Radiosensitization by Histone H3 Demethylase Inhibition in Diffuse Intrinsic Pontine Glioma
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A. Shilatifard | L. Zou | A. Seluanov | V. Gorbunova | S. Venneti | A. Piunti | Xiao Tian | A. Zhang | A. Kondo | O. Becher | S. Goldman | A. Carcaboso | R. Lulla | R. Hashizume | Kathryn Laurie | H. Katagi | Y. Shimazu | D. Unruh | Nundia Louis | Takahiro Sasaki | Q. Ma | Xingyao He | J. Lamano | Nitin R Wadhwani | Nitin R. Wadhwani | Akihide Kondo
[1] T. Yi,et al. Comprehensive profiling of JMJD3 in gastric cancer and its influence on patient survival , 2019, Scientific Reports.
[2] Krista M. Powell,et al. Targeted inhibition of histone H3K27 demethylation is effective in high-risk neuroblastoma , 2018, Science Translational Medicine.
[3] A. Sengupta,et al. KDM6 and KDM4 histone lysine demethylases emerge as molecular therapeutic targets in human acute myeloid leukemia. , 2017, Experimental hematology.
[4] S. Mueller,et al. New therapeutic approaches for brainstem tumors: a comparison of delivery routes using nanoliposomal irinotecan in an animal model , 2018, Journal of Neuro-Oncology.
[5] Y. Li,et al. Inhibitor of H3K27 demethylase JMJD3/UTX GSK-J4 is a potential therapeutic option for castration resistant prostate cancer , 2017, Oncotarget.
[6] E. Maury,et al. Epigenetic modification in chromatin machinery and its deregulation in pediatric brain tumors: Insight into epigenetic therapies , 2017, Epigenetics.
[7] B. Price,et al. Epigenetic therapy with inhibitors of histone methylation suppresses DNA damage signaling and increases glioma cell radiosensitivity , 2017, Oncotarget.
[8] R. Davis,et al. Inhibition of demethylase KDM6B sensitizes diffuse large B-cell lymphoma to chemotherapeutic drugs , 2017, Haematologica.
[9] N. Gupta,et al. Patient-derived Tumor Models for Diffuse Intrinsic Pontine Gliomas , 2016, Current neuropharmacology.
[10] M. Prados,et al. Inhibition of DNA damage repair by the CDK4/6 inhibitor palbociclib delays irradiated intracranial atypical teratoid rhabdoid tumor and glioblastoma xenograft regrowth. , 2016, Neuro-oncology.
[11] G. Reifenberger,et al. The 2016 World Health Organization Classification of Tumors of the Central Nervous System: a summary , 2016, Acta Neuropathologica.
[12] Nicholas J. Wang,et al. Functionally-defined Therapeutic Targets in Diffuse Intrinsic Pontine Glioma , 2015, Nature Medicine.
[13] D. Roos,et al. Randomized trial on radiotherapy for paediatric diffuse intrinsic pontine glioma (DIPG). , 2014, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.
[14] W. Huber,et al. Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2 , 2014, Genome Biology.
[15] C. James,et al. Pharmacologic inhibition of histone demethylation as a therapy for pediatric brainstem glioma , 2014, Nature Medicine.
[16] Jill S. Barnholtz-Sloan,et al. Childhood Brain Tumor Epidemiology: A Brain Tumor Epidemiology Consortium Review , 2014, Cancer Epidemiology, Biomarkers & Prevention.
[17] R. Jaenisch,et al. Contrasting roles for histone 3 lysine 27 demethylases in acute lymphoblastic leukemia , 2014, Nature.
[18] K. Helin,et al. The Histone Lysine Demethylase JMJD3/KDM6B Is Recruited to p53 Bound Promoters and Enhancer Elements in a p53 Dependent Manner , 2014, PloS one.
[19] M. Kieran,et al. Diffuse intrinsic pontine glioma: a reassessment , 2014, Journal of Neuro-Oncology.
[20] M. Berger,et al. Targeting Wee1 for the treatment of pediatric high-grade gliomas. , 2014, Neuro-oncology.
[21] David T. W. Jones,et al. Reduced H3K27me3 and DNA hypomethylation are major drivers of gene expression in K27M mutant pediatric high-grade gliomas. , 2013, Cancer cell.
[22] M. Lomax,et al. Biological consequences of radiation-induced DNA damage: relevance to radiotherapy. , 2013, Clinical oncology (Royal College of Radiologists (Great Britain)).
[23] G. Poirier,et al. Polycomb repressive complex 2 contributes to DNA double-strand break repair , 2013, Cell cycle.
[24] B. Garcia,et al. Inhibition of PRC2 Activity by a Gain-of-Function H3 Mutation Found in Pediatric Glioblastoma , 2013, Science.
[25] Sabine Mueller,et al. The histone H3.3K27M mutation in pediatric glioma reprograms H3K27 methylation and gene expression. , 2013, Genes & development.
[26] V. Marquez,et al. Inhibition of EZH2 suppresses self-renewal and induces radiation sensitivity in atypical rhabdoid teratoid tumor cells. , 2013, Neuro-oncology.
[27] K. Warren. Diffuse intrinsic pontine glioma: poised for progress , 2012, Front. Oncol..
[28] M. Prados,et al. Characterization of a diffuse intrinsic pontine glioma cell line: implications for future investigations and treatment , 2012, Journal of Neuro-Oncology.
[29] Christopher J. Schofield,et al. A selective jumonji H3K27 demethylase inhibitor modulates the proinflammatory macrophage response , 2012, Nature.
[30] E. Greer,et al. Histone methylation: a dynamic mark in health, disease and inheritance , 2012, Nature Reviews Genetics.
[31] L. Zitvogel,et al. Inflammasomes in carcinogenesis and anticancer immune responses , 2012, Nature Immunology.
[32] L. Ketonen,et al. Palliative Reirradiation for Progressive Diffuse Intrinsic Pontine Glioma , 2012, American journal of clinical oncology.
[33] M. Prados,et al. An experimental xenograft mouse model of diffuse pontine glioma designed for therapeutic testing , 2012, Journal of Neuro-Oncology.
[34] S. Baylin,et al. Oxidative damage targets complexes containing DNA methyltransferases, SIRT1, and polycomb members to promoter CpG Islands. , 2011, Cancer cell.
[35] S. Elledge,et al. A chromatin localization screen reveals poly (ADP ribose)-regulated recruitment of the repressive polycomb and NuRD complexes to sites of DNA damage , 2010, Proceedings of the National Academy of Sciences.
[36] Andrei Seluanov,et al. Analysis of DNA double-strand break (DSB) repair in mammalian cells. , 2010, Journal of visualized experiments : JoVE.
[37] Clifford A. Meyer,et al. Model-based Analysis of ChIP-Seq (MACS) , 2008, Genome Biology.
[38] S. Baylin,et al. Double Strand Breaks Can Initiate Gene Silencing and SIRT1-Dependent Onset of DNA Methylation in an Exogenous Promoter CpG Island , 2008, PLoS genetics.
[39] P. Sung,et al. Mechanism of eukaryotic homologous recombination. , 2008, Annual review of biochemistry.
[40] M. Lieber,et al. The Mechanism of Human Nonhomologous DNA End Joining* , 2008, Journal of Biological Chemistry.
[41] Pablo Tamayo,et al. Gene set enrichment analysis: A knowledge-based approach for interpreting genome-wide expression profiles , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[42] J. Ball,et al. Statistics review 12: Survival analysis , 2004, Critical care.
[43] Kai Rothkamm,et al. Pathways of DNA Double-Strand Break Repair during the Mammalian Cell Cycle , 2003, Molecular and Cellular Biology.
[44] Amar Gajjar,et al. Decline in corpus callosum volume among pediatric patients with medulloblastoma: longitudinal MR imaging study. , 2002, AJNR. American journal of neuroradiology.
[45] B. Moore,et al. Neurophysiological basis of cognitive deficits in long-term survivors of childhood cancer. , 1992, Archives of neurology.
[46] R. Brauner,et al. Growth and Endocrine Disorders Secondary to Cranial Irradiation , 1989, Pediatric Research.
[47] S. Holm. A Simple Sequentially Rejective Multiple Test Procedure , 1979 .