The capacity of interleukin‐4 to induce in vitro IgE synthesis by B cells of patients with common variable immunodeficiency
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[1] J. D. de Vries,et al. Regulation of human IgE synthesis. I. Human IgE synthesis in vitro is determined by the reciprocal antagonistic effects of interleukin 4 and interferon‐γ , 1990, European journal of immunology.
[2] J. D. de Vries,et al. Peripheral blood lymphocytes of patients with common variable immunodeficiency (CVI) produce reduced levels of interleukin-4, interleukin-2 and interferon-gamma, but proliferate normally upon activation by mitogens. , 1989, Clinical and experimental immunology.
[3] J. D. de Vries,et al. Modulation of IL‐4‐induced human IgE production in vitro by IFN‐γ and IL‐5: The role of soluble CD23 (s‐CD23) , 1989, Journal of cellular biochemistry.
[4] J. Banchereau,et al. Development of polyclonal and monoclonal antibodies for immunoassay and neutralization of human interleukin-4. , 1989, Journal of immunological methods.
[5] J. Banchereau,et al. Epitope mapping of recombinant human gamma interferon using monoclonal antibodies. , 1989, Molecular immunology.
[6] F. Alt,et al. Mitogen plus interleukin 4 induction of C epsilon transcripts in B lymphoid cells , 1988, The Journal of experimental medicine.
[7] K. Arai,et al. IgE production by normal human lymphocytes is induced by interleukin 4 and suppressed by interferons gamma and alpha and prostaglandin E2. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[8] J. Banchereau,et al. IgE production by normal human B cells induced by alloreactive T cell clones is mediated by IL-4 and suppressed by IFN-gamma. , 1988, Journal of immunology.
[9] J. Bonnefoy,et al. Interleukin 5 enhances interleukin 4‐induced IgE production by normal human B cells. The role of soluble CD23 antigen , 1988, European journal of immunology.
[10] D. Matheson,et al. Defect in production of B cell differentiation factor-like activity by mononuclear cells from a boy with hypogammaglobulinemia. , 1987, Journal of immunology.
[11] D. Weisdorf,et al. Impaired responsiveness to B cell growth factor in a patient with common variable hypogammaglobulinemia , 1985 .
[12] H. Kunkel,et al. Polyclonal immunoglobulin secretion in patients with common variable immunodeficiency using monoclonal B cell differentiation factors. , 1984, The Journal of clinical investigation.
[13] Y. Saeki,et al. Dissociation in the production of B cell-stimulating factors (BCGF and BCDF) and interleukin 2 by T cells from a common variable immunodeficient patient. , 1984, Journal of immunology.
[14] W. V. van Blitterswijk,et al. Serum-free medium for generation and propagation of functional human cytotoxic and helper T cell clones. , 1984, Journal of immunological methods.
[15] B. Jones. Identification of B-cell and T-helper-cell defects, and of suppressor cell hyperactivity, in humoral immunodeficiency. , 1984, Clinical immunology and immunopathology.
[16] J. Farrant,et al. The role of B cell differentiation factors and specific T cell help in the pathogenesis of primary hypogammaglobulinemia , 1984, European journal of immunology.
[17] C. Cunningham-Rundles,et al. Three distinct stages of B-cell defects in common varied immunodeficiency. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[18] M. López-Botet,et al. Relationship between IL 2 synthesis and the proliferative response to PHA in different primary immunodeficiencies. , 1982, Journal of immunology.
[19] D. Webster,et al. Immature B cells in fetal development and immunodeficiency: Studies of IgM, IgG, IgA and IgD production in vitro using epstein‐Barr virus activation , 1982, European journal of immunology.
[20] E. Reinherz,et al. Abnormalities of T cell maturation and regulation in human beings with immunodeficiency disorders. , 1981, The Journal of clinical investigation.
[21] H. Mitsuya,et al. Pathophysiologic analysis of peripheral blood lymphocytes from patients with primary immunodeficiency. I. Ig synthesis by peripheral blood lymphocytes stimulated with either pokeweed mitogen or Epstein-Barr virus in vitro. , 1981, Journal of immunology.
[22] G. Asherson,et al. Two immunologically distinct forms of late-onset hypogammaglobulinaemia. , 1981, Clinical and experimental immunology.
[23] E. Reinherz,et al. Immunodeficiency associated with loss of T4+ inducer T-cell function. , 1981, The New England journal of medicine.
[24] A. Rickinson,et al. Functional 'immaturity' of isolated B cells from patients with hypogammaglobulinaemia. , 1980, Clinical and experimental immunology.
[25] I. Magrath,et al. B cell differentiation and immunoregulatory T cell function in human cord blood lymphocytes. , 1980, The Journal of clinical investigation.
[26] H. Lazarus,et al. Bone marrow-derived lymphoid cell lines from patients with agammaglobulinemia. , 1978, The Journal of clinical investigation.
[27] Good Ra,et al. Human lymphocyte differentiation markers and their application to immune deficiency and lymphoproliferative diseases. , 1977 .
[28] G. Asherson,et al. Quantitative measurements of T- and B-cell function in "variable" primary hypogammaglobulinaemia: evidence for a consistent B-cell defect. , 1977, Clinical and experimental immunology.
[29] R. Good,et al. Suppression of B-cell differentiation by leukocytes from hypogammaglobulinemic patients. , 1976, The Journal of clinical investigation.
[30] T. Waldmann,et al. Role of suppressor T cells in pathogenesis of common variable hypogammaglobulinaemia. , 1974, Lancet.
[31] R. Geha,et al. Heterogeneity of "acquired" or common variable agammaglobulinemia. , 1974, The New England journal of medicine.
[32] M. Cooper,et al. Differentiation capacity of cultured B lymphocytes from immunodeficient patients. , 1973, The Journal of clinical investigation.
[33] C. Griscelli,et al. Immunoglobulins on the surface of lymphocytes in fifty patients with primary immunodeficiency diseases. , 1973, Clinical immunology and immunopathology.
[34] M. Cooper,et al. Agammaglobulinaemia with B lymphocytes. Specific defect of plasma-cell differentiation. , 1971, Lancet.