Interleukin 17 signaling drives Type I Interferon induced proliferative crescentic glomerulonephritis in lupus-prone mice.

[1]  M. Levesque,et al.  Type I interferon and T helper 17 cells co‐exist and co‐regulate disease pathogenesis in lupus patients , 2015, International journal of rheumatic diseases.

[2]  Youhua Liu,et al.  Complement Component C5a Permits the Coexistence of Pathogenic Th17 Cells and Type I IFN in Lupus , 2014, The Journal of Immunology.

[3]  S. Gaffen,et al.  An essential role of interleukin‐17 receptor signaling in the development of autoimmune glomerulonephritis , 2014, Journal of leukocyte biology.

[4]  D. Cua,et al.  Th17 cell development: from the cradle to the grave , 2013, Immunological reviews.

[5]  T. Mayadas,et al.  Interleukin-17 cytokines are critical in development of fatal lupus glomerulonephritis. , 2012, Immunity.

[6]  R. Panush.  Cutting edge: IL-23 receptor deficiency prevents the development of lupus nephritis in C57BL/6-lpr/lpr mice , 2011 .

[7]  Y. Iwakura,et al.  Th17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis. , 2010, Journal of the American Society of Nephrology : JASN.

[8]  V. Kuchroo,et al.  Cutting Edge: IL-23 Receptor Deficiency Prevents the Development of Lupus Nephritis in C57BL/6–lpr/lpr Mice , 2010, The Journal of Immunology.

[9]  G. Tsokos,et al.  IL-17 in Systemic Lupus Erythematosus , 2010, Journal of biomedicine & biotechnology.

[10]  L. Ivashkiv,et al.  Proliferative lesions and metalloproteinase activity in murine lupus nephritis mediated by type I interferons and macrophages , 2010, Proceedings of the National Academy of Sciences.

[11]  S. Gaffen.  The role of interleukin-17 in the pathogenesis of rheumatoid arthritis , 2009, Current rheumatology reports.

[12]  M. Schwartz,et al.  Evidence of concurrent immunopathological mechanisms determining the pathology of severe lupus nephritis , 2009, Lupus.

[13]  G. Cheng,et al.  The type I IFN induction pathway constrains Th17-mediated autoimmune inflammation in mice. , 2008, The Journal of clinical investigation.

[14]  P. Marrack,et al.  Genetic susceptibility to PolyI:C-induced IFNα/β-dependent accelerated disease in lupus-prone mice , 2006, Genes and Immunity.

[15]  P. Marrack,et al.  Genetic susceptibility to polyI:C-induced IFNalpha/beta-dependent accelerated disease in lupus-prone mice. , 2006, Genes and immunity.

[16]  P. Gregersen,et al.  The emerging role of interferon in human systemic lupus erythematosus. , 2004, Current opinion in immunology.

[17]  Sarah L. Gaffen,et al.  Functional Cooperation between Interleukin-17 and Tumor Necrosis Factor-α Is Mediated by CCAAT/Enhancer-binding Protein Family Members* , 2004, Journal of Biological Chemistry.

[18]  J. Demengeot,et al.  Type I Interferon controls the onset and severity of autoimmune manifestations in lpr mice. , 2003, Journal of autoimmunity.