Caged phosphopeptides reveal a temporal role for 14-3-3 in G1 arrest and S-phase checkpoint function

[1]  John S. Condeelis,et al.  Cofilin Promotes Actin Polymerization and Defines the Direction of Cell Motility , 2004, Science.

[2]  Michele Pagano,et al.  Degradation of Cdc25A by β-TrCP during S phase and in response to DNA damage , 2003, Nature.

[3]  H. Piwnica-Worms,et al.  Chk1 Kinase Negatively Regulates Mitotic Function of Cdc25A Phosphatase through 14-3-3 Binding , 2003, Molecular and Cellular Biology.

[4]  Michael B Yaffe,et al.  BRCT Repeats As Phosphopeptide-Binding Modules Involved in Protein Targeting , 2003, Science.

[5]  B. Imperiali,et al.  Caged phospho-amino acid building blocks for solid-phase peptide synthesis. , 2003, The Journal of organic chemistry.

[6]  M. Yaffe,et al.  Fluorescent caged phosphoserine peptides as probes to investigate phosphorylation-dependent protein associations. , 2003, Journal of the American Chemical Society.

[7]  Y. Sánchez,et al.  Regulation of Chk1 Includes Chromatin Association and 14-3-3 Binding following Phosphorylation on Ser-345* , 2003, Journal of Biological Chemistry.

[8]  Michael B Yaffe,et al.  Proteomic Screen Finds pSer/pThr-Binding Domain Localizing Plk1 to Mitotic Substrates , 2003, Science.

[9]  A. Aitken,et al.  A nonphosphorylated 14-3-3 binding motif on exoenzyme S that is functional in vivo. , 2002, European journal of biochemistry.

[10]  B. Imperiali,et al.  General method for the synthesis of caged phosphopeptides: tools for the exploration of signal transduction pathways. , 2002, Organic letters.

[11]  J. Avruch,et al.  14-3-3 Proteins: Active Cofactors in Cellular Regulation by Serine/Threonine Phosphorylation* , 2002, The Journal of Biological Chemistry.

[12]  D. Mochly‐Rosen,et al.  Molecular transporters for peptides: delivery of a cardioprotective epsilonPKC agonist peptide into cells and intact ischemic heart using a transport system, R(7). , 2001, Chemistry & biology.

[13]  H. Piwnica-Worms,et al.  14-3-3 binding regulates catalytic activity of human Wee1 kinase. , 2001, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

[14]  Tony Pawson,et al.  Multisite phosphorylation of a CDK inhibitor sets a threshold for the onset of DNA replication , 2001, Nature.

[15]  S. Masters,et al.  Functional conservation of 14-3-3 isoforms in inhibiting bad-induced apoptosis. , 2001, Experimental cell research.

[16]  J. Cadet,et al.  Direct and indirect effects of UV radiation on DNA and its components. , 2001, Journal of photochemistry and photobiology. B, Biology.

[17]  M. Yaffe,et al.  Phosphoserine/threonine-binding domains. , 2001, Current opinion in cell biology.

[18]  M. Yaffe,et al.  PhosphoSerine/threonine binding domains: you can't pSERious? , 2001, Structure.

[19]  M. Malumbres,et al.  Targeted Genomic Disruption of H-ras and N-ras, Individually or in Combination, Reveals the Dispensability of Both Loci for Mouse Growth and Development , 2001, Molecular and Cellular Biology.

[20]  J. Bartek,et al.  Pathways governing G1/S transition and their response to DNA damage , 2001, FEBS letters.

[21]  D. A. Dougherty,et al.  Tyrosine Decaging Leads to Substantial Membrane Trafficking during Modulation of an Inward Rectifier Potassium Channel , 2001, The Journal of general physiology.

[22]  Mong-Hong Lee,et al.  Association of the Cyclin-dependent Kinases and 14-3-3 Sigma Negatively Regulates Cell Cycle Progression* , 2000, The Journal of Biological Chemistry.

[23]  Y. Wang,et al.  Binding of 14-3-3beta to the carboxyl terminus of Wee1 increases Wee1 stability, kinase activity, and G2-M cell population. , 2000, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

[24]  K. Kinzler,et al.  14-3-3σ is required to prevent mitotic catastrophe after DNA damage , 1999, Nature.

[25]  E Ruoslahti,et al.  Isolation of high-affinity peptide antagonists of 14-3-3 proteins by phage display. , 1999, Biochemistry.

[26]  M. Yaffe,et al.  Structural analysis of 14-3-3 phosphopeptide complexes identifies a dual role for the nuclear export signal of 14-3-3 in ligand binding. , 1999, Molecular cell.

[27]  J. Decaprio,et al.  Cytoplasmic Localization of Human cdc25C during Interphase Requires an Intact 14-3-3 Binding Site , 1999, Molecular and Cellular Biology.

[28]  Xiao Zhen Zhou,et al.  Function of WW domains as phosphoserine- or phosphothreonine-binding modules. , 1999, Science.

[29]  Joseph Avruch,et al.  A dimeric 14-3-3 protein is an essential cofactor for Raf kinase activity , 1998, Nature.

[30]  R. Carraway,et al.  Signaling pathways underlying eosinophil cell motility revealed by using caged peptides. , 1998, Proceedings of the National Academy of Sciences of the United States of America.

[31]  A. Prochiantz,et al.  Trojan peptides: the penetratin system for intracellular delivery. , 1998, Trends in cell biology.

[32]  M. Yaffe,et al.  The Structural Basis for 14-3-3:Phosphopeptide Binding Specificity , 1997, Cell.

[33]  C. Peng,et al.  Mitotic and G2 checkpoint control: regulation of 14-3-3 protein binding by phosphorylation of Cdc25C on serine-216. , 1997, Science.

[34]  R. Liddington,et al.  Raf-1 Kinase and Exoenzyme S Interact with 14-3-3ζ through a Common Site Involving Lysine 49* , 1997, The Journal of Biological Chemistry.

[35]  R. Givens,et al.  New Photoactivated Protecting Groups. 6. p-Hydroxyphenacyl: A Phototrigger for Chemical and Biochemical Probes1,2 , 1997 .

[36]  R. Perlmutter,et al.  Positive and negative selection invoke distinct signaling pathways , 1996, The Journal of experimental medicine.

[37]  P. Allen,et al.  Interaction of 14-3-3 with Signaling Proteins Is Mediated by the Recognition of Phosphoserine , 1996, Cell.

[38]  A Aitken,et al.  Isoforms of 14‐3‐3 protein can form homo‐ and heterodimers in vivo and in vitro: implications for function as adapter proteins , 1995, FEBS letters.

[39]  D. Ogden,et al.  Kinetics of cytosolic Ca2+ concentration after photolytic release of 1-D-myo-inositol 1,4-bisphosphate 5-phosphorothioate from a caged derivative in guinea pig hepatocytes. , 1995, Biophysical journal.

[40]  A. Aitken,et al.  14-3-3 proteins on the MAP. , 1995, Trends in biochemical sciences.

[41]  A. Carr,et al.  14-3-3 protein homologs required for the DNA damage checkpoint in fission yeast. , 1994, Science.

[42]  G. Ellis‐Davies,et al.  Nitrophenyl-EGTA, a photolabile chelator that selectively binds Ca2+ with high affinity and releases it rapidly upon photolysis. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[43]  Wayne F. Patton,et al.  Mercury-arc photolysis: a method for examining second messenger regulation of endothelial cell monolayer integrity. , 1991, Analytical biochemistry.

[44]  A. Spector,et al.  Ultraviolet light induced DNA damage and repair in bovine lens epithelial cells. , 1990, Current eye research.

[45]  M. Lalande,et al.  A reversible arrest point in the late G1 phase of the mammalian cell cycle. , 1990, Experimental cell research.

[46]  G. Ellis‐Davies Development and application of caged calcium. , 2003, Methods in enzymology.

[47]  P. Doherty,et al.  Designing cell-permeant phosphopeptides to modulate intracellular signaling pathways. , 2001, Biopolymers.

[48]  H. Piwnica-Worms,et al.  14-3-3 Binding Regulates Catalytic Activity of Human Wee 1 Kinase 1 , 2001 .

[49]  S. Masters,et al.  14-3-3 proteins: structure, function, and regulation. , 2000, Annual review of pharmacology and toxicology.

[50]  F. D. de Gruijl Photocarcinogenesis: UVA vs UVB. , 2000, Methods in enzymology.

[51]  Yi Sun,et al.  Binding of 14-3-3 b to the Carboxyl Terminus of Wee 1 Increases Wee 1 Stability , Kinase Activity , and G 2M Cell Population , 2000 .

[52]  de Gruijl Fr Photocarcinogenesis: UVA vs UVB. , 2000, Methods in enzymology.