Rewiring of regenerated axons by combining treadmill training with semaphorin3A inhibition
暂无分享,去创建一个
H. Okano | Meigen Liu | Y. Toyama | S. Kaneko | A. Sano | M. Maeda | S. Shibata | Liang Zhang | K. Kikuchi | A. Kishino | Toru Kimura | M. Nakamura | Masahiko Mukaino | Shinjiro Kaneko
[1] H. Okano,et al. Rewiring of regenerated axons by combining treadmill training with semaphorin3A inhibition , 2014, Molecular Brain.
[2] J. Fawcett,et al. Chondroitinase Combined with Rehabilitation Promotes Recovery of Forelimb Function in Rats with Chronic Spinal Cord Injury , 2011, The Journal of Neuroscience.
[3] P. Ravassard,et al. Class 3 semaphorins influence oligodendrocyte precursor recruitment and remyelination in adult central nervous system. , 2011, Brain : a journal of neurology.
[4] K. Nave. Myelination and support of axonal integrity by glia , 2010, Nature.
[5] J. Fawcett,et al. Extrinsic and intrinsic factors controlling axonal regeneration after spinal cord injury , 2009, Expert Reviews in Molecular Medicine.
[6] Igor A. Lavrov,et al. Transformation of nonfunctional spinal circuits into functional states after the loss of brain input , 2009, Nature Neuroscience.
[7] J. Fawcett,et al. Chondroitinase ABC treatment opens a window of opportunity for task-specific rehabilitation , 2009, Nature Neuroscience.
[8] M. Hickey,et al. Molecular mechanisms of leukocyte trafficking in T-cell-mediated skin inflammation: insights from intravital imaging , 2009, Expert Reviews in Molecular Medicine.
[9] V Reggie Edgerton,et al. Differential effects of anti-Nogo-A antibody treatment and treadmill training in rats with incomplete spinal cord injury. , 2009, Brain : a journal of neurology.
[10] Ray D. de Leon,et al. Treadmill training enhances the recovery of normal stepping patterns in spinal cord contused rats , 2009, Experimental Neurology.
[11] Sarah A. Dunlop,et al. Activity-dependent plasticity: implications for recovery after spinal cord injury , 2008, Trends in Neurosciences.
[12] Hui Zhong,et al. Step Training Reinforces Specific Spinal Locomotor Circuitry in Adult Spinal Rats , 2008, The Journal of Neuroscience.
[13] D. Cheresh,et al. Semaphorin 3A suppresses VEGF-mediated angiogenesis yet acts as a vascular permeability factor. , 2008, Blood.
[14] H. Okano,et al. Hepatocyte growth factor promotes endogenous repair and functional recovery after spinal cord injury , 2007, Journal of neuroscience research.
[15] V. Perciavalle,et al. Synaptic plasticity modulates the spontaneous recovery of locomotion after spinal cord hemisection , 2007, Neuroscience Research.
[16] H. Okano,et al. A selective Sema3A inhibitor enhances regenerative responses and functional recovery of the injured spinal cord , 2006, Nature Medicine.
[17] V. Edgerton,et al. Use of c-fos to identify activity-dependent spinal neurons after stepping in intact adult rats , 2006, Spinal Cord.
[18] R. D. de Leon,et al. Effect of robotic-assisted treadmill training and chronic quipazine treatment on hindlimb stepping in spinally transected rats. , 2006, Journal of neurotrauma.
[19] O. Kiehn. Locomotor circuits in the mammalian spinal cord. , 2006, Annual review of neuroscience.
[20] T. Yamashita,et al. RGMa inhibition promotes axonal growth and recovery after spinal cord injury , 2006, The Journal of cell biology.
[21] R. Franco-Bourland,et al. Glutathione monoethyl ester improves functional recovery, enhances neuron survival, and stabilizes spinal cord blood flow after spinal cord injury in rats , 2005, Neuroscience.
[22] J. Williamson,et al. Changes in Supraspinal Activation Patterns following Robotic Locomotor Therapy in Motor-Incomplete Spinal Cord Injury , 2005, Neurorehabilitation and neural repair.
[23] M. Merlo,et al. A robotic device for studying rodent locomotion after spinal cord injury , 2005, IEEE Transactions on Neural Systems and Rehabilitation Engineering.
[24] V. Edgerton,et al. Hindlimb stepping movements in complete spinal rats induced by epidural spinal cord stimulation , 2005, Neuroscience Letters.
[25] V. Edgerton,et al. L1 CAM expression is increased surrounding the lesion site in rats with complete spinal cord transection as neonates , 2005, Experimental Neurology.
[26] David J. Reinkensmeyer,et al. Hindlimb loading determines stepping quantity and quality following spinal cord transection , 2005, Brain Research.
[27] V. Edgerton,et al. Exercise restores levels of neurotrophins and synaptic plasticity following spinal cord injury , 2005, Experimental Neurology.
[28] J. Fawcett,et al. The ability of axons to regenerate their growth cones depends on axonal type and age, and is regulated by calcium, cAMP and ERK , 2005, The European journal of neuroscience.
[29] J. Schwab,et al. Spinal cord injury‐induced lesional expression of the repulsive guidance molecule (RGM) , 2005, The European journal of neuroscience.
[30] Zhigang He,et al. The Nogo signaling pathway for regeneration block. , 2004, Annual review of neuroscience.
[31] V. Edgerton,et al. Plasticity of the spinal neural circuitry after injury. , 2004, Annual review of neuroscience.
[32] Martin E Schwab,et al. The injured spinal cord spontaneously forms a new intraspinal circuit in adult rats , 2004, Nature Neuroscience.
[33] Jerry Silver,et al. Regeneration beyond the glial scar , 2004, Nature Reviews Neuroscience.
[34] M. Filbin,et al. Myelin-associated inhibitors of axonal regeneration in the adult mammalian CNS , 2003, Nature Reviews Neuroscience.
[35] K. Kikuchi,et al. In Vitro and in Vivo Characterization of a Novel Semaphorin 3A Inhibitor, SM-216289 or Xanthofulvin* , 2003, Journal of Biological Chemistry.
[36] M. Schwab,et al. Systemic Deletion of the Myelin-Associated Outgrowth Inhibitor Nogo-A Improves Regenerative and Plastic Responses after Spinal Cord Injury , 2003, Neuron.
[37] S. Strittmatter,et al. Axon Regeneration in Young Adult Mice Lacking Nogo-A/B , 2003, Neuron.
[38] O. Steward,et al. Lack of Enhanced Spinal Regeneration in Nogo-Deficient Mice , 2003, Neuron.
[39] J. Gossard,et al. Spinal Cats on the Treadmill: Changes in Load Pathways , 2003, The Journal of Neuroscience.
[40] Zhigang He,et al. Oligodendrocyte-myelin glycoprotein is a Nogo receptor ligand that inhibits neurite outgrowth , 2002, Nature.
[41] D. Burke,et al. Temporal progression of angiogenesis and basal lamina deposition after contusive spinal cord injury in the adult rat , 2002, The Journal of comparative neurology.
[42] James W. Fawcett,et al. Chondroitinase ABC promotes functional recovery after spinal cord injury , 2002, Nature.
[43] P. Wood,et al. New Vascular Tissue Rapidly Replaces Neural Parenchyma and Vessels Destroyed by a Contusion Injury to the Rat Spinal Cord , 2002, Experimental Neurology.
[44] Martin E. Schwab,et al. Plasticity of motor systems after incomplete spinal cord injury , 2001, Nature Reviews Neuroscience.
[45] L. Jordan,et al. Spinal cholinergic neurons activated during locomotion: localization and electrophysiological characterization. , 2000, Journal of neurophysiology.
[46] Fumio Nakamura,et al. Identification of the Nogo inhibitor of axon regeneration as a Reticulon protein , 2000, Nature.
[47] J. Silver,et al. Robust Regeneration of Adult Sensory Axons in Degenerating White Matter of the Adult Rat Spinal Cord , 1999, The Journal of Neuroscience.
[48] M. Ruitenberg,et al. Expression of the Gene Encoding the Chemorepellent Semaphorin III Is Induced in the Fibroblast Component of Neural Scar Tissue Formed Following Injuries of Adult But Not Neonatal CNS , 1999, Molecular and Cellular Neuroscience.
[49] O. Kiehn,et al. Distribution of Central Pattern Generators for Rhythmic Motor Outputs in the Spinal Cord of Limbed Vertebrates a , 1998, Annals of the New York Academy of Sciences.
[50] T. Yagi,et al. Disruption of Semaphorin III/D Gene Causes Severe Abnormality in Peripheral Nerve Projection , 1997, Neuron.
[51] Alex L Kolodkin,et al. Neuropilin Is a Semaphorin III Receptor , 1997, Cell.
[52] M. Schwab,et al. Local Changes in Vascular Architecture Following Partial Spinal Cord Lesion in the Rat , 1997, Experimental Neurology.
[53] A. Yakovleff,et al. Recovery of locomotor activity in the adult chronic spinal rat after sublesional transplantation of embryonic nervous cells: specific role of serotonergic neurons , 1997, Experimental Brain Research.
[54] O. Kiehn,et al. Spatiotemporal characteristics of 5-HT and dopamine-induced rhythmic hindlimb activity in the in vitro neonatal rat. , 1996, Journal of neurophysiology.
[55] L. Mckerracher,et al. Identification of myelin-associated glycoprotein as a major myelin-derived inhibitor of neurite growth , 1994, Neuron.
[56] J. Silver,et al. Reduction of neurite outgrowth in a model of glial scarring following CNS injury is correlated with the expression of inhibitory molecules on reactive astrocytes , 1991, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[57] S. Rossignol,et al. Recovery of locomotion after chronic spinalization in the adult cat , 1987, Brain Research.
[58] G. Grant,et al. The cytoarchitectonic organization of the spinal cord in the rat. I. The lower thoracic and lumbosacral cord , 1984, The Journal of comparative neurology.
[59] B. Rexed. The cytoarchitectonic organization of the spinal cord in the cat , 1952, The Journal of comparative neurology.
[60] Devin L Jindrich,et al. OEG implantation and step training enhance hindlimb-stepping ability in adult spinal transected rats. , 2008, Brain : a journal of neurology.
[61] Bingbing Song,et al. Recovery of supraspinal control of stepping via indirect propriospinal relay connections after spinal cord injury , 2008, Nature Medicine.
[62] A. Holtmaat,et al. Neuropilin and class 3 semaphorins in nervous system regeneration. , 2002, Advances in experimental medicine and biology.