Prolonged High-Fat Feeding Enhances Aortic 18F-FDG and 99mTc-Annexin A5 Uptake in Apolipoprotein E-Deficient and Wild-Type C57BL/6J Mice
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F. Blankenberg | N. Tamaki | H. Strauss | Yan Zhao | Songji Zhao | Y. Kuge
[1] A. Becker,et al. Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology. , 1994, Circulation.
[2] C. Remacle,et al. Triggering of inflammatory response by myeloperoxidase-oxidized LDL. , 2006, Biochemistry and cell biology = Biochimie et biologie cellulaire.
[3] R. Virmani,et al. Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions. , 2000, Arteriosclerosis, thrombosis, and vascular biology.
[4] M. She,et al. Minimally modified low-density lipoprotein induces monocyte chemotactic protein-1 expression in vivo and a novel model for monocyte adhesion to arterial intima. , 1999, Chinese medical journal.
[5] P. Libby,et al. Inflammation and atherosclerosis: role of C-reactive protein in risk assessment. , 2004, The American journal of medicine.
[6] Shuiqing Yu,et al. Diabetes Induces Endothelial Dysfunction but Does Not Increase Neointimal Formation in High-Fat Diet Fed C57BL/6J Mice , 2005, Circulation research.
[7] R. Virmani,et al. Targeting of Apoptotic Macrophages and Experimental Atheroma With Radiolabeled Annexin V: A Technique With Potential for Noninvasive Imaging of Vulnerable Plaque , 2003, Circulation.
[8] M. Seldin,et al. Control of Expression of Insulin Resistance and Hyperglycemia by Different Genetic Factors in Diabetic C57BL/6J Mice , 1991, Diabetes.
[9] A. J. Valente,et al. Monocyte transmigration induced by modification of low density lipoprotein in cocultures of human aortic wall cells is due to induction of monocyte chemotactic protein 1 synthesis and is abolished by high density lipoprotein. , 1991, The Journal of clinical investigation.
[10] J. Manson,et al. C-reactive protein, interleukin 6, and risk of developing type 2 diabetes mellitus. , 2001, JAMA.
[11] H. Tsutsui,et al. Fatty acids increase the circulating levels of oxidative stress factors in mice with diet‐induced obesity via redox changes of albumin , 2007, The FEBS journal.
[12] H. Strauss,et al. Molecular imaging in nuclear cardiology. , 2004, Seminars in nuclear medicine.
[13] P. Ridker,et al. Increased inflammatory markers in children with familial hypercholesterolaemia , 2006, European journal of clinical investigation.
[14] M. Feinglos,et al. Diet-Induced Type II Diabetes in C57BL/6J Mice , 1988, Diabetes.
[15] M. Davies,et al. Atherosclerotic plaque caps are locally weakened when macrophages density is increased. , 1991, Atherosclerosis.
[16] A. Freiman,et al. Association of vascular 18F-FDG uptake with vascular calcification. , 2005, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[17] Masatoshi Ishibashi,et al. Simvastatin attenuates plaque inflammation: evaluation by fluorodeoxyglucose positron emission tomography. , 2006, Journal of the American College of Cardiology.
[18] G. Chisolm,et al. Oxidized LDL-induced injury and apoptosis in atherosclerosis. Potential roles for oxysterols. , 2001, Trends in cardiovascular medicine.
[19] P. Libby,et al. Evidence for apoptosis in advanced human atheroma. Colocalization with interleukin-1 beta-converting enzyme. , 1995, The American journal of pathology.
[20] Renu Virmani,et al. Pathology of the thin-cap fibroatheroma: a type of vulnerable plaque. , 2003, Journal of interventional cardiology.
[21] L Wood,et al. Adipose tissue tumor necrosis factor and interleukin-6 expression in human obesity and insulin resistance. , 2001, American journal of physiology. Endocrinology and metabolism.
[22] H. Watabe,et al. (18)F-FDG accumulation in atherosclerotic plaques: immunohistochemical and PET imaging study. , 2004, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[23] S. Ylä-Herttuala. Oxidized LDL and atherogenesis. , 1999, Annals of the New York Academy of Sciences.
[24] Yuan-Yuan Shi,et al. Hyperhomocysteinemia induced by methionine supplementation does not independently cause atherosclerosis in C57BL/6J mice , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[25] A. Hamsten,et al. Oxidized low density lipoprotein induces differentiation and adhesion of human monocytes and the monocytic cell line U937. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[26] J. Breslow,et al. The emergence of mouse models of atherosclerosis and their relevance to clinical research , 1997, Journal of internal medicine.
[27] B. Björkerud,et al. Apoptosis is abundant in human atherosclerotic lesions, especially in inflammatory cells (macrophages and T cells), and may contribute to the accumulation of gruel and plaque instability. , 1996, The American journal of pathology.
[28] A. Alavi,et al. F-18 FDG Uptake in the Large Arteries: A New Observation , 2001, Clinical nuclear medicine.
[29] A. Nègre-Salvayre,et al. Oxidized low-density lipoprotein-induced apoptosis. , 2002, Biochimica et biophysica acta.
[30] C Bogardus,et al. Circulating interleukin-6 in relation to adiposity, insulin action, and insulin secretion. , 2001, Obesity research.
[31] P. Standley,et al. Insulin and insulin-like growth factor-1 modulation of glucose transport in arterial smooth muscle cells: implication of GLUT-4 in the vasculature. , 1994, American journal of hypertension.
[32] Eran Leitersdorf,et al. Atherosclerosis in the apolipoprotein-E-deficient mouse: a decade of progress. , 2004, Arteriosclerosis, thrombosis, and vascular biology.
[33] Nagara Tamaki,et al. Detection of apoptotic tumor response in vivo after a single dose of chemotherapy with 99mTc-annexin V. , 2003, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[34] D. Connolly,et al. Regulation of hexose transport in aortic endothelial cells by vascular permeability factor and tumor necrosis factor-alpha, but not by insulin. , 1990, The Journal of biological chemistry.
[35] D. Johns,et al. Decreased vascular glucose transporter expression and glucose uptake in DOCA-salt hypertension , 2001, Journal of hypertension.
[36] C. Glass,et al. Mildly oxidized low density lipoprotein activates multiple apoptotic signaling pathways in human coronary cells , 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[37] T. Fryer,et al. Targeting the vulnerable plaque: The evolving role of nuclear imaging , 2005, Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology.
[38] F. Blankenberg,et al. Comparison of 99mTc-annexin A5 with 18F-FDG for the detection of atherosclerosis in ApoE−/− mice , 2007, European Journal of Nuclear Medicine and Molecular Imaging.
[39] M. Daemen,et al. Noninvasive detection of plaque instability with use of radiolabeled annexin A5 in patients with carotid-artery atherosclerosis. , 2004, The New England journal of medicine.
[40] R. Virmani,et al. Noninvasive imaging of atherosclerotic lesions in apolipoprotein E-deficient and low-density-lipoprotein receptor-deficient mice with annexin A5. , 2006, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.