Cellular events involved in E. coli cells inactivation by several agents for food preservation: A comparative study.

[1]  S. Condón,et al.  Oxidative stress in E. coli cells upon exposure to heat treatments. , 2017, International journal of food microbiology.

[2]  S. Condón,et al.  Influence of growth and treatment temperature on Staphylococcus aureus resistance to pulsed electric fields: Relationship with membrane fluidity , 2016 .

[3]  S. Condón,et al.  Effect of high pressure treatment on inactivation of vegetative pathogens and on denaturation of whey proteins in different media , 2015 .

[4]  G. Stan,et al.  Interplay between E. coli DnaK, ClpB and GrpE during protein disaggregation. , 2015, Journal of molecular biology.

[5]  J. Imlay,et al.  The molecular mechanisms and physiological consequences of oxidative stress: lessons from a model bacterium , 2013, Nature Reviews Microbiology.

[6]  Yuta Sakai,et al.  Knockdown of recA gene expression by artificial small RNAs in Escherichia coli. , 2013, Biochemical and biophysical research communications.

[7]  O. Pakhomova,et al.  Oxidative effects of nanosecond pulsed electric field exposure in cells and cell-free media. , 2012, Archives of biochemistry and biophysics.

[8]  T. Abee,et al.  Primary and secondary oxidative stress in Bacillus. , 2011, Environmental microbiology.

[9]  V. Lushchak.  Adaptive response to oxidative stress: Bacteria, fungi, plants and animals. , 2011, Comparative biochemistry and physiology. Toxicology & pharmacology : CBP.

[10]  E. Puértolas,et al.  Evaluation of a static treatment chamber to investigate kinetics of microbial inactivation by pulsed electric fields at different temperatures at quasi-isothermal conditions , 2010 .

[11]  S. Condón,et al.  Development of stress resistance in Staphylococcus aureus after exposure to sublethal environmental conditions. , 2010, International journal of food microbiology.

[12]  B. Kalyanaraman,et al.  Hydroethidine- and MitoSOX-derived red fluorescence is not a reliable indicator of intracellular superoxide formation: another inconvenient truth. , 2010, Free radical biology & medicine.

[13]  R. Moezelaar,et al.  Analysis of acid-stressed Bacillus cereus reveals a major oxidative response and inactivation-associated radical formation. , 2010, Environmental microbiology.

[14]  P. Mañas,et al.  The relationship between membrane damage, release of protein and loss of viability in Escherichia coli exposed to high hydrostatic pressure. , 2010, International journal of food microbiology.

[15]  T. Abee,et al.  The impact of oxygen availability on stress survival and radical formation of Bacillus cereus. , 2009, International journal of food microbiology.

[16]  S. Condón,et al.  Heat and pulsed electric field resistance of pigmented and non-pigmented enterotoxigenic strains of Staphylococcus aureus in exponential and stationary phase of growth. , 2007, International journal of food microbiology.

[17]  A. Yousef,et al.  Genes of Escherichia coli O157:H7 That Are Involved in High-Pressure Resistance , 2006, Applied and Environmental Microbiology.

[18]  C. A. Miles.  Relating Cell Killing to Inactivation of Critical Components , 2006, Applied and Environmental Microbiology.

[19]  Eduarda Fernandes,et al.  Fluorescence probes used for detection of reactive oxygen species. , 2005, Journal of biochemical and biophysical methods.

[20]  E. Nudler,et al.  NO-mediated cytoprotection: instant adaptation to oxidative stress in bacteria. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[21]  A H Geeraerd,et al.  GInaFiT, a freeware tool to assess non-log-linear microbial survivor curves. , 2005, International journal of food microbiology.

[22]  P. Mañas,et al.  Microbial inactivation by new technologies of food preservation , 2005, Journal of applied microbiology.

[23]  A. Aertsen,et al.  Induction of Oxidative Stress by High Hydrostatic Pressure in Escherichia coli , 2005, Applied and Environmental Microbiology.

[24]  C. Georgiou,et al.  Interference of non-specific peroxidases in the fluorescence detection of superoxide radical by hydroethidine oxidation: a new assay for H2O2 , 2005, Analytical and bioanalytical chemistry.

[25]  M. Mergeay,et al.  Temperature-induced changes in bacterial physiology as determined by flow cytometry , 2005 .

[26]  John W. Foster,et al.  Escherichia coli Glutamate- and Arginine-Dependent Acid Resistance Systems Increase Internal pH and Reverse Transmembrane Potential , 2004, Journal of bacteriology.

[27]  A. Aertsen,et al.  Source of tryptone in growth medium affects oxidative stress resistance in Escherichia coli , 2004, Journal of applied microbiology.

[28]  S. Ehrlich,et al.  Stress responses in lactic acid bacteria , 2002, Antonie van Leeuwenhoek.

[29]  J. Imlay,et al.  Pathways of oxidative damage. , 2003, Annual review of microbiology.

[30]  Javier Raso,et al.  Comparing predicting models for the Escherichia coli inactivation by pulsed electric fields , 2003 .

[31]  J. Imlay,et al.  High Levels of Intracellular Cysteine Promote Oxidative DNA Damage by Driving the Fenton Reaction , 2003, Journal of bacteriology.

[32]  A. Aertsen,et al.  Comparison of sublethal injury induced in Salmonella enterica serovar Typhimurium by heat and by different nonthermal treatments. , 2003, Journal of food protection.

[33]  G. Barbosa‐Cánovas,et al.  Food Processing by High Hydrostatic Pressure , 2002, Critical reviews in food science and nutrition.

[34]  A. Yousef,et al.  Alternative food-preservation technologies: efficacy and mechanisms. , 2002, Microbes and infection.

[35]  A H Geeraerd,et al.  Structural model requirements to describe microbial inactivation during a mild heat treatment. , 2000, International journal of food microbiology.

[36]  S. Knabel,et al.  Comparison of different reducing agents for enhanced detection of heat-injured Listeria monocytogenes. , 2000, Journal of food protection.

[37]  T. Robinson,et al.  Variation in Resistance of Natural Isolates ofEscherichia coli O157 to High Hydrostatic Pressure, Mild Heat, and Other Stresses , 1999, Applied and Environmental Microbiology.

[38]  A. Yousef,et al.  Adaptation to sublethal environmental stresses protects Listeria monocytogenes against lethal preservation factors , 1997, Applied and environmental microbiology.