Aspartame-fed zebrafish exhibit acute deaths with swimming defects and saccharin-fed zebrafish have elevation of cholesteryl ester transfer protein activity in hypercholesterolemia.
暂无分享,去创建一个
[1] D. Souza,et al. Chronic ethanol treatment alters purine nucleotide hydrolysis and nucleotidase gene expression pattern in zebrafish brain. , 2011, Neurotoxicology.
[2] Kyung-Hyun Cho,et al. Water extracts of cinnamon and clove exhibits potent inhibition of protein glycation and anti-atherosclerotic activity in vitro and in vivo hypolipidemic activity in zebrafish. , 2011, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[3] Jeroen Bakkers,et al. Zebrafish as a model to study cardiac development and human cardiac disease , 2011, Cardiovascular research.
[4] N. Jeoung,et al. Modified apolipoprotein (apo) A-I by artificial sweetener causes severe premature cellular senescence and atherosclerosis with impairment of functional and structural properties of apoA-I in lipid-free and lipid-bound state , 2011, Molecules and cells.
[5] M. Cayuela,et al. Behaviour of Telomere and Telomerase during Aging and Regeneration in Zebrafish , 2011, PloS one.
[6] L. Santarelli,et al. In vitro effect of aspartame in angiogenesis induction. , 2011, Toxicology in vitro : an international journal published in association with BIBRA.
[7] M. Endres,et al. The Role of Fasting Versus Non-Fasting Triglycerides in Ischemic Stroke: A Systematic Review , 2010, Front. Neur..
[8] M. Sarras,et al. Limb regeneration is impaired in an adult zebrafish model of diabetes mellitus , 2010, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.
[9] M. Davidson. Update on CETP inhibition. , 2010, Journal of clinical lipidology.
[10] E. Dennis,et al. Oxidized Cholesteryl Esters and Phospholipids in Zebrafish Larvae Fed a High Cholesterol Diet , 2010, The Journal of Biological Chemistry.
[11] E. Ikonen,et al. Zebrafish: gaining popularity in lipid research. , 2010, The Biochemical journal.
[12] Dong-gu Shin,et al. Senescence-related truncation and multimerization of apolipoprotein A-I in high-density lipoprotein with an elevated level of advanced glycated end products and cholesteryl ester transfer activity. , 2010, The journals of gerontology. Series A, Biological sciences and medical sciences.
[13] Dong-gu Shin,et al. The functional and compositional properties of lipoproteins are altered in patients with metabolic syndrome with increased cholesteryl ester transfer protein activity. , 2009, International journal of molecular medicine.
[14] C. Burkhart. 'Lone' atrial fibrillation precipitated by monosodium glutamate and aspartame. , 2009, International journal of cardiology.
[15] Soo-Ho Choi,et al. Toll-like receptor-4 and lipoprotein accumulation in macrophages. , 2009, Trends in cardiovascular medicine.
[16] Kyung-Hyun Cho,et al. Biomedicinal implications of high-density lipoprotein: its composition, structure, functions, and clinical applications. , 2009, BMB reports.
[17] Soo-Ho Choi,et al. Vascular Lipid Accumulation, Lipoprotein Oxidation, and Macrophage Lipid Uptake in Hypercholesterolemic Zebrafish , 2009, Circulation research.
[18] Kyung-Hyun Cho,et al. Synthesis of reconstituted high density lipoprotein (rHDL) containing apoA-I and apoC-III: the functional role of apoC-III in rHDL , 2009, Molecules and cells.
[19] Dong-gu Shin,et al. Myocardial infarction patients show altered lipoprotein properties and functions when compared with stable angina pectoris patients , 2009, Experimental & Molecular Medicine.
[20] E. Pretorius,et al. Direct and indirect cellular effects of aspartame on the brain , 2008, European Journal of Clinical Nutrition.
[21] Utpal Banerjee,et al. Distinct mitochondrial retrograde signals control the G1-S cell cycle checkpoint , 2008, Nature Genetics.
[22] M. Soffritti,et al. Consequences of exposure to carcinogens beginning during developmental life. , 2008, Basic & clinical pharmacology & toxicology.
[23] A. Mukherjee,et al. Genotoxicity Testing of Low-Calorie Sweeteners: Aspartame, Acesulfame-K, and Saccharin , 2008, Drug and chemical toxicology.
[24] B. P. Bergstrom,et al. Aspartame decreases evoked extracellular dopamine levels in the rat brain: An in vivo voltammetry study , 2007, Neuropharmacology.
[25] P. McBride,et al. Triglycerides and risk for coronary heart disease. , 2007, JAMA.
[26] P. Libby. Fat fuels the flame: triglyceride-rich lipoproteins and arterial inflammation. , 2007, Circulation research.
[27] L. Lambertini,et al. First Experimental Demonstration of the Multipotential Carcinogenic Effects of Aspartame Administered in the Feed to Sprague-Dawley Rats , 2005, Environmental health perspectives.
[28] V. Diehl,et al. Artificial sweeteners--do they bear a carcinogenic risk? , 2004, Annals of oncology : official journal of the European Society for Medical Oncology.
[29] L. Zon,et al. Fishing for lymphoid genes. , 2001, Trends in immunology.
[30] R. Morton. Cholesteryl ester transfer protein and its plasma regulator: lipid transfer inhibitor protein. , 1999, Current opinion in lipidology.
[31] E. Spitznagel,et al. Increasing Brain Tumor Rates: Is There a Link to Aspartame? , 1996, Journal of neuropathology and experimental neurology.
[32] C. Berlin,et al. Effect of Dietary Aspartame on Plasma Concentrations of Phenylalanine and Tyrosine in Normal and Homozygous Phenylketonuric Patients , 1992, Clinical pediatrics.
[33] D. W. Andersen,et al. Metabolism of aspartame and its L-phenylalanine methyl ester decomposition product by the porcine gut. , 1991, Metabolism: clinical and experimental.
[34] P. J. Babin,et al. Plasma lipoproteins in fish. , 1989, Journal of lipid research.
[35] R. Coulombe,et al. Effects of repeated doses of aspartame on serotonin and its metabolite in various regions of the mouse brain. , 1987, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.
[36] M. D. Reuber. Carcinogenicity of saccharin. , 1978, Environmental health perspectives.
[37] Kyung-Hyun Cho,et al. The function, composition, and particle size of high-density lipoprotein were severely impaired in an oliguric phase of hemorrhagic fever with renal syndrome patients. , 2008, Clinical biochemistry.
[38] J. Olney. Excitotoxins in foods. , 1994, Neurotoxicology.