Sprague Dawley rats: A model of successful heart aging

Graphical abstract

[1]  T. Fulmer,et al.  Successful aging. , 2002, The American journal of nursing.

[2]  J. Farber,et al.  Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice. , 2010, The Journal of clinical investigation.

[3]  S. Campello,et al.  Mitochondrial shape changes: orchestrating cell pathophysiology , 2010, EMBO reports.

[4]  C. Gelfi,et al.  Protein modulation in mouse heart under acute and chronic hypoxia , 2011, Proteomics.

[5]  D. Klionsky,et al.  Regulation mechanisms and signaling pathways of autophagy. , 2009, Annual review of genetics.

[6]  K. Okamoto,et al.  Development of a strain of spontaneously hypertensive rats. , 1963, Japanese circulation journal.

[7]  E. Arriaga,et al.  Mitochondrial turnover and aging of long-lived postmitotic cells: the mitochondrial-lysosomal axis theory of aging. , 2010, Antioxidants & redox signaling.

[8]  G. L. Coleman,et al.  Pathological Changes During Aging in, Barrier-Reared Fischer 344 Male Rats1 , 1977, Journal of gerontology.

[9]  Keiko Takahashi,et al.  Angiotensin II receptor antagonist attenuates expression of aging markers in diabetic mouse heart. , 2006, Circulation journal : official journal of the Japanese Circulation Society.

[10]  G. McFadden,et al.  The serpin solution; targeting thrombotic and thrombolytic serine proteases in inflammation. , 2013, Cardiovascular & hematological disorders drug targets.

[11]  P. Rabinovitch,et al.  Cardiac aging in mice and humans: the role of mitochondrial oxidative stress. , 2009, Trends in cardiovascular medicine.

[12]  N. Mizushima,et al.  Autophagy: process and function. , 2007, Genes & development.

[13]  N. Sundaresan,et al.  SIRT3 Is a Stress-Responsive Deacetylase in Cardiomyocytes That Protects Cells from Stress-Mediated Cell Death by Deacetylation of Ku70 , 2008, Molecular and Cellular Biology.

[14]  J. G. Pastorino,et al.  Sirtuin-3 deacetylation of cyclophilin D induces dissociation of hexokinase II from the mitochondria , 2010, Journal of Cell Science.

[15]  R. Clark,et al.  The Effects of Overfeeding and Dietary Restriction on Sprague-Dawley Rat Survival and Early Pathology Biomarkers of Aging , 1994, Toxicologic pathology.

[16]  W. Dröge.  Oxidative stress and aging. , 2003, Advances in experimental medicine and biology.

[17]  M. Mann,et al.  Accurate Quantification of More Than 4000 Mouse Tissue Proteins Reveals Minimal Proteome Changes During Aging* , 2010, Molecular & Cellular Proteomics.

[18]  C. Franceschi,et al.  Mitochondria hyperfusion and elevated autophagic activity are key mechanisms for cellular bioenergetic preservation in centenarians , 2014, Aging.

[19]  Cecilia Gelfi,et al.  Comparative proteomic profile of rat sciatic nerve and gastrocnemius muscle tissues in ageing by 2‐D DIGE , 2009, Proteomics.

[20]  A. Raval,et al.  Proteomic profiling of aging in the mouse heart: Altered expression of mitochondrial proteins. , 2008, Archives of biochemistry and biophysics.

[21]  K. Rakušan,et al.  Distribution of mitochondria in normal and hypertrophic myocytes from the rat heart. , 1986, Journal of molecular and cellular cardiology.

[22]  R. S. Sohal,et al.  Coenzyme Q, oxidative stress and aging. , 2007, Mitochondrion.

[23]  M Matsuo,et al.  [Oxidative stress and aging]. , 1997, Nihon Ronen Igakkai zasshi. Japanese journal of geriatrics.

[24]  J. Rowe,et al.  Successful aging. , 1998, Aging.

[25]  Daniel Levy,et al.  Arterial and cardiac aging: major shareholders in cardiovascular disease enterprises: Part I: aging arteries: a "set up" for vascular disease. , 2003, Circulation.

[26]  John B. Shoven,et al.  I , Edinburgh Medical and Surgical Journal.

[27]  C. Gelfi,et al.  Specific protein changes contribute to the differential muscle mass loss during ageing , 2016, Proteomics.

[28]  P. Brigidi,et al.  Serum profiling of healthy aging identifies phospho- and sphingolipid species as markers of human longevity , 2014, Aging.

[29]  D. Sinclair,et al.  Regulation of the mPTP by SIRT3-mediated deacetylation of CypD at lysine 166 suppresses age-related cardiac hypertrophy , 2010, Aging.

[30]  Miss A.O. Penney.  (b) , 1974, The New Yale Book of Quotations.

[31]  P. Codogno,et al.  Canonical and non-canonical autophagy: variations on a common theme of self-eating? , 2011, Nature Reviews Molecular Cell Biology.

[32]  M. Barbagallo,et al.  Cardiovascular risk factors in centenarians , 2008, Experimental Gerontology.

[33]  B. Vallee,et al.  New molecular forms of human liver alcohol dehydrogenase: isolation and characterization of ADHIndianapolis. , 1980, Proceedings of the National Academy of Sciences of the United States of America.

[34]  M. Chandler,et al.  Impaired contractile function due to decreased cardiac myosin binding protein C content in the sarcomere. , 2013, American journal of physiology. Heart and circulatory physiology.

[35]  Daniel Levy,et al.  Arterial and cardiac aging: major shareholders in cardiovascular disease enterprises: Part II: the aging heart in health: links to heart disease. , 2003, Circulation.

[36]  T. Kawamoto,et al.  Manganese superoxide dismutase and aldehyde dehydrogenase deficiency increase mitochondrial oxidative stress and aggravate age-dependent vascular dysfunction. , 2008, Cardiovascular research.

[37]  이화영.  X , 1960, Chinese Plants Names Index 2000-2009.

[38]  Lloyd M. Smith,et al.  Proteoform: a single term describing protein complexity , 2013, Nature Methods.

[39]  T. Shioi,et al.  Aging as a substrate of heart failure. , 2012, Journal of cardiology.

[40]  S. Sadayappan,et al.  Phosphorylation and function of cardiac myosin binding protein-C in health and disease. , 2010, Journal of molecular and cellular cardiology.

[41]  C. Leeuwenburgh,et al.  New insights into the role of mitochondria in aging: mitochondrial dynamics and more , 2010, Journal of Cell Science.

[42]  J. Pang,et al.  Alpha-lipoic acid ameliorates oxidative stress by increasing aldehyde dehydrogenase-2 activity in patients with acute coronary syndrome. , 2013, The Tohoku journal of experimental medicine.