论文信息 - Cytoprotective roles of GSH, SOD and solcoseryl against ischemic damage and reperfusion injury to warm ischemic lung. Study of Canine warm ischemic lung.

Cytoprotective roles of GSH, SOD and solcoseryl against ischemic damage and reperfusion injury to warm ischemic lung. Study of Canine warm ischemic lung.

Title Cytoprotective roles of GSH, SOD and solcoseryl against ischemic damage and reperfusion injury to warm ischemic lung. Study of Canine warm ischemic lung. Author(s) Taniguchi, Hideki; Kawahara, Katsunobu; Nakasone, Tomonori; Ikari, Hideki; Honjoh, Seiji; Hisano, Hiroshi; Takahashi, Takao; Kusano, Hiroyuki; Ayabe, Hiroyoshi; Tomita, Masao Citation Acta medica Nagasakiensia. 1990, 35(1-4), p.110-115

[1] H. Taniguchi. Experimental study on active oxygen species to warm ischemic lung. - Availability of GSH, SOD, and alloprinol - , 1989 .

[2] J. McCord,et al. Oxygen-derived free radicals in postischemic tissue injury. , 1985, The New England journal of medicine.

[3] J. Crapo,et al. Reimplantation response in isografted rat lungs. Analysis of causal factors. , 1984, The Journal of thoracic and cardiovascular surgery.

[4] K. Vähäkangas,et al. Glutathione conjugation by isolated lung cells and the isolated, perfused lung. Effect of extracellular glutathione. , 1984, European journal of biochemistry.

[5] W. Pryor. Free Radicals in Biology , 1976 .

[6] S. Siegelman,et al. Pulmonary Reimplantation Response , 1973, Annals of surgery.

[7] I. Fridovich,et al. Superoxide dismutase. An enzymic function for erythrocuprein (hemocuprein). , 1969, The Journal of biological chemistry.

[8] I. Clark. Tissue Damage Caused by Free Oxygen Radicals , 1986, Pathology.

[9] M. Hess,et al. Identification of hydrogen peroxide and hydroxyl radicals as mediators of leukocyte-induced myocardial dysfunction. Limitation of infarct size with neutrophil inhibition and depletion. , 1985, Advances in myocardiology.