Intensive care medicine
暂无分享,去创建一个
[1] Abraham Weizman,et al. Insulin-like-growth-factor-I (IGF-I) antagonizes apoptosis induced by serum deficiency and doxorubicin in neuronal cell culture. , 1999, Growth hormone & IGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society.
[2] G. Kroemer,et al. Redox regulation of apoptosis: Impact of thiol oxidation status on mitochondrial function , 1997, European journal of immunology.
[3] R. Simon,et al. Apoptosis Repressor Genes Bcl-2 and Bcl-x-Long are Expressed in the Rat Brain following Global Ischemia , 1997, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[4] D. Nicholson,et al. Caspase structure, proteolytic substrates, and function during apoptotic cell death , 1999, Cell Death and Differentiation.
[5] D. Olson,et al. Initial evidence of endothelial cell apoptosis as a mechanism of systemic capillary leak syndrome. , 2001, Chest.
[6] Jay X. Tang,et al. Caspase-3-generated fragment of gelsolin: effector of morphological change in apoptosis. , 1997, Science.
[7] S. Matoba,et al. Important role of energy-dependent mitochondrial pathways in cultured rat cardiac myocyte apoptosis. , 2001, American journal of physiology. Heart and circulatory physiology.
[8] C. F. Chen,et al. De novo demonstration and co-localization of free-radical production and apoptosis formation in rat kidney subjected to ischemia/reperfusion. , 2001, Journal of the American Society of Nephrology : JASN.
[9] C. Potten,et al. Stem cells: the intestinal stem cell as a paradigm. , 2000, Carcinogenesis.
[10] S H Kaufmann,et al. Mammalian caspases: structure, activation, substrates, and functions during apoptosis. , 1999, Annual review of biochemistry.
[11] Junying Yuan,et al. Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.
[12] S. Korsmeyer,et al. Bax-Deficient Mice with Lymphoid Hyperplasia and Male Germ Cell Death , 1995, Science.
[13] D. Holtzman,et al. Clusterin contributes to caspase-3–independent brain injury following neonatal hypoxia-ischemia , 2001, Nature Medicine.
[14] M. Ashraf,et al. Mitochondrial K(ATP) channel activation reduces anoxic injury by restoring mitochondrial membrane potential. , 2001, American journal of physiology. Heart and circulatory physiology.
[15] U. Felderhoff‐Mueser,et al. Fas/CD95/APO‐1 Can Function as a Death Receptor for Neuronal Cells in Vitro and in Vivo and is Upregulated Following Cerebral Hypoxic‐Ischemic Injury to the Developing Rat Brain , 2000, Brain pathology.
[16] G M Bokoch,et al. Membrane and morphological changes in apoptotic cells regulated by caspase-mediated activation of PAK2. , 1997, Science.
[17] J. Cadet,et al. Methamphetamine-induced apoptosis is attenuated in the striata of copper-zinc superoxide dismutase transgenic mice. , 2000, Brain research. Molecular brain research.
[18] N. Thornberry,et al. A Combinatorial Approach Defines Specificities of Members of the Caspase Family and Granzyme B , 1997, The Journal of Biological Chemistry.
[19] Z. Endre,et al. Bcl‐2 genes and growth factors in the pathology of ischaemic acute renal failure , 1999, Immunology and cell biology.
[20] N. Rothwell,et al. An ICE inhibitor, z-VAD-DCB attenuates ischaemic brain damage in the rat. , 1996, Neuroreport.
[21] T. Ohtsuka,et al. Expression of Interleukin-1β Converting Enzyme Gene Family and bcl-2 Gene Family in the Rat Brain following Permanent Occlusion of the Middle Cerebral Artery , 1997, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[22] Scott W. Lowe,et al. p53 is required for radiation-induced apoptosis in mouse thymocytes , 1993, Nature.