Pressure-Induced Matrix Metalloproteinase-9 Contributes to Early Hypertensive Remodeling
暂无分享,去创建一个
A. Tedgui | H. Lijnen | B. Esposito | S. Lehoux | C. Lemarié
[1] Z. Galis,et al. Matrix Metalloproteinase-2 and −9 Differentially Regulate Smooth Muscle Cell Migration and Cell-Mediated Collagen Organization , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[2] Robert M. Nerem,et al. The Role of Matrix Metalloproteinase-2 in the Remodeling of Cell-Seeded Vascular Constructs Subjected to Cyclic Strain , 2001, Annals of Biomedical Engineering.
[3] A. Tedgui,et al. Pressure-Induced Vascular Activation of Nuclear Factor-&kgr;B: Role in Cell Survival , 2003, Circulation research.
[4] Steven M. Holland,et al. Mechanical Stretch Enhances mRNA Expression and Proenzyme Release of Matrix Metalloproteinase‐2 (MMP‐2) via NAD(P)H Oxidase‐Derived Reactive Oxygen Species , 2003, Circulation research.
[5] Chad Johnson,et al. Uniaxial strain upregulates matrix-degrading enzymes produced by human vascular smooth muscle cells. , 2003, American journal of physiology. Heart and circulatory physiology.
[6] P. Toutouzas,et al. Plasma levels of active extracellular matrix metalloproteinases 2 and 9 in patients with essential hypertension before and after antihypertensive treatment , 2003, Journal of Human Hypertension.
[7] B. Lévy,et al. Excessive Microvascular Adaptation to Changes in Blood Flow in Mice Lacking Gene Encoding for Desmin , 2002, Arteriosclerosis, thrombosis, and vascular biology.
[8] Avrum I. Gotlieb,et al. Wall Tissue Remodeling Regulates Longitudinal Tension in Arteries , 2002, Circulation research.
[9] Richard T. Lee,et al. Mechanical Strain Induces Specific Changes in the Synthesis and Organization of Proteoglycans by Vascular Smooth Muscle Cells* , 2001, The Journal of Biological Chemistry.
[10] Christopher J. O’Callaghan,et al. Mechanical Strain–Induced Extracellular Matrix Production by Human Vascular Smooth Muscle Cells: Role of TGF-&bgr;1 , 2000, Hypertension.
[11] G. Lip,et al. Matrix metalloproteinase-9 and tissue inhibitor metalloproteinase-1 levels in essential hypertension. Relationship to left ventricular mass and anti-hypertensive therapy. , 2000, International journal of cardiology.
[12] D. Ku,et al. Early effects of arterial hemodynamic conditions on human saphenous veins perfused ex vivo. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[13] D. Ku,et al. Transmural pressure induces matrix-degrading activity in porcine arteries ex vivo. , 1999, The American journal of physiology.
[14] D. Ku,et al. Transmural pressure induces matrix-degrading activity in porcine arteries ex vivo. , 1999, American journal of physiology. Heart and circulatory physiology.
[15] Z. Galis,et al. Mechanical stretching of human saphenous vein grafts induces expression and activation of matrix-degrading enzymes associated with vascular tissue injury and repair. , 1999, Experimental and molecular pathology.
[16] B. L. Langille,et al. Atrophic remodeling of the artery-cuffed artery. , 1999, Arteriosclerosis, thrombosis, and vascular biology.
[17] K. Birukov,et al. Intraluminal pressure is essential for the maintenance of smooth muscle caldesmon and filamin content in aortic organ culture. , 1998, Arteriosclerosis, thrombosis, and vascular biology.
[18] P. Libby,et al. Small Mechanical Strains Selectively Suppress Matrix Metalloproteinase-1 Expression by Human Vascular Smooth Muscle Cells* , 1998, Journal of Biological Chemistry.
[19] A. Tedgui,et al. Pressure and angiotensin II synergistically induce aortic fibronectin expression in organ culture model of rabbit aorta. Evidence for a pressure-induced tissue renin-angiotensin system. , 1996, Circulation research.