Depletion of polycistronic transcripts using short interfering RNAs: cDNA synthesis method affects levels of non-targeted genes determined by quantitative PCR
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[1] Anton J. Enright,et al. Lack of correlation between predicted and actual off-target effects of short-interfering RNAs targeting the human papillomavirus type 16 E7 oncogene , 2013, British Journal of Cancer.
[2] David V. Schaffer,et al. Antiviral RNAi: Translating Science Towards Therapeutic Success , 2011, Pharmaceutical Research.
[3] M. Stanley,et al. In vitro progression of human papillomavirus 16 episome-associated cervical neoplasia displays fundamental similarities to integrant-associated carcinogenesis. , 2010, Cancer research.
[4] K. Ui-Tei,et al. New highly potent and specific E6 and E7 siRNAs for treatment of HPV16 positive cervical cancer , 2008, Cancer Gene Therapy.
[5] Zhi-Ming Zheng,et al. Papillomavirus genome structure, expression, and post-transcriptional regulation. , 2006, Frontiers in bioscience : a journal and virtual library.
[6] J. McCoy,et al. Short-term induction and long-term suppression of HPV16 oncogene silencing by RNA interference in cervical cancer cells , 2006, Oncogene.
[7] J. Milner,et al. Selective silencing of viral gene expression in HPV-positive human cervical carcinoma cells treated with siRNA, a primer of RNA interference , 2002, Oncogene.
[8] M. Stanley. Human papillomavirus and cervical carcinogenesis. , 2001, Best practice & research. Clinical obstetrics & gynaecology.
[9] T. Tuschl,et al. Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells , 2001, Nature.
[10] R. Pattillo,et al. Tumor antigen and human chorionic gonadotropin in CaSki cells: a new epidermoid cervical cancer cell line. , 1977, Science.