Induction and reversal of long-term potentiation by low- and high- intensity theta pattern stimulation
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W A Wilson | D S Barr | N A Lambert | K L Hoyt | S D Moore | Natacha Lambert | DS Barr | KL Hoyt | SD Moore | Wallace C. Wilson | S. Moore
[1] Charles F. Stevens,et al. Reversal of long-term potentiation by inhibitors of haem oxygenase , 1993, Nature.
[2] R. Anwyl,et al. The effects of external calcium on long-term potentiation in the rat hippocampal slice , 1988, Brain Research.
[3] T. Bliss,et al. A synaptic model of memory: long-term potentiation in the hippocampus , 1993, Nature.
[4] T J Teyler,et al. Multideterminant role of calcium in hippocampal synaptic plasticity , 1994, Hippocampus.
[5] G. Collingridge,et al. Metabotropic glutamate receptors contribute to the induction of long-term depression in the CA1 region of the hippocampus. , 1993, European journal of pharmacology.
[6] P. Stanton,et al. Priming of homosynaptic long-term depression in hippocampus by previous synaptic activity. , 1993, Neuroreport.
[7] G. Lynch,et al. Stable hippocampal long-term potentiation elicited by ‘theta’ pattern stimulation , 1987, Brain Research.
[8] T. Dunwiddie,et al. Characteristics of hippocampal primed burst potentiation in vitro and in the awake rat , 1988, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[9] R. Nicoll,et al. NMDA-receptor-dependent synaptic plasticity: multiple forms and mechanisms , 1993, Trends in Neurosciences.
[10] Roberto Malinow,et al. Persistent protein kinase activity underlying long-term potentiation , 1988, Nature.
[11] Thompson Pj. Memory function in patients with epilepsy. , 1991 .
[12] P. Thompson. Memory function in patients with epilepsy. , 1991, Advances in neurology.
[13] G. Collingridge,et al. MK-801 blocks NMDA receptor-mediated synaptic transmission and long term potentiation in rat hippocampal slices , 1987, Neuroscience Letters.
[14] G. Collingridge,et al. Excitatory amino acids in synaptic transmission in the Schaffer collateral‐commissural pathway of the rat hippocampus. , 1983, The Journal of physiology.
[15] G. Lynch,et al. Induction of synaptic potentiation in hippocampus by patterned stimulation involves two events. , 1986, Science.
[16] Hiroshi Kato,et al. Reversal of long-term potentiation (depotentiation) induced by tetanus stimulation of the input to CA1 neurons of guinea pig hippocampal slices , 1991, Brain Research.
[17] Gary Lynch,et al. Reversal of LTP by theta frequency stimulation , 1993, Brain Research.
[18] R. Malenka,et al. Mechanisms underlying induction of homosynaptic long-term depression in area CA1 of the hippocampus , 1992, Neuron.
[19] E. Kandel,et al. Low-frequency stimulation erases LTP through an NMDA receptor-mediated activation of protein phosphatases. , 1994, Learning & memory.
[20] Gary Lynch,et al. Stable depression of potentiated synaptic responses in the hippocampus with 1–5 Hz stimulation , 1990, Brain Research.
[21] G. Lynch,et al. Patterned stimulation at the theta frequency is optimal for the induction of hippocampal long-term potentiation , 1986, Brain Research.
[22] E. Halgren,et al. Recall deficits produced by afterdischarges in the human hippocampal formation and amygdala. , 1985, Electroencephalography and clinical neurophysiology.
[23] R. Malenka,et al. An essential role for protein phosphatases in hippocampal long-term depression. , 1993, Science.
[24] G. Lynch,et al. Long‐term potentiation and depression of synaptic responses in the rat hippocampus: localization and frequency dependency. , 1978, The Journal of physiology.
[25] R. Lester,et al. Frequency‐dependent N‐methyl‐D‐aspartate receptor‐mediated synaptic transmission in rat hippocampus. , 1988, The Journal of physiology.
[26] M. Bear,et al. Homosynaptic long-term depression in area CA1 of hippocampus and effects of N-methyl-D-aspartate receptor blockade. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[27] J. Lisman,et al. A mechanism for the Hebb and the anti-Hebb processes underlying learning and memory. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[28] H. Wigström,et al. Facilitation of hippocampal long-term potentiation in slices perfused with high concentrations of calcium , 1988, Brain Research.
[29] T. Teyler,et al. Reversible loss of hippocampal long term potentiation following electroconvulsive seizures , 1976, Nature.
[30] Hiroshi Kase,et al. Differential effects of protein kinase inhibitors on pre-established long-term potentiation in rat hippocampal neurons in vitro , 1991, Neuroscience Letters.
[31] E. Bienenstock,et al. Theory for the development of neuron selectivity: orientation specificity and binocular interaction in visual cortex , 1982, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[32] G. Collingridge,et al. NMDA Receptor‐dependent Transient Homo‐ and Heterosynaptic Depression in Picrotoxin‐treated Hippocampal Slices , 1992, The European journal of neuroscience.
[33] W. Abraham,et al. Priming of associative long-term depression in the dentate gyrus by θ frequency synaptic activity , 1992, Neuron.
[34] J. L. Stringer,et al. Elimination of long-term potentiation in the hippocampus by phencyclidine and ketamine , 1983, Brain Research.
[35] G. Collingridge,et al. A molecular switch activated by metabotropic glutamate receptors regulates induction of long-term potentiation , 1994, Nature.
[36] R. Malenka,et al. The influence of prior synaptic activity on the induction of long-term potentiation. , 1992, Science.
[37] L. Iversen,et al. 7-Chlorokynurenic acid is a selective antagonist at the glycine modulatory site of the N-methyl-D-aspartate receptor complex. , 1988, Proceedings of the National Academy of Sciences of the United States of America.
[38] SM Dudek,et al. Bidirectional long-term modification of synaptic effectiveness in the adult and immature hippocampus , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.
[39] M. Bear,et al. Synaptic plasticity: LTP and LTD , 1994, Current Opinion in Neurobiology.
[40] D. Linden,et al. Long-term synaptic depression in the mammalian brain , 1994, Neuron.
[41] L. Squire. Memory Functions as Affected by Electroconvulsive Therapy a , 1986, Annals of the New York Academy of Sciences.
[42] G. Lynch,et al. The effects of repetitive low frequency stimulation on control and "potentiated" synaptic responses in the hippocampus. , 1980, Life sciences.
[43] B. Alger,et al. Perfusion with high potassium plus glutamate can cause LTP erasure or persistent loss of neuronal responsiveness in the CA1 region of the hippocampal slice , 1993, Brain Research.
[44] C. Yamamoto,et al. Important factors in induction of long-term potentiation in thin hippocampal sections , 1981, Experimental Neurology.
[45] S. D. Moore,et al. Seizure-like activity disrupts LTP in vitro , 1993, Neuroscience Letters.