Ischemic Damage in Hippocampal CA1 is Dependent on Glutamate Release and Intact Innervation from CA3
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H. Benveniste | N. Diemer | H. Hagberg | M. Sandberg | T. Christensen | M. B. Jørgensen | Mats Sandberg | Henrik Hagberg | Helene Benveniste | Martin Balslev Jørgensen | Thomas Christensen | Nils Henrik Diemer
[1] A. Guidotti. Neurotoxicity of excitatory amino acids , 1990 .
[2] H. Benveniste. Brain Microdialysis , 1989, Journal of neurochemistry.
[3] H. Benveniste,et al. Determination of Brain Interstitial Concentrations by Microdialysis , 1989, Journal of neurochemistry.
[4] David G. Nicholls,et al. Release of Glutamate, Aspartate, and γ‐Aminobutyric Acid from Isolated Nerve Terminals , 1989 .
[5] D. Nicholls. Release of glutamate, aspartate, and gamma-aminobutyric acid from isolated nerve terminals. , 1989, Journal of neurochemistry.
[6] H. Benveniste,et al. Calcium accumulation by glutamate receptor activation is involved in hippocampal cell damage after ischemia , 1988, Acta neurologica Scandinavica.
[7] R. Busto,et al. Effect of Ischemia on the In Vivo Release of Striatal Dopamine, Glutamate, and γ‐Aminobutyric Acid Studied by Intracerebral Microdialysis , 1988, Journal of neurochemistry.
[8] R. Kauppinen,et al. Ca2+-dependent and Ca2+- independent glutamate release, energy status and cytosolic free Ca2+ concentration in isolated nerve terminals following metabolic inhibition: Possible relevance to hyoglycaemia and anoxia , 1988, Neuroscience.
[9] J. Korf,et al. Increases in Striatal and Hippocampal Impedance and Extracellular Levels of Amino Acids by Cardiac Arrest in Freely Moving Rats , 1988, Journal of neurochemistry.
[10] B. Meldrum,et al. Long-Term Development of Selective Neuronal Loss and the Mechanism of Protection by 2-Amino-7-Phosphonoheptanoate in a Rat Model of Incomplete Forebrain Ischaemia , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[11] W. Dalton Dietrich,et al. Small Differences in Intraischemic Brain Temperature Critically Determine the Extent of Ischemic Neuronal Injury , 1987, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[12] D. Choi. Ionic dependence of glutamate neurotoxicity , 1987, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[13] C. Nicholson,et al. The Migration of Substances in the Neuronal Microenvironment a , 1986, Annals of the New York Academy of Sciences.
[14] U. Ungerstedt,et al. Microdialysis in the study of extracellular levels of amino acids in the rat brain. , 1986, Acta physiologica Scandinavica.
[15] S. Butcher,et al. Extracellular Overflow of Neuroactive Amino Acids During Severe Insulin‐Induced Hypoglycemia: In Vivo Dialysis of the Rat Hippocampus , 1986, Journal of neurochemistry.
[16] Stephen J. Smith,et al. NMDA-receptor activation increases cytoplasmic calcium concentration in cultured spinal cord neurones , 1986, Nature.
[17] A. Hamberger,et al. Ischemia-Induced Shift of Inhibitory and Excitatory Amino Acids from Intra- to Extracellular Compartments , 1985, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[18] H. Benveniste,et al. Cellular Origin of Ischemia‐Induced Glutamate Release from Brain Tissue In Vivo and In Vitro , 1985, Journal of neurochemistry.
[19] F. Gage,et al. Evidence for amelioration of ischaemic neuronal damage in the hippocampal formation by lesions of the perforant path. , 1985, Neurological research.
[20] A. Hamberger,et al. Liquid Chromatographic Determination of Amino Acids After Precolumn Fluorescence Derivatization , 1985 .
[21] A. Hansen,et al. Effect of anoxia on ion distribution in the brain. , 1985, Physiological reviews.
[22] H. Benveniste,et al. Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral Microdialysis , 1984, Journal of neurochemistry.
[23] B. Siesjö. Cell Damage in the Brain: A Speculative Synthesis , 1984, Acta psychiatrica Scandinavica. Supplementum.
[24] G. Paxinos,et al. The Rat Brain in Stereotaxic Coordinates , 1983 .
[25] N. Diemer,et al. Selective neuron loss after cerebral ischemia in the rat: Possible role of transmitter glutamate , 1982, Acta neurologica Scandinavica.
[26] Fred Plum,et al. Temporal profile of neuronal damage in a model of transient forebrain ischemia , 1982, Annals of neurology.
[27] R. Schwarcz,et al. Monosodium glutamate: increased neurotoxicity after removal of neuronal re-uptake sites , 1981, Brain Research.
[28] A. Hansen,et al. Brain extracellular space during spreading depression and ischemia. , 1980, Acta physiologica Scandinavica.
[29] J. Farber,et al. Calcium dependence of toxic cell death: a final common pathway. , 1979, Science.
[30] A. R. Gardner-Medwin,et al. Diffusion from an iontophoretic point source in the brain: role of tortuosity and volume fraction , 1979, Brain Research.
[31] J. Brierley,et al. A New Model of Bilateral Hemispheric Ischemia in the Unanesthetized Rat , 1979, Stroke.