Hyperparathyroidism in a patient with autoimmune polyglandular syndrome.
暂无分享,去创建一个
[1] P. Vestergaard,et al. Discriminative power of three indices of renal calcium excretion for the distinction between familial hypocalciuric hypercalcaemia and primary hyperparathyroidism: a follow‐up study on methods , 2008, Clinical endocrinology.
[2] L. Peltonen,et al. Autoimmune polyendocrine syndrome type 1 and NALP5, a parathyroid autoantigen. , 2008, The New England journal of medicine.
[3] T. Fujita,et al. An acquired hypocalciuric hypercalcemia autoantibody induces allosteric transition among active human Ca-sensing receptor conformations , 2007, Proceedings of the National Academy of Sciences.
[4] E. Brown,et al. The calcium-sensing receptor is a target of autoantibodies in patients with autoimmune polyendocrine syndrome type 1. , 2007, The Journal of clinical endocrinology and metabolism.
[5] W. Taylor,et al. Prevalence of glucose intolerance in primary hyperparathyroidism and the benefit of parathyroidectomy , 2007, Diabetes/metabolism research and reviews.
[6] M. Rubin,et al. Asymptomatic primary hyperparathyroidism. , 2006, Arquivos brasileiros de endocrinologia e metabologia.
[7] J. Barker. Clinical review: Type 1 diabetes-associated autoimmunity: natural history, genetic associations, and screening. , 2006, The Journal of clinical endocrinology and metabolism.
[8] J. Bilezikian,et al. Rare causes of hypercalcemia , 2005 .
[9] H. Vidal,et al. Calcium-sensing receptor autoantibodies are relevant markers of acquired hypoparathyroidism. , 2004, The Journal of clinical endocrinology and metabolism.
[10] E. Brown,et al. Acquired hypocalciuric hypercalcemia due to autoantibodies against the calcium-sensing receptor. , 2004, The New England journal of medicine.
[11] S. Silverberg,et al. Clinical practice. Asymptomatic primary hyperparathyroidism. , 2004, The New England journal of medicine.
[12] R. Thakker. Diseases associated with the extracellular calcium-sensing receptor. , 2004, Cell calcium.
[13] E. Brown,et al. Activating antibodies to the calcium-sensing receptor in two patients with autoimmune hypoparathyroidism. , 2004, The Journal of clinical endocrinology and metabolism.
[14] J. Garber,et al. A syndrome of hypocalciuric hypercalcemia caused by autoantibodies directed at the calcium-sensing receptor. , 2003, The Journal of clinical endocrinology and metabolism.
[15] J. Rastad,et al. Summary statement from a workshop on asymptomatic primary hyperparathyroidism: a perspective for the 21st century. , 2002, The Journal of clinical endocrinology and metabolism.
[16] P. Gonzalo,et al. Autoantibodies directed against the ribosomal P proteins are not only directed against a common epitope of the P0, P1 and P2 proteins. , 1999, Journal of autoimmunity.
[17] W. Taylor,et al. Prevalence of Primary Hyperparathyroidism in Patients with Diabetes Mellitus , 1997, Diabetic medicine : a journal of the British Diabetic Association.
[18] Y. Li,et al. Autoantibodies to the extracellular domain of the calcium sensing receptor in patients with acquired hypoparathyroidism. , 1996, The Journal of clinical investigation.
[19] J. Seidman,et al. Mutations in the human Ca2+-sensing receptor gene cause familial hypocalciuric hypercalcemia and neonatal severe hyperparathyroidism , 1993, Cell.
[20] C. Boitard,et al. Age-dependent HLA genetic heterogeneity of type 1 insulin-dependent diabetes mellitus. , 1992, The Journal of clinical investigation.
[21] J. Seidman,et al. The gene responsible for familial hypocalciuric hypercalcemia maps to chromosome 3q in four unrelated families , 1992, Nature Genetics.