Prostate cancer of transition zone origin lacks TMPRSS2–ERG gene fusion

[1]  W. Gerald,et al.  TMPRSS2-ERG gene fusion is not associated with outcome in patients treated by prostatectomy. , 2009, Cancer research.

[2]  P. Scardino,et al.  Do Prostatic Transition Zone Tumors Have a Distinct Morphology? , 2008, The American journal of surgical pathology.

[3]  S. Varambally,et al.  Characterization of TMPRSS2:ETV5 and SLC45A3:ETV5 gene fusions in prostate cancer. , 2008, Cancer research.

[4]  E. Noel,et al.  Differential gene expression in the peripheral zone compared to the transition zone of the human prostate gland , 2008, Prostate Cancer and Prostatic Diseases.

[5]  J. Trachtenberg,et al.  Expression of the TMPRSS2:ERG fusion gene predicts cancer recurrence after surgery for localised prostate cancer , 2007, British Journal of Cancer.

[6]  M. Rubin,et al.  TMPRSS2-ERG fusion heterogeneity in multifocal prostate cancer: clinical and biologic implications. , 2007, Urology.

[7]  R. Shah,et al.  Heterogeneity of TMPRSS2 gene rearrangements in multifocal prostate adenocarcinoma: molecular evidence for an independent group of diseases. , 2007, Cancer research.

[8]  Debashis Ghosh,et al.  Comprehensive assessment of TMPRSS2 and ETS family gene aberrations in clinically localized prostate cancer , 2007, Modern Pathology.

[9]  S. Leung,et al.  Frequency of the TMPRSS2:ERG gene fusion is increased in moderate to poorly differentiated prostate cancers , 2007, Journal of Clinical Pathology.

[10]  N. Ragavan,et al.  Gene expression profiling of the human prostate zones , 2006, BJU international.

[11]  G. Jenster,et al.  Gene expression profiling of the human prostate zones , 2006, BJU international.

[12]  Arul M Chinnaiyan,et al.  TMPRSS2:ETV4 gene fusions define a third molecular subtype of prostate cancer. , 2006, Cancer research.

[13]  J. Tchinda,et al.  Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer. , 2006, Science.

[14]  Hideaki Miyake,et al.  A comparison of the biological features between prostate cancers arising in the transition and peripheral zones , 2005, BJU international.

[15]  Lang Li,et al.  Heterogeneity of Gleason grade in multifocal adenocarcinoma of the prostate , 2004, Cancer.

[16]  R. Cohen,et al.  Transition zone carcinoma of the prostate gland: a common indolent tumour type that occasionally manifests aggressive behaviour , 2003, Pathology.

[17]  M. Graefen,et al.  Tumour grade, proliferation, apoptosis, microvessel density, p53, and bcl-2 in prostate cancers: differences between tumours located in the transition zone and in the peripheral zone. , 2002, European urology.

[18]  D. Johnston,et al.  Detailed mapping of prostate carcinoma foci , 2000, Cancer.

[19]  T. Stamey,et al.  An analysis of 148 consecutive transition zone cancers: clinical and histological characteristics. , 2000, The Journal of urology.

[20]  L. Liotta,et al.  Evidence of independent origin of multiple tumors from patients with prostate cancer. , 1998, Journal of the National Cancer Institute.

[21]  L. Sullivan,et al.  Transition zone carcinoma of the prostate. , 1995, The Canadian journal of urology.

[22]  P. Scardino,et al.  A comparison of the morphological features of cancer arising in the transition zone and in the peripheral zone of the prostate. , 1991, The Journal of urology.

[23]  T. Stamey,et al.  Zonal Distribution of Prostatic Adenocarcinoma: Correlation with Histologic Pattern and Direction of Spread , 1988, The American journal of surgical pathology.

[24]  J. McNeal,et al.  Normal histology of the prostate. , 1988, The American journal of surgical pathology.

[25]  J. McNeal.  The zonal anatomy of the prostate , 1981, The Prostate.