Spinal mechanisms of acute and persistent pain.

[1]  F. Lembeck,et al.  Substance P in peripheral sensory processes. , 2008, Ciba Foundation symposium.

[2]  C. Epstein,et al.  Primary afferent tachykinins are required to experience moderate to intense pain , 1998, Nature.

[3]  A. Basbaum,et al.  Inflammation Increases the Distribution of Dorsal Horn Neurons That Internalize the Neurokinin-1 Receptor in Response to Noxious and Non-Noxious Stimulation , 1997, The Journal of Neuroscience.

[4]  S. Tonegawa,et al.  Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma. , 1997, Science.

[5]  W. Snider,et al.  IB4-Binding DRG Neurons Switch from NGF to GDNF Dependence in Early Postnatal Life , 1997, Neuron.

[6]  A. Basbaum,et al.  Diminished Inflammation and Nociceptive Pain with Preservation of Neuropathic Pain in Mice with a Targeted Mutation of the Type I Regulatory Subunit of cAMP-Dependent Protein Kinase , 1997, The Journal of Neuroscience.

[7]  R. North,et al.  Immunohistochemical study of the P2X2 and P2X3 receptor subunits in rat and monkey sensory neurons and their central terminals , 1997, Neuropharmacology.

[8]  B J Allen,et al.  Noxious Cutaneous Thermal Stimuli Induce a Graded Release of Endogenous Substance P in the Spinal Cord: Imaging Peptide ActionIn Vivo , 1997, The Journal of Neuroscience.

[9]  A. Basbaum,et al.  NMDA-receptor regulation of substance P release from primary afferent nociceptors , 1997, Nature.

[10]  Y. de Koninck,et al.  Quantitative analysis of substance P‐immunoreactive boutons on physiologically characterized dorsal horn neurons in the cat lumbar spinal cord , 1996, The Journal of comparative neurology.

[11]  J. L. Brown,et al.  Spinal cord substance P receptor immunoreactivity increases in both inflammatory and nerve injury models of persistent pain , 1996, Neuroscience.

[12]  E. Kandel,et al.  A genetic test of the effects of mutations in PKA on mossy fiber ltp and its relation to spatial and contextual learning , 1995, Cell.

[13]  S. Tonegawa,et al.  Impaired synapse elimination during cerebellar development in PKCγ mutant mice , 1995, Cell.

[14]  C. Maggi,et al.  The mammalian tachykinin receptors. , 1995, General pharmacology.

[15]  A. Basbaum,et al.  Receptor endocytosis and dendrite reshaping in spinal neurons after somatosensory stimulation. , 1995, Science.

[16]  A. Basbaum,et al.  Morphological characterization of substance P receptor‐immunoreactive neurons in the rat spinal cord and trigeminal nucleus caudalis , 1995, The Journal of comparative neurology.

[17]  A I Basbaum,et al.  Synaptic relationship between substance P and the substance P receptor: light and electron microscopic characterization of the mismatch between neuropeptides and their receptors. , 1994, Proceedings of the National Academy of Sciences of the United States of America.

[18]  C. Woolf,et al.  Preemptive analgesia--treating postoperative pain by preventing the establishment of central sensitization. , 1993 .

[19]  J. Henry,et al.  Excitatory amino acid receptor mediation of sensory inputs to functionally identified dorsal horn neurons in cat spinal cord , 1993, Neuroscience.

[20]  M. Satoh,et al.  Detection of capsaicin-evoked release of glutamate from spinal dorsal horn slices of rat with on-line monitoring system , 1993, Neuroscience Letters.

[21]  A I Basbaum,et al.  Peptides and the primary afferent nociceptor , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[22]  W. Willis,et al.  Combined application of excitatory amino acids and substance P produces long-lasting changes in responses of primate spinothalamic tract neurons , 1993, Brain Research Reviews.

[23]  G. Gebhart,et al.  Nitric oxide (NO) and nociceptive processing in the spinal cord , 1993, Pain.

[24]  R. Lefkowitz,et al.  Beta-adrenergic receptor sequestration. A potential mechanism of receptor resensitization. , 1993, The Journal of biological chemistry.

[25]  Y. Koninck,et al.  Spinal neurons exhibiting a specific nociceptive response receive abundant substance P-containing synaptic contacts. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[26]  G. Bennett,et al.  Increased neuropeptide Y (NPY)-like immunoreactivity in rat sensory neurons following peripheral axotomy , 1991, Neuroscience Letters.

[27]  H. Schaible,et al.  Release, spread and persistence of immunoreactive neurokinin A in the dorsal horn of the cat following noxious cutaneous stimulation. Studies with antibody microprobes , 1990, Neuroscience.

[28]  Ronald Dubner,et al.  A novel behavioral model of neuropathic pain disorders produced in rats by partial sciatic nerve injury , 1990, Pain.

[29]  M. Randić,et al.  Substance P modulates glutamate-induced currents in acutely isolated rat spinal dorsal horn neurones , 1990, Neuroscience Letters.

[30]  E. Goetzl,et al.  Mediation of primary afferent peripheral hyperalgesia by the cAMP second messenger system , 1989, Neuroscience.

[31]  T. Hökfelt,et al.  Neuropeptide expression in rat dorsal root ganglion cells and spinal cord after peripheral nerve injury with special reference to galanin , 1989, Neuroscience.

[32]  H. Frenk,et al.  Is substance P a primary afferent neurotransmitter for noniceptive input? II. Spinalization does not reduce and intrathecal morphine potentiates behavioral responses to substance P , 1988, Brain Research.

[33]  W. D. Hutchinson,et al.  Cutaneous stimuli releasing immunoreactive substance P in the dorsal horn of the cat , 1988, Brain Research.

[34]  J. Chirgwin,et al.  Three rat preprotachykinin mRNAs encode the neuropeptides substance P and neurokinin A. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[35]  H. Takagi,et al.  Evidence that substance P and somatostatin transmit separate information related to pain in the spinal dorsal horn , 1985, Brain Research.

[36]  G. Wilcox,et al.  Intrathecal substance P elicits a caudally-directed biting and scratching behavior in mice , 1981, Brain Research.

[37]  F. Lembeck,et al.  Increase of substance P in primary afferent nerves during chronic pain , 1981, Neuropeptides.

[38]  T. Jessell,et al.  Intrathecal morphine inhibits substance P release from mammalian spinal cord in vivo , 1980, Nature.

[39]  D. Dubuisson,et al.  The formalin test: A quantitative study of the analgesic effects of morphine, meperidine, and brain stem stimulation in rats and cats , 1977, Pain.

[40]  P. Wall,et al.  The laminar organization of dorsal horn and effects of descending impulses , 1967, The Journal of physiology.

[41]  T. Yamamoto,et al.  Stereospecific effects of a nonpeptidic NK1 selective antagonist, CP-96,345: antinociception in the absence of motor dysfunction. , 1991, Life sciences.

[42]  L. Kruger,et al.  Acid phosphatase as a selective marker for a class of small sensory ganglion cells in several mammals: spinal cord distribution, histochemical properties, and relation to fluoride-resistant acid phosphatase (FRAP) of rodents. , 1988, Somatosensory research.