Effects of nitric oxide on neutrophil influx depends on the tissue: role of leukotriene B4 and adhesion molecules
暂无分享,去创建一个
F. Rocha | V. Girão | F. Cunha | S Y Fukada | D. Dal‐Secco | Acrm Leite
[1] M. Rossi,et al. Nitric oxide inhibits neutrophil migration by a mechanism dependent on ICAM-1: role of soluble guanylate cyclase. , 2006, Nitric oxide : biology and chemistry.
[2] J. Silva,et al. Neutrophil recruitment in immunized mice depends on MIP‐2 inducing the sequential release of MIP‐1α, TNF‐α and LTB4 , 2006 .
[3] Chinh N. Tran,et al. Synovial biology and T cells in rheumatoid arthritis. , 2005, Pathophysiology : the official journal of the International Society for Pathophysiology.
[4] M. Teixeira,et al. Blockade of leukotriene B4 prevents articular incapacitation in rat zymosan-induced arthritis. , 2004, European journal of pharmacology.
[5] F. Cunha,et al. Tumour necrosis factor-alpha mediates neutrophil migration to the knee synovial cavity during immune inflammation. , 2004, European journal of pharmacology.
[6] Elahé T. Crockett,et al. Gene deletion of P-Selectin and ICAM-1 does not inhibit neutrophil infiltration into peritoneal cavity following cecal ligation-puncture , 2004, BMC clinical pathology.
[7] J. O'dell,et al. Therapeutic strategies for rheumatoid arthritis. , 2004, The New England journal of medicine.
[8] D. Shibata,et al. In vitro mutation artifacts after formalin fixation and error prone translesion synthesis during PCR , 2004, BMC clinical pathology.
[9] J. Emmerich,et al. Inhibition of leukotriene B4-induced CD11B/CD18 (Mac-1) expression by BIIL 284, a new long acting LTB4 receptor antagonist, in patients with rheumatoid arthritis , 2004, Annals of the rheumatic diseases.
[10] F. Cunha,et al. Neutrophil migration in inflammation: nitric oxide inhibits rolling, adhesion and induces apoptosis. , 2003, Nitric oxide : biology and chemistry.
[11] M. Peters-Golden,et al. Prolonged lipopolysaccharide inhibits leukotriene synthesis in peritoneal macrophages: mediation by nitric oxide and prostaglandins. , 2003, Prostaglandins & other lipid mediators.
[12] W. Muller. Leukocyte-endothelial-cell interactions in leukocyte transmigration and the inflammatory response. , 2003, Trends in immunology.
[13] F. Cunha,et al. Involvement of NO in the failure of neutrophil migration in sepsis induced by Staphylococcus aureus , 2002, British journal of pharmacology.
[14] F. Cunha,et al. Dual effect of nitric oxide in articular inflammatory pain in zymosan‐induced arthritis in rats , 2002, British journal of pharmacology.
[15] F. Cunha,et al. Nitric Oxide Mediates the Inhibition of Neutrophil Migration Induced by Systemic Administration of LPS , 2001, Inflammation.
[16] S. Wahl,et al. Selective Inhibition of Inducible Nitric Oxide Synthase Exacerbates Erosive Joint Disease , 2001, The Journal of Immunology.
[17] F. Cunha,et al. Role of nitric oxide in the failure of neutrophil migration in sepsis. , 2000, The Journal of infectious diseases.
[18] J. Cyster,et al. Tumor necrosis factor: a master-regulator of leukocyte movement. , 2000, Immunology today.
[19] M. M. Pompeu,et al. Periarthritis promotes gait disturbance in zymosan-induced arthritis in rats , 1999, Inflammation Research.
[20] X. Q. Zhang,et al. Leukocyte entry into sites of inflammation requires overlapping interactions between the L-selectin and ICAM-1 pathways. , 1999, Journal of immunology.
[21] J. Hogg,et al. Nitric oxide reduces the sequestration of polymorphonuclear leukocytes in lung by changing deformability and CD18 expression. , 1999, American journal of respiratory and critical care medicine.
[22] M. Perretti,et al. Role of inducible nitric oxide synthase in the regulation of neutrophil migration in zymosan‐induced inflammation , 1998, Immunology.
[23] A. Lentsch,et al. In Vivo Recruitment of Neutrophils: Consistent Requirements for L-Arginine and Variable Requirements for Complement and Adhesion Molecules , 1998, Inflammation.
[24] J. Liao,et al. Differential regulation of endothelial cell adhesion molecule expression by nitric oxide donors and antioxidants , 1998, Journal of leukocyte biology.
[25] W. B. van den Berg,et al. Reduced cartilage proteoglycan loss during zymosan-induced gonarthritis in NOS2-deficient mice and in anti-interleukin-1-treated wild-type mice with unabated joint inflammation. , 1998, Arthritis and rheumatism.
[26] G. Haskó,et al. The crucial role of IL‐10 in the suppression of the immunological response in mice exposed to staphylococcal enterotoxin B , 1998, European journal of immunology.
[27] R. Armstrong,et al. Investigation of the role of nitric oxide and cyclic GMP in both the activation and inhibition of human neutrophils , 1997, British journal of pharmacology.
[28] P. Kubes,et al. Role of nitric oxide in regulation of leucocyte‐endothelial cell interactions , 1997, Experimental physiology.
[29] I. Laurindo,et al. Nitric oxide synthase inhibitor influences prostaglandin and interleukin-1 production in experimental arthritic joints , 1997, Inflammation Research.
[30] A. M. Lefer,et al. The role of nitric oxide and cell adhesion molecules on the microcirculation in ischaemia-reperfusion. , 1996, Cardiovascular research.
[31] J. Vansteveninck,et al. Modulation of neutrophil migration by exogenous gaseous nitric oxide , 1996, Journal of leukocyte biology.
[32] G. Constantin,et al. Interleukin 10 (IL-10) inhibits the release of proinflammatory cytokines from human polymorphonuclear leukocytes. Evidence for an autocrine role of tumor necrosis factor and IL-1 beta in mediating the production of IL-8 triggered by lipopolysaccharide , 1993, The Journal of experimental medicine.
[33] M. Teixeira,et al. E‐type prostaglandins enhance local oedema formation and neutrophil accumulation but suppress eosinophil accumulation in guinea‐pig skin , 1993, British journal of pharmacology.
[34] S. Wahl,et al. Suppression of arthritis by an inhibitor of nitric oxide synthase , 1993, The Journal of experimental medicine.
[35] T. Billiar,et al. Inhibition of chemotaxis Ng-monomethyl-L-arginine: a role for cyclic GMP. , 1989, Blood.
[36] D. Gilroy,et al. Nitric Oxide Synthase Inhibitors Have Opposite Effects on Acute Inflammation Depending on Their Route of Administration , 2000 .
[37] M J Smith,et al. Leukotriene B4. , 1981, General pharmacology.