Nitric oxide and pathogenic mechanisms involved in the development of vascular diseases
暂无分享,去创建一个
[1] Luo Yu-yu. Role of Krüppel-like transcription factors in endothelial biology , 2009 .
[2] L. Lerman,et al. The Interaction Between Coronary Endothelial Dysfunction, Local Oxidative Stress, and Endogenous Nitric Oxide in Humans , 2008, Hypertension.
[3] M. Bouhlel,et al. Peroxisome proliferator‐activated receptors – from active regulators of macrophage biology to pharmacological targets in the treatment of cardiovascular disease , 2007, Journal of internal medicine.
[4] D. Harrison,et al. Molecular Mechanisms of Angiotensin II–Mediated Mitochondrial Dysfunction: Linking Mitochondrial Oxidative Damage and Vascular Endothelial Dysfunction , 2007, Circulation research.
[5] Paul L Huang,et al. Cardiovascular roles of nitric oxide: a review of insights from nitric oxide synthase gene disrupted mice. , 2007, Cardiovascular research.
[6] E. Schiffrin. Oxidative stress, nitric oxide synthase, and superoxide dismutase: a matter of imbalance underlies endothelial dysfunction in the human coronary circulation. , 2008, Hypertension.
[7] M. Toborek,et al. The role of caveolin-1 in PCB77-induced eNOS phosphorylation in human-derived endothelial cells. , 2007, American journal of physiology. Heart and circulatory physiology.
[8] Johan Kuiper,et al. Prolonged shear stress and KLF2 suppress constitutive proinflammatory transcription through inhibition of ATF2. , 2007, Blood.
[9] J. Kuiper,et al. KLF2 Suppresses TGF-&bgr; Signaling in Endothelium Through Induction of Smad7 and Inhibition of AP-1 , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[10] L. Ignarro,et al. High glucose downregulates the number of caveolae in monocytes through oxidative stress from NADPH oxidase: implications for atherosclerosis. , 2007, Biochimica et biophysica acta.
[11] C. Napoli,et al. Polymorphisms in endothelial nitric oxide synthase and carotid artery atherosclerosis , 2006, Journal of Clinical Pathology.
[12] C. Napoli,et al. Nitric oxide and atherosclerosis: an update. , 2006, Nitric oxide : biology and chemistry.
[13] N. Maniatis,et al. Novel Mechanism of Endothelial Nitric Oxide Synthase Activation Mediated by Caveolae Internalization in Endothelial Cells , 2006, Circulation research.
[14] M. Rondaij,et al. KLF2 provokes a gene expression pattern that establishes functional quiescent differentiation of the endothelium. , 2006, Blood.
[15] Yuzhi Zhang,et al. Integration of flow-dependent endothelial phenotypes by Kruppel-like factor 2. , 2005, The Journal of clinical investigation.
[16] T. Rabelink,et al. Endothelial nitric oxide synthase: host defense enzyme of the endothelium? , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[17] P. Reaven,et al. Peroxisome Proliferator-Activated Receptor-Alpha and Atherosclerosis: From Basic Mechanisms to Clinical Implications , 2005, Cardiology.
[18] Jurgen Seppen,et al. Endothelial KLF2 links local arterial shear stress levels to the expression of vascular tone-regulating genes. , 2005, The American journal of pathology.
[19] G. Garcı́a-Cardeña,et al. Statins Exert Endothelial Atheroprotective Effects via the KLF2 Transcription Factor* , 2005, Journal of Biological Chemistry.
[20] G. Garcı́a-Cardeña,et al. Kruppel-Like Factor 2 (KLF2) Regulates Endothelial Thrombotic Function , 2005, Circulation research.
[21] C. Napoli,et al. Novel features of nitric oxide, endothelial nitric oxide synthase, and atherosclerosis , 2005, Current atherosclerosis reports.
[22] F. Luscinskas,et al. KLF2 Is a Novel Transcriptional Regulator of Endothelial Proinflammatory Activation , 2004, The Journal of experimental medicine.
[23] D. Eichler,et al. Argininosuccinate Synthase Expression Is Required to Maintain Nitric Oxide Production and Cell Viability in Aortic Endothelial Cells* , 2004, Journal of Biological Chemistry.
[24] Xin Xu,et al. Peroxisome Proliferator-Activated Receptor &agr; Agonists Increase Nitric Oxide Synthase Expression in Vascular Endothelial Cells , 2004 .
[25] C. Napoli,et al. Nitric oxide-releasing drugs. , 2003, Annual review of pharmacology and toxicology.
[26] C. Napoli,et al. Oxidation-sensitive mechanisms, vascular apoptosis and atherosclerosis. , 2003, Trends in molecular medicine.
[27] V. Shah,et al. The Proline-rich Domain of Dynamin-2 Is Responsible for Dynamin-dependent in Vitro Potentiation of Endothelial Nitric-oxide Synthase Activity via Selective Effects on Reductase Domain Function* , 2003, The Journal of Biological Chemistry.
[28] J. Loscalzo,et al. Glycoxidized low-density lipoprotein downregulates endothelial nitricoxide synthase in human coronary cells. , 2002, Journal of the American College of Cardiology.
[29] J. Loscalzo,et al. Nitric oxide donors and cardiovascular agents modulating the bioactivity of nitric oxide: an overview. , 2002, Circulation research.
[30] C. Napoli,et al. Age-Related Effects on Atherogenesis and Scavenger Enzymes of Intracranial and Extracranial Arteries in Men Without Classic Risk Factors for Atherosclerosis , 2001, Stroke.
[31] C. Napoli,et al. Involvement of oxidation-sensitive mechanisms in the cardiovascular effects of hypercholesterolemia. , 2001, Mayo Clinic proceedings.
[32] C. Napoli,et al. Nitric oxide and atherosclerosis. , 2001, Nitric oxide : biology and chemistry.
[33] C. Napoli,et al. Nitric oxide as a signaling molecule in the vascular system: an overview. , 1999, Journal of cardiovascular pharmacology.
[34] C. Napoli,et al. Intracranial arteries of human fetuses are more resistant to hypercholesterolemia-induced fatty streak formation than extracranial arteries. , 1999, Circulation.
[35] Roger Fan,et al. Dynamic activation of endothelial nitric oxide synthase by Hsp90 , 1998, Nature.
[36] C. Napoli,et al. Mildly oxidized low-density lipoprotein impairs responses of carotid but not basilar artery in rabbits. , 1997, Stroke.