Patterns of cerebral amyloid angiopathy define histopathological phenotypes in Alzheimer's disease
暂无分享,去创建一个
D. Mann | J. Snowden | A. Robinson | J Snowden | D M A Mann | N Allen | A C Robinson | Y S Davidson | Y. Davidson | D. Mann | N. Allen | Y. S. Davidson
[1] D. Mann,et al. Negative association between amyloid plaques and cerebral amyloid angiopathy in Alzheimer's disease , 2003, Neuroscience Letters.
[2] H. Soininen,et al. Alzheimer pathology of patients carrying apoliprotein E ϵ4 allele , 1995, Neurobiology of Aging.
[3] S. D. Preston,et al. Capillary and arterial cerebral amyloid angiopathy in Alzheimer's disease: defining the perivascular route for the elimination of amyloid β from the human brain , 2003, Neuropathology and applied neurobiology.
[4] Nick C Fox,et al. Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease, and shows evidence for additional susceptibility genes , 2009, Nature Genetics.
[5] H. Braak,et al. Phases of Aβ-deposition in the human brain and its relevance for the development of AD , 2002, Neurology.
[6] D. Andreu,et al. Lack of oestrogen protection in amyloid-mediated endothelial damage due to protein nitrotyrosination. , 2005, Brain : a journal of neurology.
[7] C. Holmes,et al. Neuropathology after active Aβ42 immunotherapy: implications for Alzheimer’s disease pathogenesis , 2010, Acta Neuropathologica.
[8] J. Vonsattel,et al. Cerebral amyloid angiopathy without and with cerebral hemorrhages: A comparative histological study , 1991, Annals of neurology.
[9] B. Hyman,et al. Apolipoprotein E epsilon 4 and cerebral hemorrhage associated with amyloid angiopathy. , 1995, Annals of neurology.
[10] T. Iwatsubo,et al. Visualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43) , 1994, Neuron.
[11] J. Bading,et al. Cerebrovascular Accumulation and Increased Blood‐Brain Barrier Permeability to Circulating Alzheimer's Amyloid β Peptide in Aged Squirrel Monkey with Cerebral Amyloid Angiopathy , 1998, Journal of neurochemistry.
[12] Nick C Fox,et al. Letter abstract - Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's Disease , 2009 .
[13] D. Dickson,et al. Extracellular Deposits of Aβ Produced in Cultures of Alzheimer Disease Brain Vascular Smooth Muscle Cells , 2005 .
[14] C. Jack,et al. Alzheimer's Disease Neuroimaging Initiative , 2008 .
[15] D. Mann,et al. Preferential deposition of amyloid β protein (Aβ) in the form Aβ 40 in Alzheimer's disease is associated with a gene dosage effect of the apolipoprotein E E4 allele , 1997, Neuroscience Letters.
[16] B T Hyman,et al. Senile dementia associated with amyloid beta protein angiopathy and tau perivascular pathology but not neuritic plaques in patients homozygous for the APOE-epsilon4 allele. , 2000, Acta neuropathologica.
[17] Julie S. Snowden,et al. Distinct Memory Profiles in Alzheimer's Disease , 2007, Cortex.
[18] T. Iwatsubo,et al. Visualization of A beta 42(43) and A beta 40 in senile plaques with end-specific A beta monoclonals: evidence that an initially deposited species is A beta 42(43). , 1994, Neuron.
[19] O. J. Dunn. Multiple Comparisons Using Rank Sums , 1964 .
[20] H. Braak,et al. Neuropathological stageing of Alzheimer-related changes , 2004, Acta Neuropathologica.
[21] J. Attems,et al. Capillary cerebral amyloid angiopathy identifies a distinct APOE ε4-associated subtype of sporadic Alzheimer’s disease , 2010, Acta Neuropathologica.
[22] Nick C Fox,et al. Common variants in ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer’s disease , 2011, Nature Genetics.
[23] K. Jellinger,et al. Review: Sporadic cerebral amyloid angiopathy , 2011, Neuropathology and applied neurobiology.
[24] H. Braak,et al. Two Types of Sporadic Cerebral Amyloid Angiopathy , 2002, Journal of neuropathology and experimental neurology.
[25] C. Behl. Oestrogen as a neuroprotective hormone , 2002, Nature Reviews Neuroscience.
[26] R N Kalaria,et al. Apolipoprotein E-epsilon4 alleles in cerebral amyloid angiopathy and cerebrovascular pathology associated with Alzheimer's disease. , 1996, The American journal of pathology.
[27] H. Soininen,et al. Alzheimer pathology of patients carrying apolipoprotein E epsilon 4 allele. , 1995, Neurobiology of aging.
[28] S. M. Sumi,et al. The Consortium to Establish a Registry for Alzheimer's Disease (CERAD) , 1991, Neurology.
[29] Joseph B Hiatt,et al. PI3K/AKT pathway mutations cause a spectrum of brain malformations from megalencephaly to focal cortical dysplasia. , 2015, Brain : a journal of neurology.
[30] B. Hyman,et al. Apolipoprotein E ϵ4 and cerebral hemorrhage associated with amyloid angiopathy , 1995 .
[31] S. Love,et al. APOEɛ4 influences the pathological phenotype of Alzheimer's disease by favouring cerebrovascular over parenchymal accumulation of Aβ protein , 2003 .
[32] J. Schneider,et al. National Institute on Aging–Alzheimer's Association guidelines for the neuropathologic assessment of Alzheimer's disease , 2012, Alzheimer's & Dementia.
[33] L. Thal,et al. Cerebral infarction in Alzheimer's disease is associated with severe amyloid angiopathy and hypertension. , 1995, Archives of neurology.
[34] Bradley T. Hyman,et al. Senile dementia associated with amyloid β protein angiopathy and tau perivascular pathology but not neuritic plaques in patients homozygous for the APOE-ε4 allele , 2000, Acta Neuropathologica.
[35] D. Graham,et al. The Apolipoprotein E ∈2 Allele and the Pathological Features in Cerebral Amyloid Angiopathy-related Hemorrhage , 1999 .
[36] D. Dickson,et al. Extracellular deposits of A beta produced in cultures of Alzheimer disease brain vascular smooth muscle cells. , 2005, Journal of neuropathology and experimental neurology.
[37] B. Hyman,et al. Apolipoprotein E in sporadic Alzheimer's disease: Allelic variation and receptor interactions , 1993, Neuron.
[38] Cornelia M van Duijn,et al. Dense-core senile plaques in the Flemish variant of Alzheimer's disease are vasocentric. , 2002, The American journal of pathology.
[39] P. Eikelenboom,et al. Characteristics of Dyshoric Capillary Cerebral Amyloid Angiopathy , 2010, Journal of neuropathology and experimental neurology.
[40] D. Mann,et al. The prevalence of amyloid (A4) protein deposits within the cerebral and cerebellar cortex in Down's syndrome and Alzheimer's disease , 2004, Acta Neuropathologica.
[41] K. Jellinger,et al. Alzheimer’s disease pathology influences severity and topographical distribution of cerebral amyloid angiopathy , 2005, Acta Neuropathologica.
[42] PhilipR. Wenham,et al. Apolipoprotein E genotyping by one-stage PCR , 1991, The Lancet.