Increased indoleamine 2,3-dioxygenase activity in type 2 diabetic nephropathy.

[1]  J. Egido,et al.  Tubular overexpression of Gremlin in transgenic mice aggravates renal damage in diabetic nephropathy. , 2015, American journal of physiology. Renal physiology.

[2]  P. Peduzzi,et al.  Combined angiotensin inhibition for the treatment of diabetic nephropathy. , 2013, The New England journal of medicine.

[3]  M. Malbrain,et al.  Changes in Plasma Kynurenic Acid Concentration in Septic Shock Patients Undergoing Continuous Veno-Venous Haemofiltration , 2013, Inflammation.

[4]  B. Baban,et al.  Endoplasmic reticulum stress response and inflammatory cytokines in type 2 diabetic nephropathy: role of indoleamine 2,3-dioxygenase and programmed death-1. , 2013, Experimental and molecular pathology.

[5]  H. Makino,et al.  Inflammation and the pathogenesis of diabetic nephropathy. , 2013, Clinical science.

[6]  Rahul Shinde,et al.  Tolerance to apoptotic cells is regulated by indoleamine 2,3-dioxygenase , 2012, Proceedings of the National Academy of Sciences.

[7]  T. Eleftheriadis,et al.  Indoleamine 2,3-dioxygenase is increased in hemodialysis patients and affects immune response to hepatitis B vaccination. , 2011, Vaccine.

[8]  N. Su,et al.  ATF4-dependent transcription mediates signaling of amino acid limitation , 2009, Trends in Endocrinology & Metabolism.

[9]  H. Volk,et al.  Increased indoleamine 2,3-dioxygenase (IDO) activity and elevated serum levels of tryptophan catabolites in patients with chronic kidney disease: a possible link between chronic inflammation and uraemic symptoms. , 2009, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[10]  A. Jevnikar,et al.  Indoleamine 2,3-dioxygenase expression promotes renal ischemia-reperfusion injury. , 2008, American journal of physiology. Renal physiology.

[11]  H. Parving,et al.  Effect of a Multifactorial Intervention on Mortality in Type II Diabetes , 2008 .

[12]  U. Grohmann,et al.  IDO and regulatory T cells: a role for reverse signalling and non-canonical NF-κB activation , 2007, Nature Reviews Immunology.

[13]  D. Larkin,et al.  Function of indoleamine 2,3‐dioxygenase in corneal allograft rejection and prolongation of allograft survival by over‐expression , 2006, European journal of immunology.

[14]  B. Baban,et al.  GCN2 kinase in T cells mediates proliferative arrest and anergy induction in response to indoleamine 2,3-dioxygenase. , 2005, Immunity.

[15]  SalimYusuf,et al.  Relationship of Anti-60 kDa Heat Shock Protein and Anti-Cholesterol Antibodies to Cardiovascular Events , 2002 .

[16]  O. Pedersen,et al.  Intensified multifactorial intervention in patients with type 2 diabetes mellitus and microalbuminuria: the Steno type 2 randomised study , 1999, The Lancet.

[17]  M. Lishner,et al.  Long-Term Stabilizing Effect of Angiotensin-Converting Enzyme Inhibition on Plasma Creatinine and on Proteinuria in Normotensive Type II Diabetic Patients , 1993, Annals of Internal Medicine.

[18]  R. Dantzer,et al.  Interferon-gamma and tumor necrosis factor-alpha mediate the upregulation of indoleamine 2,3-dioxygenase and the induction of depressive-like behavior in mice in response to bacillus Calmette-Guerin. , 2009, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[19]  Byung Jin Kim,et al.  Serum phosphate levels and the risk of cardiovascular disease and metabolic syndrome: a double-edged sword. , 2009, Diabetes research and clinical practice.

[20]  J. Schalken,et al.  Interferon and Tumor Necrosis Factor in Renal Cell Carcinoma Model Systems , 1991 .