Graded bidirectional synaptic plasticity is composed of switch-like unitary events.
暂无分享,去创建一个
[1] A. Hodgkin,et al. A quantitative description of membrane current and its application to conduction and excitation in nerve , 1952, The Journal of physiology.
[2] T. Bliss,et al. Long‐lasting potentiation of synaptic transmission in the dentate area of the anaesthetized rabbit following stimulation of the perforant path , 1973, The Journal of physiology.
[3] E. Oja. Simplified neuron model as a principal component analyzer , 1982, Journal of mathematical biology.
[4] J J Hopfield,et al. Neural networks and physical systems with emergent collective computational abilities. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[5] E. Bienenstock,et al. Theory for the development of neuron selectivity: orientation specificity and binocular interaction in visual cortex , 1982, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[6] T. H. Brown,et al. Associative long-term potentiation in hippocampal slices. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[7] C. Stevens,et al. Presynaptic mechanism for long-term potentiation in the hippocampus , 1990, Nature.
[8] R. Tsien,et al. Presynaptic enhancement shown by whole-cell recordings of long-term potentiation in hippocampal slices , 1990, Nature.
[9] M. Bear,et al. Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[10] R. Malenka,et al. Mechanisms underlying induction of homosynaptic long-term depression in area CA1 of the hippocampus , 1992, Neuron.
[11] T. Bliss,et al. A synaptic model of memory: long-term potentiation in the hippocampus , 1993, Nature.
[12] K M Harris,et al. Occurrence and three-dimensional structure of multiple synapses between individual radiatum axons and their target pyramidal cells in hippocampal area CA1 , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[13] H. Wigström,et al. Long‐term Depression in the Hippocampal CA1 Region is Associated with Equal Changes in AMPA and NMDA Receptor‐mediated Synaptic Potentials , 1994, The European journal of neuroscience.
[14] J. Isaac,et al. Evidence for silent synapses: Implications for the expression of LTP , 1995, Neuron.
[15] R. Malinow,et al. Activation of postsynaptically silent synapses during pairing-induced LTP in CA1 region of hippocampal slice , 1995, Nature.
[16] Morten Raastad,et al. Extracellular Activation of Unitary Excitatory Synapses Between Hippocampal CA3 and CA1 Pyramidal Cells , 1995, The European journal of neuroscience.
[17] J. Isaac,et al. Long-term potentiation at single fiber inputs to hippocampal CA1 pyramidal cells. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[18] D. Johnston,et al. The role of dendritic action potentials and Ca2+ influx in the induction of homosynaptic long-term depression in hippocampal CA1 pyramidal neurons. , 1996, Learning & memory.
[19] C. Stevens,et al. Heterogeneity of Release Probability, Facilitation, and Depletion at Central Synapses , 1997, Neuron.
[20] J J Jack,et al. Quantal analysis of excitatory synapses in rat hippocampal CA1 In Vitro during low‐frequency depression , 1997, The Journal of physiology.
[21] H. Markram,et al. Regulation of Synaptic Efficacy by Coincidence of Postsynaptic APs and EPSPs , 1997, Science.
[22] D E Koshland,et al. The Era of Pathway Quantification , 1998, Science.
[23] J. Hopfield,et al. All-or-none potentiation at CA3-CA1 synapses. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[24] J E Ferrell,et al. The biochemical basis of an all-or-none cell fate switch in Xenopus oocytes. , 1998, Science.
[25] R. Nicoll,et al. Development of excitatory circuitry in the hippocampus. , 1998, Journal of neurophysiology.
[26] D. Debanne,et al. Heterogeneity of Synaptic Plasticity at Unitary CA3–CA1 and CA3–CA3 Connections in Rat Hippocampal Slice Cultures , 1999, The Journal of Neuroscience.
[27] R. Zucker,et al. Selective induction of LTP and LTD by postsynaptic [Ca2+]i elevation. , 1999, Journal of neurophysiology.
[28] R. Nicoll,et al. Long-term potentiation--a decade of progress? , 1999, Science.
[29] K. Svoboda,et al. Rapid spine delivery and redistribution of AMPA receptors after synaptic NMDA receptor activation. , 1999, Science.
[30] J. Sanes,et al. Can molecules explain long-term potentiation? , 1999, Nature Neuroscience.
[31] M. Ehlers,et al. Reinsertion or Degradation of AMPA Receptors Determined by Activity-Dependent Endocytic Sorting , 2000, Neuron.
[32] S. Leibler,et al. An ultrasensitive bacterial motor revealed by monitoring signaling proteins in single cells. , 2000, Science.
[33] M. Bear,et al. Regulation of distinct AMPA receptor phosphorylation sites during bidirectional synaptic plasticity , 2000, Nature.
[34] D. Wilkin,et al. Neuron , 2001, Brain Research.
[35] J. Lisman,et al. A Model of Synaptic Memory A CaMKII/PP1 Switch that Potentiates Transmission by Organizing an AMPA Receptor Anchoring Assembly , 2001, Neuron.
[36] J. Montgomery,et al. State-Dependent Heterogeneity in Synaptic Depression between Pyramidal Cell Pairs , 2002, Neuron.
[37] W. Regehr,et al. Short-term synaptic plasticity. , 2002, Annual review of physiology.
[38] G. Collingridge,et al. Multiple, Developmentally Regulated Expression Mechanisms of Long-Term Potentiation at CA1 Synapses , 2004, The Journal of Neuroscience.
[39] G. Ellis‐Davies,et al. Structural basis of long-term potentiation in single dendritic spines , 2004, Nature.
[40] B. Gustafsson,et al. Creation of AMPA-silent synapses in the neonatal hippocampus , 2004, Nature Neuroscience.