A putative loop connection between VTA dopamine neurons and nucleus accumbens encodes positive valence to compensate for hunger
暂无分享,去创建一个
Lei Xiao | Qiuping Tong | Xiao Cui | Hao Xu | Chuantong Xie
[1] G. Bi,et al. Distinct reward processing by subregions of the nucleus accumbens. , 2023, Cell reports.
[2] Xiaoshuang Zhang,et al. D1 receptor-expressing neurons in ventral tegmental area alleviate mouse anxiety-like behaviors via glutamatergic projection to lateral septum , 2022, Molecular Psychiatry.
[3] Camarin E. Rolle,et al. Pilot study of responsive nucleus accumbens deep brain stimulation for loss-of-control eating , 2022, Nature Medicine.
[4] Chang-lin Li,et al. A circuit from lateral septum neurotensin neurons to tuberal nucleus controls hedonic feeding , 2022, Molecular Psychiatry.
[5] M. Krashes,et al. Acts of appetite: neural circuits governing the appetitive, consummatory, and terminating phases of feeding , 2022, Nature Metabolism.
[6] Yanlin He,et al. A D2 to D1 shift in dopaminergic inputs to midbrain 5-HT neurons causes anorexia in mice , 2022, Nature Neuroscience.
[7] Lei Xiao,et al. Morpho-Electric Properties and Diversity of Oxytocin Neurons in Paraventricular Nucleus of Hypothalamus in Female and Male Mice , 2022, The Journal of Neuroscience.
[8] Yiming Zhou,et al. A distinct D1-MSN subpopulation down-regulates dopamine to promote negative emotional state , 2021, Cell Research.
[9] D. Mahadevia,et al. Dopamine promotes aggression in mice via ventral tegmental area to lateral septum projections , 2021, Nature Communications.
[10] J. Betley,et al. Reverse-translational identification of a cerebellar satiation network , 2021, Nature.
[11] M. Horiuchi,et al. Refeeding activates neurons in the dorsomedial hypothalamus to inhibit food intake and promote positive valence , 2021, Molecular metabolism.
[12] Feng Liu,et al. 5-HT recruits distinct neurocircuits to inhibit hunger-driven and non-hunger-driven feeding , 2021, Molecular Psychiatry.
[13] Y. Kupchik,et al. The role of the nucleus accumbens and ventral pallidum in feeding and obesity , 2021, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[14] Yong Xu,et al. A hindbrain dopaminergic neural circuit prevents weight gain by reinforcing food satiation , 2021, Science Advances.
[15] R. Malenka,et al. Input-specific modulation of murine nucleus accumbens differentially regulates hedonic feeding , 2021, Nature Communications.
[16] B. Popkin,et al. Individuals with obesity and COVID‐19: A global perspective on the epidemiology and biological relationships , 2020, Obesity reviews : an official journal of the International Association for the Study of Obesity.
[17] Christopher M. Mazzone,et al. High fat food biases hypothalamic and mesolimbic expression of consummatory drives , 2020, Nature Neuroscience.
[18] K. Hajifathalian,et al. Obesity is Associated with Worse Outcomes in COVID‐19: Analysis of Early Data from New York City , 2020, Obesity.
[19] Huang-yuan Li,et al. Ventral tegmental area GABAergic neurons induce anxiety-like behaviors and promote palatable food intake , 2020, Neuropharmacology.
[20] JaneR . Taylor,et al. Medial Nucleus Accumbens Projections to the Ventral Tegmental Area Control Food Consumption , 2020, The Journal of Neuroscience.
[21] R. Wise,et al. Control of food approach and eating by a GABAergic projection from lateral hypothalamus to dorsal pons , 2020, Proceedings of the National Academy of Sciences.
[22] Brian Zingg,et al. Synaptic Specificity and Application of Anterograde Transsynaptic AAV for Probing Neural Circuitry , 2020, The Journal of Neuroscience.
[23] R. Wise,et al. Dopamine and Addiction. , 2020, Annual review of psychology.
[24] Ilana B. Witten,et al. Specialized coding of sensory, motor, and cognitive variables in VTA dopamine neurons , 2019, Nature.
[25] R. Malenka,et al. Nucleus Accumbens Modulation in Reward and Aversion , 2019, Cold Spring Harbor symposia on quantitative biology.
[26] Christina K. Kim,et al. A Neural Circuit Mechanism for Encoding Aversive Stimuli in the Mesolimbic Dopamine System , 2019, Neuron.
[27] Liqun Luo,et al. Topological Organization of Ventral Tegmental Area Connectivity Revealed by Viral-Genetic Dissection of Input-Output Relations , 2019, Cell reports.
[28] Mohammad Hosein Farzaei,et al. Global, regional, and national comparative risk assessment of 84 behavioural, environmental and occupational, and metabolic risks or clusters of risks for 195 countries and territories, 1990–2017: a systematic analysis for the Global Burden of Disease Study 2017 , 2018, Lancet.
[29] Karl Deisseroth,et al. Mapping projections of molecularly defined dopamine neuron subtypes using intersectional genetic approaches , 2018, Nature Neuroscience.
[30] Evan Z. Macosko,et al. Molecular Diversity and Specializations among the Cells of the Adult Mouse Brain , 2018, Cell.
[31] M. Priest,et al. Oxytocin functions as a spatiotemporal filter for excitatory synaptic inputs to VTA dopamine neurons , 2018, eLife.
[32] S. Lammel,et al. Nucleus Accumbens Subnuclei Regulate Motivated Behavior via Direct Inhibition and Disinhibition of VTA Dopamine Subpopulations , 2018, Neuron.
[33] Mark A. Rossi,et al. Overlapping Brain Circuits for Homeostatic and Hedonic Feeding. , 2018, Cell metabolism.
[34] M. Luijendijk,et al. Does activation of midbrain dopamine neurons promote or reduce feeding? , 2017, International Journal of Obesity.
[35] A. Bonci,et al. Circuit specificity in the inhibitory architecture of the VTA regulates cocaine-induced behavior , 2017, Nature Neuroscience.
[36] T. Wadden,et al. Mechanisms, Pathophysiology, and Management of Obesity , 2017, The New England journal of medicine.
[37] Elyssa B. Margolis,et al. Ventral tegmental area: cellular heterogeneity, connectivity and behaviour , 2017, Nature Reviews Neuroscience.
[38] E. Roh,et al. Emerging role of the brain in the homeostatic regulation of energy and glucose metabolism , 2016, Experimental & Molecular Medicine.
[39] R. Wise,et al. Lateral hypothalamic circuits for feeding and reward , 2016, Nature Neuroscience.
[40] Guillem R. Esber,et al. Brief optogenetic inhibition of dopamine neurons mimics endogenous negative reward prediction errors , 2015, Nature Neuroscience.
[41] C. Lüscher,et al. Accumbal D1R Neurons Projecting to Lateral Hypothalamus Authorize Feeding , 2015, Neuron.
[42] S. Sternson. Hunger: The carrot and the stick , 2015, Molecular metabolism.
[43] Zhiping P Pang,et al. Endogenous Glucagon-like Peptide-1 Suppresses High-Fat Food Intake by Reducing Synaptic Drive onto Mesolimbic Dopamine Neurons. , 2015, Cell reports.
[44] Liqun Luo,et al. Circuit Architecture of VTA Dopamine Neurons Revealed by Systematic Input-Output Mapping , 2015, Cell.
[45] S. Nakanishi,et al. Aversive behavior induced by optogenetic inactivation of ventral tegmental area dopamine neurons is mediated by dopamine D2 receptors in the nucleus accumbens , 2014, Proceedings of the National Academy of Sciences.
[46] K. Deisseroth,et al. Medial prefrontal D1 dopamine neurons control food intake , 2014, Nature Neuroscience.
[47] S. Ikemoto,et al. Similar Roles of Substantia Nigra and Ventral Tegmental Dopamine Neurons in Reward and Aversion , 2014, The Journal of Neuroscience.
[48] K. Deisseroth,et al. Input-specific control of reward and aversion in the ventral tegmental area , 2012, Nature.
[49] Jovi C. Y. Wong,et al. Insulin in the ventral tegmental area reduces hedonic feeding and suppresses dopamine concentration via increased reuptake , 2012, The European journal of neuroscience.
[50] Kelly R. Tan,et al. GABA Neurons of the VTA Drive Conditioned Place Aversion , 2012, Neuron.
[51] G. Stuber,et al. Activation of VTA GABA Neurons Disrupts Reward Consumption , 2012, Neuron.
[52] B. Swinburn,et al. The global obesity pandemic: shaped by global drivers and local environments , 2011, The Lancet.
[53] B. Roth,et al. Rapid, reversible activation of AgRP neurons drives feeding behavior in mice. , 2011, The Journal of clinical investigation.
[54] P. Kenny. Reward Mechanisms in Obesity: New Insights and Future Directions , 2011, Neuron.
[55] M. Nicolelis,et al. Remote Control of Neuronal Activity in Transgenic Mice Expressing Evolved G Protein-Coupled Receptors , 2009, Neuron.
[56] S. Lammel,et al. Unique Properties of Mesoprefrontal Neurons within a Dual Mesocorticolimbic Dopamine System , 2008, Neuron.
[57] Charles R. Gerfen,et al. Targeting Cre Recombinase to Specific Neuron Populations with Bacterial Artificial Chromosome Constructs , 2007, The Journal of Neuroscience.
[58] Xiao-Bing Gao,et al. Ghrelin modulates the activity and synaptic input organization of midbrain dopamine neurons while promoting appetite. , 2006, The Journal of clinical investigation.
[59] Xiao-Bing Gao,et al. Leptin Receptor Signaling in Midbrain Dopamine Neurons Regulates Feeding , 2006, Neuron.
[60] M. W. Schwartz,et al. Central nervous system control of food intake and body weight , 2006, Nature.
[61] B. Hoffer,et al. Characterization of a mouse strain expressing Cre recombinase from the 3′ untranslated region of the dopamine transporter locus , 2006, Genesis.
[62] R. Palmiter,et al. Dopamine Production in the Caudate Putamen Restores Feeding in Dopamine-Deficient Mice , 2001, Neuron.
[63] R. Palmiter,et al. Feeding behavior in dopamine-deficient mice. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[64] A. Kelley,et al. Feeding induced by GABA(A) receptor stimulation within the nucleus accumbens shell: regional mapping and characterization of macronutrient and taste preference. , 1999, Behavioral neuroscience.
[65] R. Palmiter,et al. Dopamine-deficient mice are severely hypoactive, adipsic, and aphagic , 1995, Cell.