Targeted expression of heme oxygenase-1 prevents the pulmonary inflammatory and vascular responses to hypoxia
暂无分享,去创建一个
T. Minamino | N. Abraham | C. Hsieh | S. Kourembanas | S. A. Mitsialis | H. Christou | M. Perrella | Yuxiang Liu | V. Dhawan | S. Mitsialis
[1] Adrian M Heilbut,et al. Complete reversal of fatal pulmonary hypertension in rats by a serine elastase inhibitor , 2000, Nature Medicine.
[2] A. Choi,et al. Carbon monoxide has anti-inflammatory effects involving the mitogen-activated protein kinase pathway , 2000, Nature Medicine.
[3] J Zaky,et al. Upregulation of heme oxygenase-1 protects genetically fat Zucker rat livers from ischemia/reperfusion injury. , 1999, The Journal of clinical investigation.
[4] B. Gewertz,et al. Endothelial permeability and IL-6 production during hypoxia: role of ROS in signal transduction. , 1999, American journal of physiology. Lung cellular and molecular physiology.
[5] T. Standiford,et al. Chemokines in lung injury: Thomas A. Neff Lecture. , 1999, Chest.
[6] L. Kobzik,et al. Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice. , 1999, The Journal of clinical investigation.
[7] A. Choi,et al. Exogenous administration of heme oxygenase-1 by gene transfer provides protection against hyperoxia-induced lung injury. , 1999, The Journal of clinical investigation.
[8] M. Jordana,et al. Cytokines and pulmonary inflammatory and immune diseases. , 1999, Histology and histopathology.
[9] N. Chandel,et al. Mitochondrial reactive oxygen species trigger hypoxia-induced transcription. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[10] R. Colvin,et al. Expression of heme oxygenase-1 can determine cardiac xenograft survival , 1998, Nature Medicine.
[11] I. Charo,et al. Decreased lesion formation in CCR2−/− mice reveals a role for chemokines in the initiation of atherosclerosis , 1998, Nature.
[12] S. Kourembanas,et al. Increased vascular endothelial growth factor production in the lungs of rats with hypoxia-induced pulmonary hypertension. , 1998, American journal of respiratory cell and molecular biology.
[13] K. Matsushima,et al. Alleviation of monocrotaline-induced pulmonary hypertension by antibodies to monocyte chemotactic and activating factor/monocyte chemoattractant protein-1. , 1998, Laboratory investigation; a journal of technical methods and pathology.
[14] M. Jordana,et al. IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses. , 1998, The Journal of clinical investigation.
[15] S. Kourembanas,et al. Carbon Monoxide Controls the Proliferation of Hypoxic Vascular Smooth Muscle Cells* , 1997, The Journal of Biological Chemistry.
[16] B. Rollins,et al. Tissue Factor Is Induced by Monocyte Chemoattractant Protein-1 in Human Aortic Smooth Muscle and THP-1 Cells* , 1997, The Journal of Biological Chemistry.
[17] M. Maines,et al. Isolation and characterization of a cDNA from the rat brain that encodes hemoprotein heme oxygenase-3. , 1997, European journal of biochemistry.
[18] P. Jones,et al. Tenascin-C, proliferation and subendothelial fibronectin in progressive pulmonary vascular disease. , 1997, The American journal of pathology.
[19] Y. S. Zhou,et al. Monocytes and tissue factor promote thrombosis in a murine model of oxygen deprivation. , 1997, The Journal of clinical investigation.
[20] S. Kourembanas,et al. Mechanisms by which oxygen regulates gene expression and cell-cell interaction in the vasculature. , 1997, Kidney international.
[21] M. Rabinovitch,et al. Exogenous leukocyte and endogenous elastases can mediate mitogenic activity in pulmonary artery smooth muscle cells by release of extracellular matrix‐bound basic fibroblast growth factor , 1996, Journal of cellular physiology.
[22] N. Abraham,et al. The Biological Significance and Physiological Role of Heme Oxygenase , 1996 .
[23] S. Kourembanas,et al. Endothelial cell expression of vasoconstrictors and growth factors is regulated by smooth muscle cell-derived carbon monoxide. , 1995, The Journal of clinical investigation.
[24] M. Gerritsen,et al. Transfection of the human heme oxygenase gene into rabbit coronary microvessel endothelial cells: protective effect against heme and hemoglobin toxicity. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[25] M. Humbert,et al. Increased interleukin-1 and interleukin-6 serum concentrations in severe primary pulmonary hypertension. , 1995, American journal of respiratory and critical care medicine.
[26] S. Kourembanas,et al. Smooth muscle cell-derived carbon monoxide is a regulator of vascular cGMP. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[27] M. Nauck,et al. Intracellular interleukin 6 mediates platelet-derived growth factor-induced proliferation of nontransformed cells. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[28] N. Voelkel,et al. Interleukin-1 receptor antagonist treatment reduces pulmonary hypertension generated in rats by monocrotaline. , 1994, American journal of respiratory cell and molecular biology.
[29] T. Yue,et al. Interleukin-8. A mitogen and chemoattractant for vascular smooth muscle cells. , 1994, Circulation research.
[30] B. Groves,et al. Exuberant endothelial cell growth and elements of inflammation are present in plexiform lesions of pulmonary hypertension. , 1994, The American journal of pathology.
[31] M. Rabinovitch,et al. Chronic hypoxic pulmonary hypertension in rats and increased elastolytic activity. , 1991, The American journal of physiology.
[32] S. Potter,et al. Cis-acting sequences from a human surfactant protein gene confer pulmonary-specific gene expression in transgenic mice. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[33] R. Mecham,et al. Smooth muscle-mediated connective tissue remodeling in pulmonary hypertension. , 1987, Science.
[34] W. Zapol,et al. Pulmonary hypertension in severe acute respiratory failure. , 1977, The New England journal of medicine.