Candidate gene variants of the immune system and sudden infant death syndrome

[1]  T. Dörk,et al.  Polymorphisms in genes of respiratory control and sudden infant death syndrome , 2015, Zeitschrift für Rechtsmedizin.

[2]  Gabi Kastenmüller,et al.  SNiPA: an interactive, genetic variant-centered annotation browser , 2014, Bioinform..

[3]  M. Klintschar,et al.  Mitochondrial deoxyribonucleic acid may play a role in a subset of sudden infant death syndrome cases , 2014, Acta paediatrica.

[4]  M. Klintschar,et al.  Association between polymorphisms in the P2RY1 and SSTR2 genes and sudden infant death syndrome , 2013, International Journal of Legal Medicine.

[5]  M. Carroll,et al.  Congenital central hypoventilation syndrome and sudden infant death syndrome: disorders of autonomic regulation. , 2013, Seminars in pediatric neurology.

[6]  T. Rognum,et al.  Gene variants predisposing to SIDS: current knowledge , 2011, Forensic science, medicine, and pathology.

[7]  C. Courts,et al.  Genetics of the sudden infant death syndrome. , 2010, Forensic science international.

[8]  M. Cohen,et al.  Contribution of bacteriology and virology in sudden unexpected death in infancy , 2010, Archives of Disease in Childhood.

[9]  H. Kinney,et al.  Interleukin-6 and the serotonergic system of the medulla oblongata in the sudden infant death syndrome , 2009, Acta Neuropathologica.

[10]  H. Kinney Brainstem mechanisms underlying the sudden infant death syndrome: evidence from human pathologic studies. , 2009, Developmental psychobiology.

[11]  M. Klintschar,et al.  A functional polymorphism in the tyrosine hydroxylase gene indicates a role of noradrenalinergic signaling in sudden infant death syndrome. , 2008, The Journal of pediatrics.

[12]  J. Weiss,et al.  The bactericidal/permeability-increasing protein (BPI) in infection and inflammatory disease. , 2007, Clinica chimica acta; international journal of clinical chemistry.

[13]  Dan M Roden,et al.  Cardiac potassium channel dysfunction in sudden infant death syndrome. , 2007, Journal of molecular and cellular cardiology.

[14]  P. Schwartz,et al.  Cardiac Sodium Channel Dysfunction in Sudden Infant Death Syndrome , 2007, Circulation.

[15]  B. Roep,et al.  Mannose-binding lectin: clinical implications for infection, transplantation, and autoimmunity. , 2006, Human immunology.

[16]  Sophia M. Moscovis,et al.  Cytokine responses and sudden infant death syndrome: genetic, developmental, and environmental risk factors , 2005, Journal of leukocyte biology.

[17]  R Holle,et al.  KORA - A Research Platform for Population Based Health Research , 2005, Gesundheitswesen (Bundesverband der Arzte des Offentlichen Gesundheitsdienstes (Germany)).

[18]  M. Korachi,et al.  Interleukin 10 genotype as a risk factor for sudden infant death syndrome: determination of IL-10 genotype from wax-embedded postmortem samples. , 2004, FEMS immunology and medical microbiology.

[19]  Sophia M. Moscovis,et al.  Ethnicity, infection and sudden infant death syndrome. , 2004, FEMS immunology and medical microbiology.

[20]  Roger W Byard,et al.  Sudden infant death syndrome and unclassified sudden infant deaths: a definitional and diagnostic approach. , 2004, Pediatrics.

[21]  J. Cherry,et al.  A controlled study of the relationship between Bordetella pertussis infections and sudden unexpected deaths among German infants. , 2004, Pediatrics.

[22]  W. Köpcke,et al.  German study on sudden infant death (GeSID): design, epidemiological and pathological profile , 2004, International Journal of Legal Medicine.

[23]  T. Rognum,et al.  IL-10 gene polymorphisms are associated with infectious cause of sudden infant death. , 2003, Human immunology.

[24]  F. Hauck,et al.  Sleep environment and the risk of sudden infant death syndrome in an urban population: the Chicago Infant Mortality Study. , 2003, Pediatrics.

[25]  A. Hajeer,et al.  Association of IL-10 genotype with sudden infant death syndrome. , 2000, Human immunology.

[26]  C. Wilson,et al.  Sudden infant death syndrome and Canadian Aboriginals: bacteria and infections. , 1999, FEMS immunology and medical microbiology.

[27]  T. Rognum,et al.  IL-6 Cerebrospinal Fluid Levels Are Related to Laryngeal IgA and Epithelial HLA-DR Response in Sudden Infant Death Syndrome , 1999, Pediatric Research.

[28]  A. Busuttil,et al.  Mannan binding lectin and the sudden infant death syndrome. , 1998, Forensic science international.

[29]  P. J. Fleming,et al.  Smoking and the sudden infant death syndrome: results from 1993-5 case-control study for confidential inquiry into stillbirths and deaths in infancy , 1996 .

[30]  A. S. Douglas,et al.  Seasonality and the sudden infant death syndrome during 1987-9 and 1991-3 in Australia and Britain , 1996, BMJ.

[31]  A. Busuttil,et al.  Infectious agents, the inflammatory responses of infants and sudden infant death syndrome (SIDS). , 1995, Molecular medicine today.

[32]  O. Saugstad,et al.  S cases have increased levels of interleukin‐6 in cerebrospinal fluid , 1995, Acta paediatrica.

[33]  F. Stanley,et al.  The association between the prone sleeping position and sudden infant death syndrome (SIDS): An editorial overview , 1991, Journal of paediatrics and child health.

[34]  L. S. James,et al.  Defining the sudden infant death syndrome (SIDS): deliberations of an expert panel convened by the National Institute of Child Health and Human Development. , 1991, Pediatric pathology.

[35]  M. N. Marra,et al.  Bactericidal/permeability-increasing protein has endotoxin-neutralizing activity. , 1990, Journal of Immunology.

[36]  H. Hoffman,et al.  Risk Factors for SIDS , 1988, Annals of the New York Academy of Sciences.

[37]  M. Sakanoue [Sudden infant death syndrome]. , 1985, [Kango gijutsu] : [Nursing technique].

[38]  C. Poets,et al.  Hyperthermia in sudden infant death , 2005, International Journal of Legal Medicine.

[39]  M. Willinger Defining the sudden infant death syndrome (SIDS) , 1991 .

[40]  H. Hoffman,et al.  Risk factors for SIDS. Results of the National Institute of Child Health and Human Development SIDS Cooperative Epidemiological Study. , 1988, Annals of the New York Academy of Sciences.