论文信息 - Gain-of-function mutation S422L in the KCNJ8-encoded cardiac K(ATP) channel Kir6.1 as a pathogenic substrate for J-wave syndromes. - 字舞流文

Gain-of-function mutation S422L in the KCNJ8-encoded cardiac K(ATP) channel Kir6.1 as a pathogenic substrate for J-wave syndromes.

P. Schwartz | D. Tester | M. Ackerman | J. Makielski | A. Medeiros-Domingo | L. Eckhardt | L. Crotti | Bi-hua Tan | Chunhua Song | Qing Zhou | A. Cuoretti | Stacie Kroboth | Doug Kopp | M. Ackerman