Hypoxia-induced PPFIA4 accelerates the progression of ovarian cancer through glucose metabolic reprogramming

[1]  Ziyou Lin,et al.  Retraction Note: Upregulation of OSBPL3 by HIF1A promotes colorectal cancer progression through activation of RAS signaling pathway , 2022, Cell Death and Disease.

[2]  Qianqian Zhou,et al.  PPFIA4 promotes castration-resistant prostate cancer by enhancing mitochondrial metabolism through MTHFD2 , 2022, Journal of Experimental & Clinical Cancer Research.

[3]  Q. Liao,et al.  TRPM7 silencing modulates glucose metabolic reprogramming to inhibit the growth of ovarian cancer by enhancing AMPK activation to promote HIF-1α degradation , 2022, Journal of Experimental & Clinical Cancer Research.

[4]  Chien-Hsiu Li,et al.  The Metabolism Reprogramming of microRNA Let-7-Mediated Glycolysis Contributes to Autophagy and Tumor Progression , 2021, International journal of molecular sciences.

[5]  Qing Yang,et al.  Establishment of a novel glycolysis-related prognostic gene signature for ovarian cancer and its relationships with immune infiltration of the tumor microenvironment , 2021, Journal of Translational Medicine.

[6]  Fangfang Bi,et al.  Establishment of a novel glycolysis-related prognostic gene signature for ovarian cancer and its relationships with immune infiltration of the tumor microenvironment , 2021, Journal of translational medicine.

[7]  Q. Liao,et al.  TRPM7 silencing modulates glucose metabolic reprogramming to inhibit the growth of ovarian cancer by enhancing AMPK activation to promote HIF-1α degradation , 2021, Journal of experimental & clinical cancer research : CR.

[8]  C. C. Wong,et al.  Hypoxia, Metabolic Reprogramming, and Drug Resistance in Liver Cancer , 2021, Cells.

[9]  Xinxiang Li,et al.  LncRNA MIR17HG promotes colorectal cancer liver metastasis by mediating a glycolysis-associated positive feedback circuit , 2021, Oncogene.

[10]  Nian Fu,et al.  PPFIA4 Promotes Colon Cancer Cell Proliferation and Migration by Enhancing Tumor Glycolysis , 2021, Frontiers in Oncology.

[11]  Lin Xu,et al.  circDCUN1D4 suppresses tumor metastasis and glycolysis in lung adenocarcinoma by stabilizing TXNIP expression , 2020, Molecular therapy. Nucleic acids.

[12]  Y. Tao,et al.  Epigenetic crosstalk between hypoxia and tumor driven by HIF regulation , 2020, Journal of experimental & clinical cancer research : CR.

[13]  M. Packer Mechanisms Leading to Differential Hypoxia Inducible Factor Signaling in the Diabetic Kidney: Modulation by SGLT2 Inhibitors and Hypoxia Mimetics. , 2020, American journal of kidney diseases : the official journal of the National Kidney Foundation.

[14]  Chandrasekhar Yadavalli,et al.  Ameliorative Effect of Ananas comosus on Cobalt Chloride-Induced Hypoxia in Caco2 cells via HIF-1α, GLUT 1, VEGF, ANG and FGF , 2020, Biological Trace Element Research.

[15]  Ziyou Lin,et al.  RETRACTED ARTICLE: Upregulation of OSBPL3 by HIF1A promotes colorectal cancer progression through activation of RAS signaling pathway , 2020, Cell Death & Disease.

[16]  W. Xue,et al.  Hypoxia-induced GBE1 expression promotes tumor progression through metabolic reprogramming in lung adenocarcinoma , 2020, Signal Transduction and Targeted Therapy.

[17]  Lei Wu,et al.  STIM1 is a metabolic checkpoint regulating the invasion and metastasis of hepatocellular carcinoma , 2020, Theranostics.

[18]  A. Schürmann,et al.  Polymorphisms in miRNA binding sites involved in metabolic diseases in mice and humans , 2020, Scientific Reports.

[19]  B. Tang Glucose, glycolysis, and neurodegenerative diseases , 2020, Journal of cellular physiology.

[20]  C. Sarkar,et al.  NFкB is a critical transcriptional regulator of atypical cadherin FAT1 in glioma , 2020, BMC Cancer.

[21]  A. Façanha,et al.  Multi-cancer V-ATPase molecular signatures: A distinctive balance of subunit C isoforms in esophageal carcinoma , 2020, EBioMedicine.

[22]  V. Fellman,et al.  A sensitive assay for dNTPs based on long synthetic oligonucleotides, EvaGreen dye and inhibitor-resistant high-fidelity DNA polymerase , 2019, bioRxiv.

[23]  M. Xie,et al.  Role of hypoxia in cancer therapy by regulating the tumor microenvironment , 2019, Molecular Cancer.

[24]  Phillip A. Richmond,et al.  JASPAR 2020: update of the open-access database of transcription factor binding profiles , 2019, Nucleic Acids Res..

[25]  T. Kietzmann,et al.  DUBs, Hypoxia, and Cancer. , 2019, Trends in cancer.

[26]  Gang Yang,et al.  The enhancement of glycolysis regulates pancreatic cancer metastasis , 2019, Cellular and Molecular Life Sciences.

[27]  Y. Hayashi,et al.  Hypoxia/pseudohypoxia‐mediated activation of hypoxia‐inducible factor‐1α in cancer , 2019, Cancer science.

[28]  Masayo Umebayashi,et al.  Liprin-α4 as a New Therapeutic Target for SCLC as an Upstream Mediator of HIF1α , 2019, AntiCancer Research.

[29]  Zhi-Yong Wu,et al.  YWHAZ promotes ovarian cancer metastasis by modulating glycolysis. , 2018, Oncology reports.

[30]  K. Selvendiran,et al.  Hypoxia-induced exosomes contribute to a more aggressive and chemoresistant ovarian cancer phenotype: a novel mechanism linking STAT3/Rab proteins , 2018, Oncogene.

[31]  J. Zenklusen,et al.  SnapShot: TCGA-Analyzed Tumors , 2018, Cell.

[32]  Lei Jin,et al.  LncRNA IDH1-AS1 links the functions of c-Myc and HIF1α via IDH1 to regulate the Warburg effect , 2018, Proceedings of the National Academy of Sciences.

[33]  N. Hattori,et al.  Carboplatin plus Weekly Paclitaxel Combined with Bevacizumab as First-line Treatment for Non-small Cell Lung Cancer. , 2017, Anticancer research.

[34]  S. Påhlman,et al.  Therapeutic targeting of hypoxia and hypoxia-inducible factors in cancer. , 2016, Pharmacology & therapeutics.

[35]  F. Gage,et al.  Metabolic reprogramming during neuronal differentiation from aerobic glycolysis to neuronal oxidative phosphorylation , 2016, eLife.

[36]  Hong Zhu,et al.  Wortmannin influences hypoxia-inducible factor-1 alpha expression and glycolysis in esophageal carcinoma cells. , 2016, World journal of gastroenterology.

[37]  E. Rankin,et al.  Hypoxic control of metastasis , 2016, Science.

[38]  Wei Li,et al.  HIF-1α pathway: role, regulation and intervention for cancer therapy , 2015, Acta pharmaceutica Sinica. B.

[39]  J. Geschwind,et al.  Tumor glycolysis as a target for cancer therapy: progress and prospects , 2013, Molecular Cancer.

[40]  L. Neckers,et al.  Molecular chaperone TRAP1 regulates a metabolic switch between mitochondrial respiration and aerobic glycolysis , 2013, Proceedings of the National Academy of Sciences.

[41]  M. Sachs,et al.  Liprin-α4 is a new hypoxia-inducible target gene required for maintenance of cell-cell contacts. , 2010, Experimental cell research.

[42]  C. Cruciat,et al.  Requirement of Prorenin Receptor and Vacuolar H+-ATPase–Mediated Acidification for Wnt Signaling , 2010, Science.

[43]  Yibin Kang,et al.  The Multifaceted Role of MTDH/AEG-1 in Cancer Progression , 2009, Clinical Cancer Research.

[44]  F. Lee,et al.  YC-1 inhibits HIF-1 expression in prostate cancer cells: contribution of Akt/NF-κB signaling to HIF-1α accumulation during hypoxia , 2007, Oncogene.

[45]  A. Harris,et al.  GLUT‐1 and CAIX as intrinsic markers of hypoxia in carcinoma of the cervix: Relationship to pimonidazole binding , 2003, International journal of cancer.

[46]  Yuan Zhang,et al.  Identification of a Protein with Homology to hsp90 That Binds the Type 1 Tumor Necrosis Factor Receptor (*) , 1995, The Journal of Biological Chemistry.

[47]  F. Esposito,et al.  New insights into TRAP1 pathway. , 2012, American journal of cancer research.

[48]  F. Lee,et al.  YC-1 inhibits HIF-1 expression in prostate cancer cells: contribution of Akt/NF-kappaB signaling to HIF-1alpha accumulation during hypoxia. , 2007, Oncogene.

[49]  T. Acker,et al.  Hypoxia and hypoxia inducible factors (HIF) as important regulators of tumor physiology. , 2004, Cancer treatment and research.