al. GAB2 alleles modify Alzheimer's risk in APOE epsilon4 carriers. , 2007, Neuron.
[33] Tanya M. Teslovich,et al. Biological, Clinical, and Population Relevance of 95 Loci for Blood Lipids , 2010, Nature.