Immune system impairment and hepatic fibrosis in mice lacking the dioxin-binding Ah receptor
暂无分享,去创建一个
T. Pineau | S. Rudikoff | D. Hilbert | D. Nebert | F. Gonzalez | P. Fernández-Salguero | S. Kimura | T. McPhail | S. Lee | SS-Y Lee | DM Hilbert | DW Nebert | JM Ward | F. González | Jerrold M. Ward
[1] R F Standaert,et al. Inhibition of proteasome activities and subunit-specific amino-terminal threonine modification by lactacystin , 1995, Science.
[2] J. Abel,et al. Ah receptor in different tissues of C57BL/6J and DBA/2J mice: use of competitive polymerase chain reaction to measure Ah-receptor mRNA expression. , 1994, Archives of biochemistry and biophysics.
[3] L. Poellinger,et al. Identification of transactivation and repression functions of the dioxin receptor and its basic helix-loop-helix/PAS partner factor Arnt: inducible versus constitutive modes of regulation , 1994, Molecular and cellular biology.
[4] H. Li,et al. Transcriptional activation function of the mouse Ah receptor nuclear translocator. , 1994, The Journal of biological chemistry.
[5] L. Birnbaum. Endocrine effects of prenatal exposure to PCBs, dioxins, and other xenobiotics: implications for policy and future research. , 1994, Environmental health perspectives.
[6] P. Harper,et al. Molecular biology of the aromatic hydrocarbon (dioxin) receptor. , 1994, Trends in pharmacological sciences.
[7] R. Wells,et al. cDNA cloning, sequence analysis, and induction by aryl hydrocarbons of a murine cytochrome P450 gene, Cyp1b1. , 1994, DNA and cell biology.
[8] C. Jefcoate,et al. Mouse cytochrome P-450EF, representative of a new 1B subfamily of cytochrome P-450s. Cloning, sequence determination, and tissue expression. , 1994, The Journal of biological chemistry.
[9] L. Zhang,et al. Aryl hydrocarbon-induced interactions at multiple DNA elements of diverse sequence--a multicomponent mechanism for activation of cytochrome P4501A1 (CYP1A1) gene transcription. , 1994, Nucleic acids research.
[10] E. Jabs,et al. Complete cDNA sequence of a human dioxin-inducible mRNA identifies a new gene subfamily of cytochrome P450 that maps to chromosome 2. , 1994, The Journal of biological chemistry.
[11] O. Gotoh,et al. Interindividual difference in expression of human Ah receptor and related P450 genes. , 1994, Carcinogenesis.
[12] L. Birnbaum,et al. Ah receptor in embryonic mouse palate and effects of TCDD on receptor expression. , 1994, Toxicology and applied pharmacology.
[13] R. Pohjanvirta,et al. Developmental toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) in the most TCDD-resistant and -susceptible rat strains. , 1994, Toxicology and applied pharmacology.
[14] D. Nebert. Drug-metabolizing enzymes in ligand-modulated transcription. , 1994, Biochemical pharmacology.
[15] M. DeVito,et al. Dose-response relationships in mice following subchronic exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin: CYP1A1, CYP1A2, estrogen receptor, and protein tyrosine phosphorylation. , 1994, Toxicology and applied pharmacology.
[16] H. Swanson,et al. Cloning and expression of a human Ah receptor cDNA. , 1993, Molecular pharmacology.
[17] H. Swanson,et al. The AH-receptor: genetics, structure and function. , 1993, Pharmacogenetics.
[18] S. Rudikoff,et al. Long‐term thymic reconstitution by peripheral CD4 and CD8 single‐positive lymphocytes , 1993, European journal of immunology.
[19] Maria Teresa Landi,et al. Cancer Incidence in a Population Accidentally Exposed to 2,3,7,8‐Tetrachlorodibenzo-para‐dioxin , 1993, Epidemiology.
[20] M. Gallo,et al. Receptor mechanisms and dose-response models for the effects of dioxins. , 1993, Environmental health perspectives.
[21] D. Nebert,et al. Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1. , 1992, DNA and cell biology.
[22] O. Gotoh,et al. cDNA cloning and structure of mouse putative Ah receptor. , 1992, Biochemical and biophysical research communications.
[23] D. Nebert,et al. Human AH locus polymorphism and cancer: inducibility of CYP1A1 and other genes by combustion products and dioxin. , 1991, Pharmacogenetics.
[24] K. Dold,et al. Targets for dioxin: genes for plasminogen activator inhibitor-2 and interleukin-1 beta. , 1991, Science.
[25] O. Hankinson,et al. Cloning of a factor required for activity of the Ah (dioxin) receptor. , 1991, Science.
[26] J. Miners,et al. Drug glucuronidation in humans. , 1991, Pharmacology & therapeutics.
[27] D. Nebert,et al. Evolution of the P450 gene superfamily: animal-plant 'warfare', molecular drive and human genetic differences in drug oxidation. , 1990, Trends in genetics : TIG.
[28] M. Capecchi,et al. Altering the genome by homologous recombination. , 1989, Science.
[29] D W Nebert,et al. The Ah locus: genetic differences in toxicity, cancer, mutation, and birth defects. , 1989, Critical reviews in toxicology.
[30] F. Gonzalez,et al. The molecular biology of cytochrome P450s. , 1988, Pharmacological reviews.
[31] Mario R. Capecchi,et al. Disruption of the proto-oncogene int-2 in mouse embryo-derived stem cells: a general strategy for targeting mutations to non-selectable genes , 1988, Nature.
[32] S. Orrenius,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin kills immature thymocytes by Ca2+-mediated endonuclease activation. , 1988, Science.
[33] D. Nebert,et al. Tissue-specific expression of the mouse dioxin-inducible P(1)450 and P(3)450 genes: differential transcriptional activation and mRNA stability in liver and extrahepatic tissues , 1986, Molecular and cellular biology.
[34] D. Nebert,et al. Autoregulation plus upstream positive and negative control regions associated with transcriptional activation of the mouse P1(450) gene. , 1985, Nucleic acids research.
[35] A Poland,et al. 2,3,7,8-tetrachlorodibenzo-p-dioxin and related halogenated aromatic hydrocarbons: examination of the mechanism of toxicity. , 1982, Annual review of pharmacology and toxicology.
[36] Joseph F. Williams. Annual Review of Pharmacology , 1975 .