Sézary syndrome with elevated serum IgE and hypereosinophilia: role of dysregulated cytokine production.

[1]  A J Wardlaw,et al.  Expression of mRNA for interleukin-5 in mucosal bronchial biopsies from asthma. , 1991, The Journal of clinical investigation.

[2]  P. Nowell,et al.  Malignant and nonmalignant T cell lines from human T cell lymphotropic virus type I-negative patients with Sézary syndrome. , 1991, Journal of immunology.

[3]  L. Borish,et al.  267 Identification of interleukin 2 and 4 messenger RNA in bronchial alveolar lavage cells and allergen specific cell lines from atopic individuals , 1991 .

[4]  H. Jansen,et al.  Evidence for compartmentalization of functional subsets of CD2+ T lymphocytes in atopic patients. , 1990, Journal of immunology.

[5]  J. Zubeldia,et al.  Murine eosinophils and IL-1: alpha IL-1 mRNA detection by in situ hybridization. Production and release of IL-1 from peritoneal eosinophils. , 1990, Journal of immunology.

[6]  H. Koh,et al.  Sézary syndrome: diagnosis, prognosis, and critical review of treatment options. , 1990, Journal of the American Academy of Dermatology.

[7]  S. Romagnani.  Regulation and deregulation of human IgE synthesis. , 1990, Immunology today.

[8]  A. W. Tam,et al.  Message amplification phenotyping (MAPPing): a technique to simultaneously measure multiple mRNAs from small numbers of cells. , 1989, BioTechniques.

[9]  D. Umetsu,et al.  Induction of human IgE synthesis by CD4+ T cell clones. Requirement for interleukin 4 and low molecular weight B cell growth factor , 1989, The Journal of experimental medicine.

[10]  W. Owen,et al.  IL-5-dependent conversion of normodense human eosinophils to the hypodense phenotype uses 3T3 fibroblasts for enhanced viability, accelerated hypodensity, and sustained antibody-dependent cytotoxicity. , 1989, Journal of immunology.

[11]  R. Geha,et al.  Endogenous interleukin 6 plays an obligatory role in interleukin 4‐dependent human IgE synthesis , 1989, European journal of immunology.

[12]  C. Sanderson,et al.  Human interleukin-5 (IL-5) regulates the production of eosinophils in human bone marrow cultures: comparison and interaction with IL-1, IL-3, IL-6, and GMCSF. , 1989, Blood.

[13]  J. Voorhees,et al.  Markedly diminished epidermal keratinocyte expression of intercellular adhesion molecule-1 (ICAM-1) in Sézary syndrome. , 1989, JAMA.

[14]  D. Vercelli,et al.  Induction of human IgE synthesis requires interleukin 4 and T/B cell interactions involving the T cell receptor/CD3 complex and MHC class II antigens , 1989, The Journal of experimental medicine.

[15]  R. Strieter,et al.  Endothelial cell gene expression of a neutrophil chemotactic factor by TNF-alpha, LPS, and IL-1 beta. , 1989, Science.

[16]  J. Voorhees,et al.  Gamma interferon induces different keratinocyte cellular patterns of expression of HLA‐DR and DQ and intercellular adhesion molecule‐I (ICAM‐I) antigens , 1989, The British journal of dermatology.

[17]  R. Coffman,et al.  TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties. , 1989, Annual review of immunology.

[18]  B. Nickoloff,et al.  Role of interferon-gamma in cutaneous trafficking of lymphocytes with emphasis on molecular and cellular adhesion events. , 1988, Archives of dermatology.

[19]  H. Kikutani,et al.  Two species of human Fcε receptor II ( FcεRII CD23 ): Tissue-specific and IL-4-specific regulation of gene expression , 1988, Cell.

[20]  K. Arai,et al.  IgE production by normal human lymphocytes is induced by interleukin 4 and suppressed by interferons gamma and alpha and prostaglandin E2. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[21]  J. Banchereau,et al.  IgE production by normal human B cells induced by alloreactive T cell clones is mediated by IL-4 and suppressed by IFN-gamma. , 1988, Journal of immunology.

[22]  M J Banda,et al.  Wound macrophages express TGF-alpha and other growth factors in vivo: analysis by mRNA phenotyping. , 1988, Science.

[23]  J. Banchereau,et al.  IL-4 is an essential factor for the IgE synthesis induced in vitro by human T cell clones and their supernatants. , 1988, Journal of immunology.

[24]  J. Bonnefoy,et al.  Interleukin 5 enhances interleukin 4‐induced IgE production by normal human B cells. The role of soluble CD23 antigen , 1988, European journal of immunology.

[25]  W. Owen,et al.  Human eosinophils have prolonged survival, enhanced functional properties, and become hypodense when exposed to human interleukin 3. , 1988, The Journal of clinical investigation.

[26]  F. Finkelman,et al.  The role of L3T4+ and Lyt-2+ T cells in the IgE response and immunity to Nippostrongylus brasiliensis. , 1988, Journal of immunology.

[27]  Michael Loran Dustin,et al.  Adhesion of T lymphoblasts to epidermal keratinocytes is regulated by interferon gamma and is mediated by intercellular adhesion molecule 1 (ICAM-1) , 1988, The Journal of experimental medicine.

[28]  R. Geha,et al.  Human recombinant interleukin 4 induces Fc epsilon R2/CD23 on normal human monocytes , 1988, The Journal of experimental medicine.

[29]  R. Coffman,et al.  IFN-gamma regulates the isotypes of Ig secreted during in vivo humoral immune responses. , 1988, Journal of immunology.

[30]  J. Gamble,et al.  Recombinant human interleukin 5 is a selective activator of human eosinophil function , 1988, The Journal of experimental medicine.

[31]  W. Owen,et al.  Regulation of human eosinophil viability, density, and function by granulocyte/macrophage colony-stimulating factor in the presence of 3T3 fibroblasts , 1987, The Journal of experimental medicine.

[32]  E. Kieff,et al.  Epstein-Barr virus nuclear antigen 2 specifically induces expression of the B-cell activation antigen CD23. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[33]  W. Paul,et al.  B-cell stimulatory factor-1/interleukin 4. , 1987, Annual review of immunology.

[34]  J. Gamble,et al.  Recombinant human granulocyte-macrophage colony-stimulating factor stimulates in vitro mature human neutrophil and eosinophil function, surface receptor expression, and survival. , 1986, The Journal of clinical investigation.

[35]  R. Warnke,et al.  Leu-8 and Leu-9 antigen phenotypes: immunologic criteria for the distinction of mycosis fungoides from cutaneous inflammation. , 1986, Journal of the American Academy of Dermatology.

[36]  R. Coffman,et al.  Two types of murine helper T cell clone. I. Definition according to profiles of lymphokine activities and secreted proteins. , 1986, Journal of immunology.

[37]  R. Coffman,et al.  A T cell activity that enhances polyclonal IgE production and its inhibition by interferon-gamma. , 1986, Journal of immunology.

[38]  G. Klaus,et al.  Eosinophil differentiation factor also has B-cell growth factor activity: proposed name interleukin 4. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[39]  A. Capron,et al.  Involvement of immunoglobulin E in the secretory processes of alveolar macrophages from asthmatic patients. , 1983, The Journal of clinical investigation.

[40]  A. Cohen,et al.  Characterization of Fc receptors for IgE on human alveolar macrophages. , 1982, Clinical and experimental immunology.

[41]  R. Warnke,et al.  Subtypes of cutaneous T-cell lymphoma defined by expression of leu-1 and Ia. , 1982, Blood.

[42]  B. Haynes,et al.  Phenotypic characterization of cutaneous T-cell lymphoma. Use of monoclonal antibodies to compare with other malignant T cells. , 1981, The New England journal of medicine.

[43]  貞紀 長尾,et al.  [Cutaneous T-Cell Lymphoma]. , 1981 .

[44]  H. Spiegelberg,et al.  Fc receptors for IgE on a subpopulation of human peripheral blood monocytes. , 1980, Journal of immunology.

[45]  H. Spiegelberg,et al.  Fc receptors specific for IgE on subpopulations of human lymphocytes and monocytes. , 1980, Clinical immunology and immunopathology.

[46]  M. Suko,et al.  IgE levels in nude mice. , 1979, International archives of allergy and applied immunology.

[47]  T. Waldmann,et al.  The Sézary syndrome: a malignant proliferation of helper T cells. , 1976, The Journal of clinical investigation.

[48]  T. Yoshida,et al.  Migration inhibitory activity in serum and cell supernatants in patients with Sezary syndrome. , 1975, Journal of Immunology.

[49]  I. Bernstein,et al.  Thymus dependence of reaginic antibody formation in mice. , 1973, Journal of immunology.

[50]  P. Beeson,et al.  MECHANISM OF EOSINOPHILIA I. FACTORS AFFECTING THE EOSINOPHIL RESPONSE OF RATS TO TRICHINELLA SPIRALIS , 1970 .

[51]  P. Beeson,et al.  Mechanism of eosinophilia. II. Role of the lymphocyte. , 1970 .