Impairment of T-cell growth-promoting lymphokines in human insulin-dependent diabetes mellitus

[1]  N. Terada,et al.  IL-4 and IL-2 promote human T-cell proliferation through symmetrical but independent pathways. , 1992, Clinical immunology and immunopathology.

[2]  O. Majdic,et al.  In vitro and in vivo activated T cells display increased sensitivity to PGE2. , 1991, Cellular immunology.

[3]  K. Zier,et al.  Low interleukin-2 synthesis by type 1 diabetics is regulated at the pretranslational level. , 1991, Clinical immunology and immunopathology.

[4]  L. Harrison,et al.  Evidence for IL-6 production by and effects on the pancreatic beta-cell. , 1989, Journal of Immunology.

[5]  W. Paul.  Pleiotropy and redundancy: T cell-derived lymphokines in the immune response , 1989, Cell.

[6]  A. López-Rivas,et al.  Prostaglandin E2 and the increase of intracellular cAMP inhibit the expression of interleukin 2 receptors in human T cells , 1988, European journal of immunology.

[7]  A. Dalgleish,et al.  The interleukins in acquired disease. , 1988, Clinical and experimental immunology.

[8]  D. Eizirik,et al.  Functional Characteristics of Rat Pancreatic Islets Maintained in Culture After Exposure to Human Interleukin 1 , 1988, Diabetes.

[9]  K. Amano,et al.  Evidence for Initial Involvement of Macrophage in Development of Insulitis in NOD Mice , 1988, Diabetes.

[10]  K. Arai,et al.  Coordinate regulation of immune and inflammatory responses by T cell‐derived lymphokines , 1988, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[11]  A. Thomson,et al.  The influence of cyclosporin A on cell-mediated immunity. , 1988, Clinical and experimental immunology.

[12]  J. Nerup,et al.  Human tumor necrosis factor potentiates human interleukin 1-mediated rat pancreatic beta-cell cytotoxicity. , 1987, Journal of immunology.

[13]  C. R. Kahn,et al.  Acquired defect in interleukin-2 production in patients with type I diabetes mellitus. , 1986, The New England journal of medicine.

[14]  Hoskins,et al.  PATHOGENESIS OF INSULIN-DEPENDENT DIABETES: A ROLE FOR ACTIVATED T LYMPHOCYTES , 1984, The Lancet.

[15]  R. Spielman,et al.  Decreased Synthesis of Interleukin-2 (IL-2) in Insulin-dependent Diabetes Mellitus , 1984, Diabetes.

[16]  T. Seemayer,et al.  Immune dysfunction in diabetes-prone BB rats. Interleukin 2 production and other mitogen-induced responses are suppressed by activated macrophages , 1984, The Journal of experimental medicine.

[17]  D. Cantrell,et al.  Transient expression of interleukin 2 receptors. Consequences for T cell growth , 1983, The Journal of experimental medicine.

[18]  J. Ceuppens,et al.  Regulation of the immune response by prostaglandins , 1983, Journal of Clinical Immunology.

[19]  S. Baba,et al.  Suppression of concanavalin A-induced responses in splenic lymphocytes by activated macrophages in the non-obese diabetic mouse , 2004, Diabetologia.

[20]  G. Eisenbarth,et al.  Type-I diabetes: a chronic autoimmune disease of human, mouse, and rat. , 1990, Annual review of immunology.

[21]  M. Plaut.  Lymphocyte hormone receptors. , 1987, Annual review of immunology.

[22]  G. Bottazzo,et al.  Islet-cell and insulin autoantibodies in diabetes , 1987 .

[23]  D. Webb,et al.  Regulation of the immune response by prostaglandins. , 1980, Clinical immunology and immunopathology.