PET "reversed mismatch pattern" early after acute myocardial infarction: follow-up of flow, metabolism and function

[1]  D. Le Guludec,et al.  Relationship between resting 201Tl reverse redistribution, microvascular perfusion, and functional recovery in acute myocardial infarction. , 2000, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[2]  K. Hirata,et al.  A reverse flow-metabolism mismatch pattern on PET is related to multivessel disease in patients with acute myocardial infarction. , 1999, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[3]  M. Schwaiger,et al.  Reverse flow-metabolism mismatch: what does it mean? , 1999, Journal of Nuclear Medicine.

[4]  F. Van de Werf,et al.  Importance of flow/metabolism studies in predicting late recovery of function following reperfusion in patients with acute myocardial infarction. , 1997, European heart journal.

[5]  F. Van de Werf,et al.  Impaired myocardial tissue perfusion early after successful thrombolysis. Impact on myocardial flow, metabolism, and function at late follow-up. , 1995, Circulation.

[6]  L. Barrios,et al.  A randomized trial of recombinant staphylokinase versus alteplase for coronary artery patency in acute myocardial infarction. The STAR Trial Group. , 1995, Circulation.

[7]  J. Nuyts,et al.  Histological Alterations in Chronically Hypoperfused Myocardium: Correlation With PET Findings , 1994, Circulation.

[8]  V. Dilsizian,et al.  Clinical significance of reduced regional myocardial glucose uptake in regions with normal blood flow in patients with chronic coronary artery disease. , 1994, Journal of the American College of Cardiology.

[9]  P. Wouters,et al.  Measurement of organ blood flow with coloured microspheres: a first time-saving improvement using automated spectrophotometry , 1993, Proceedings of Computers in Cardiology Conference.

[10]  U. Ruotsalainen,et al.  Euglycemic hyperinsulinemic clamp and oral glucose load in stimulating myocardial glucose utilization during positron emission tomography. , 1992, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[11]  B. Sobel,et al.  Dependence of recovery of contractile function on maintenance of oxidative metabolism after myocardial infarction. , 1992, Journal of the American College of Cardiology.

[12]  C. Patlak,et al.  Graphical Evaluation of Blood-to-Brain Transfer Constants from Multiple-Time Uptake Data. Generalizations , 1985, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[13]  C S Patlak,et al.  Graphical Evaluation of Blood-to-Brain Transfer Constants from Multiple-Time Uptake Data , 1983, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[14]  R. DeFronzo,et al.  Glucose clamp technique: a method for quantifying insulin secretion and resistance. , 1979, The American journal of physiology.

[15]  O Muzik,et al.  Validation of nitrogen-13-ammonia tracer kinetic model for quantification of myocardial blood flow using PET. , 1993, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.

[16]  Frans Van de Werf,et al.  An international randomized trial comparing four thrombolytic strategies for acute myocardial infarction. , 1993, The New England journal of medicine.

[17]  P Suetens,et al.  Delineation of ECT images using global constraints and dynamic programming. , 1991, IEEE transactions on medical imaging.

[18]  H. S. Mueller,et al.  The Thrombolysis in Myocardial Infarction (TIMI) trial. Phase I findings. , 1985, The New England journal of medicine.